Emergency Paediatrics Flashcards
What do you do if a child has NICE traffic light
a) amber signs
b) red signs
a) can go home with strict safety netting or refer to hospital
b) immediate transfer to hospital
What are the NICE traffic light amber signs?
- pale
- reduced activity, wake with stimulation
- nasal flaring, sats <95%
- poor feeding, reduced UO
- temp >39
What are the NICE traffic light red signs?
- blue, mottled
- not rousable, high pitched cry
- grunting or chest indrawing or RR >66
- reduced skin turgor
- temp >38 if <3 months old
- bulging fontanelle, non blanching rash, seizures
What is grunting?
Trying to maintain PEEP - creates an end pressure to prevent the alveoli from closing
What are the signs of potential circulatory failure?
Cap refill - sternum for 5 seconds BP, HR Urine output Skin colour/temperature - skin mottling Look for resp failure, distress, agitation, conscious level, rapid deep breathing due to metabolic acidosis Drowsy, still child, unresponsive
What are signs of potential central neurological failure?
Conscious levels - AVPU, GCS
GCS of 8 - unresponsive that you no longer protect airway, so may need to intubate, aprox P of AVPU
Posture
Pupillary signs
What are red flags in children?
Hypoxia - can compensate for long time
Hypotension
Silent chest
Unequal pupils - late sign, raised ICP dilatation pressure on oculomotor means herniation
(fixed and dilated - dead, one reactive - one squished)
Posturing - decorticate vs decerebrate can indicate brain injury or herniation occurring/about to occur
What congenital heart defect is the most common cause of heart failure?
VSD
What are the types of poisoning in a child?
Accidental
Deliberate by older sibling
Non accidental - abuse
Iatrogenic
What are examples of potential accidental poisonings?
Low - COCP, abx, chalk, crayons, washing powder
Intermediate - paracetamol, salbutamol, bleach, disinfectants, foschia
High - alcohol, digoxin, iron, salicylate, TCAs, acids, yew
What are the clinical features of poisons?
Aspirin, CO - tachypnoea Opiates - resp depression Alcohol - resp depression TCA, beta blocks - hypotension TCA, organophosphates - convulsions TCA, drugs - large pupils Organophosphates - small pupils
What is the management of poisoning?
Identify agent
Assessment of agent’s toxicity via TOXBASE
Removal of poison:
Activated charcoal, ineffective for iron, insecticides, aspiration can cause pneumonitis
By NG or oral
Gastric lavage, rarely used in children only if large quantity
Induced vomiting with ipecac rare
Investigations:
Blood glucose - alcohol
Blood levels
Toxicology screen
Plan clinical management Low toxicity - home Intermediate - observe High - admit Specific antidotes
Assess social circumstances
What is the management for button batteries?
Monitor progress with chest and abdo x-rays
Almost all pass within 2 days
Remove batteries if in oesophagus or sign of disintegration
What injuries may a child suffer with trauma?
Abdominal injuries e.g. ruptured spleen, liver, kidney
Scans, x-rays, observation
Chest injuries - pneumothorax, haemopericardium
What is the management of burns and scalds?
Is airway, breathing, circulation satisfactory
Any smoke inhalation
Depth of burn - if full thickness will require graft
Surface area of burn
Involvement of special sites
What primary damage occurs from head injuries in children?
Cerebral contusions or lacerations
Dural tears
Diffuse axonal damage
What secondary damage occurs from head injuries in children?
Hypoxia from airway obstruction, or inadequate ventilation
Hypoglycaemia
Hyperglycaemia
Reduced cerebral perfusion due to hypotension from bleeding, raised ICP
Haematoma, infection from open wound or CSF leak
What is the primary survey in a head injury?
A-E assessment
What indicates a potentially serious head injury?
Witnessed LOC >5 mins
Amnesia >5 mins
Abnormal drowsiness
3 or more episodes of vomiting
Clinical suspicion of NAI
post traumatic seizure, but no history of epilepsy
GCS <15
Suspicion of open/depressed skull injury, tense fontanelle
Basal skull fracture signs - panda eyes, battle sign, haemotympanum
Dangerous mechanism e.g. fall, RTA, high speed object
What are signs of secondary damage?
Persisting coma
Deteriorating GCS
Seizures without full recovery
Focal neurological signs