Elbow/wrist/hand Flashcards
Elbow deformities- cubits varus
Aka gun-stock
Most obvious when elbows are ext + elevated
Most common cause is malunion of sypracondyl ar fracture (abnormal healing) s
Can be corrected by wedge osteopathy of lower humerus
Larger q-angle
Elbow deformities- cubits valgus
Most common cause is non-union of fractured lat epicondyle, may give gross deformity + bony knob on inner side of Jt
Increases liability of ulnar palsy development years after injury, Pt notices weakness of hand, numbness + tingling of ulnar fingers (ring + little)
Doesn’t need treatment, but for delayed ulnar palsy the nerve needs to be transposed to front of elbow (stops pinching)
Elbow deformities- stiff elbow
Can be severe impediment
Pt may be unable to reach out to, or bring back from, their environment
Cannot turn palm downwards to pick something up, or palm up to lift
Causes include congenital disorders, trauma, and arthritis
If physio doesn’t help, surgery is needed
Clinical presentation of TB
TB rarely seen until arthritis supervenes
Onset is insidious, long Hx of aching + stiffness
Marked wasting
While disease is active, Jt is held flexed, looks swollen, feels warm + tender
Limited ROM
P + spasm
Prognosis of TB
Anti-TB drugs
Rest in splint
Later treatment- collar + cuff
Surgery rarely needed
RA clinical presentation
Elbow involved in more than 50% of cases
Rheumatoid nodules can be detected over olecranon
P + tenderness, especially around head of radius
Eventually whole elbow becomes swollen + unstable
Often bilateral
X-ray shows bony erosion, gradual destruction of radial head + widening of trochlear notch of ulnar
Prognosis of RA
Splinted during periods of synovitis
If entire Jt is severely damaged, replacement should be considered
OA clinical presentation
Uncommon site for OA
P + stiffness
Jt may become unstable
Elbow looks + feels enlarged
Limited ROM
X-ray shows reduced Jt space with sub Honduran sclerosis + osteopahytes, one or more loose bodies may be seen
Prognosis of OA
Rarely requires more than symptomatic treatment
Loose bodies should be removed if they cause locking
If stiffness is disabling, removal of osteopahytes can improve ROM
If signs of ulnar neuritis the N can be transposed to front of elbow
Loose bodies epidemiology
Trauma
Osteochondritis dissects of capitulum
Multiple may occur with OA or synovial chondromatosis
Clinical presentation of loose bodies
Locking of elbow
Prognosis of loose bodies
Can be removed= reduction in symptoms
Olecranon bursitis epidemiology
Olecranon bursa can enlarge due to pressure or friction
When accompanied with P, cause is more likely to be from infection- gout (if Hx of previous attacks), RA
Clinical presentation of olecranon bursitis
Bilateral
X-ray shows calcification of bursa (gout)
Swelling + nodularity over olecranon, erosion of Jt may cause instability- RA
Prognosis of olecranon bursitis
Underlying disorders must be treated
Septic bursitis may need draining
Chronically large may need to be removed
Difference between tennis + golfers elbow
Tennis- lateral epicondylitis
Golfers- medial epicondylitis
Lateral epi Hx
Repetitive motion (gripping, hammering, lifting, tennis backhand)
Lat epi SSx
Tender to palpation over lateral epicondyle/common extensor tendon
Initiated/aggravated by movement
Risk factors of tennis/golfers
Resistive flex/ext
Certain jobs
Medial epi SSx
Tender to palpation over medial epicondyle/common flexor tendon
Prognosis of tennis/golfers elbow
Rest/avoiding precipitating activity
Splint + physio may help
If P severe area of max tenderness is injected with mixture of corticosteroid + local anaesthetic
Persistent P with no response to treatment may require surgery
Pronator teres syndrome Hx
Repetitive motions (gripping with pronation)
Tingling and weakness in hand
Pronator teres syndrome SSx
Tender to palpation over mid pronator teres
Weakness in wrist flexion
UCL rupture Hx
Prior elbow dislocation
Throwing injury or chronic overload
UCL rupture SSx
Tenderness to palpation of UCL
+ve valgus stress test
Wrist deformities- congenital variation
Embryonic abnormalities of upper limb, likely to affect more than one segment of limb
Anomalies occur together in forearm, wrist, hand
Other organs may be affected + may be associated congenital abnormalities
Overall incidence of upper limb abnormalities estimated to be 1/600 live births, only a fraction severe enough for surgery
Wrist deformities- radial dysplasia
Infant born with marked radial deviation
Aka club hand
1/2 Pt affected bilaterally
Absence of whole or part of radius, usually small thumbs
Treatment of radial dysplasia
Neonatal includes gentle stretching + splintage
Serious cases treated with distraction prior to tension free soft tissue correction
Has less effect on growth of carpus + distal ulnar
Prolonged splintage still required to avoid reoccurrence of deformity
If function deteriorates, centralisation over ulnar recommended, preferably before age 3