Electrophysiology Flashcards

1
Q

The SA node is supplied by which arteries?

A

In 55% of ppl, the RCA.

In 45% of ppl, the LCA.

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2
Q

The AV node is supplied by which artery?

A

The SA nodal artery.

It arises from:

In 90% of ppl, the RCA.

In 10% of ppl, the LAD.

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3
Q

The RBB and LASF bundle are supplied by which artery?

A

In 50% of ppl, the LAD.

In 50% of ppl, the AV nodal artery and the LAD.

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4
Q

The LPIF is supplied by what arteries?

Will LAD infarction affect the LPIF?

A

In 50% of ppl, the AV nodal artery, and 50% the AV nodal artery and LAD.

LAD infarction will usually cause block of the RBB and LASF, but very rarely will it affect the LPIF.

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5
Q

How should you break down tachycardias?

A
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6
Q

What is meant by SVT?

Classically, what are the two rhytms that are classified as an SVT?

A

A tachycardia originating above the AV node.

AvnRT and AVRT.

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7
Q

What percentage of people with SVT have AvnRT vs AVRT?

A

60% have AvnRT (AV re-entry)

20% have AVRT (bypass tract re-entry)

20% have SVT that re-enters at orther sites

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8
Q

What are characteristics of AvnRT?

A

Rate: 180-250 bpm in both atria and ventricles

Small R’ wave, and QRS <120msec

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9
Q

How is AvnRT treated?

A

Vagal manouevers: Valsalva (must be held for at least 10 seconds, 20-25% success), carotid massage, ice on face (used only for children)

Adenosine (6mg): (90% success rate, safe in unstable, safe in pregnancy, and in narrow complex WPW)

Sync. Cardioversion:

CCBs and Beta Blockers: 2nd line. Have hypotensive effects.

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10
Q

What are the characteristics of AVRT?

A

Most are anterograde thru the AV, and re-enterant through the bypass tract.

85% of WPW are ORTHOdromic in nature (narrow complex)

ANTIdromic (wide complex) is hard to differentiate from VT, and should be treated as such in the ED.

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11
Q

What defines WPW type A?

WPW type B?

A

Type A: Positive delta wave in V1, with dominant R wave

Type B: Negative delta wave in V1, with dominant S wave

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12
Q

A small portion of WPW may have a bypass circuit that has a quicker refractory time than the AV node.

If this person developed a Afib or AF, excessive stimulation of the ventricles may occur.

ANTIdromic WPW with Afib or AF (a venticular rate >300 on monitor) should be watched for what?

And what should be avoided in their treatment?

A

Precipitation into VF.

Avoid Beta blockers, CCBs, and adenosine, as it may block the AV node, which favors the 1:1 conduction through the fast bypass circuit, and will further increase the chances of potentiating a VF or VT.

Treatment should be synced cardioversion or procainamide (this does block the bypass circuit)

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13
Q

What gives you L axis deviation?

A

LVH

Inferior MI

LAFB

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14
Q

What gives you R axis deviation?

A

RVH

LPFB

Dextrocardia

Mixing up of electrodes

Lung hyperinflation (heart rotates more vertical)

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15
Q

What causes early R wave progression?

A

RVH (uniform progression)

Posterior MI (2 contig leads)

RBBB

WPW

HOCM

Peds ECG

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