Cervical conditions Flashcards
Causes of cervical strain
Rear ended car collision, sports trauma, receptive occupational injuries
Hx of overuse or trauma
Risk factors of cervical strain
Radiographic image used to rule out more severe injuries
Clinical presentation of cervical strain
Treat only as diagnosis of exclusion
Inspect neck for skin integrity, edema (swelling), ecchymosis (bruising), or asymmetry
Palpate tenderness- SPs –> paraspinal soft tissue
Assess ROM if Pt able to
Cervical strain DDX
Cervical sprain
Facet syndrome
Meniscoid entrapment
Physical findings of cervical strain
Palpable bogginess of posterior neck muscles, cervical tissue oedema (not pitting- application of pressure causes indentation), limited ROM
What is cervical strain
Whiplash
Result of sudden hyperextension followed by hyeprflexion of neck
Muscles and ligaments stretch beyond capacity = inflammation in local tissues
Associated symptoms of cervical strain
Persistent stiffness, trapezoidal pain, back pain, muscle spasm, headache
Symptoms begin acutely, hours after injury
Prognosis of cervical strain
Initially causes distress, minimal long term sequela (condition which is a consequence of previous injury)
Degeneration
Accumulated wear and tear that occurs over a long period of time
Degenerate age group
Elderly people
Risk factors for degeneration
Aging, hard manual labour, contact sports
Clinical presentation of degeneration
Neck pain/stiffness, inflexibility, limited ROM
Pt >50 years
Gradual onset
Crepitus
Morning stiffness
Dull achy P
Associated symptoms of degeneration
Numbness, tingling, potential weakness in neck, arms or shoulders because nerves in cervical region become irritated or pinched
Prognosis for degeneration
Treatment involves rest, pain medication, NSAIDs (non-steroid anti-inflammatory drugs), and physical therapy
Goal of physical is to increase flexibility, postural training and strengthen parapsinal muscles
Restoring flexibility prevents further repetitive microtrauma from poor movement patterns
Herniation
Tear or rupture of fibrocartilagneous material (annulus) that surrounds interverebral disc
Most common spinal levels- L4-5, L5-S1 (95-98% of all herniations)
Patho herniation
Secondary to disc degeneration (annulus fibrosis)
External protrusion of gelatinous nucleus pulposus through annular fibres potentially causing compression of spinal nerve segment (radiculopathy)
In addition to damage and comp, there is also potential chemical irritation from the release of inflammatory products and local oedema
Epidemiology of herniation
Traumatic events, many cases occur spontaneously
Herniation- age groups affected
Young adults
Herniation risk factors
High contact sports
Herniation clinical presentation
Can be mistaken for shoulder/forearm pathology (assess to rule out)
Complaints of weakness, numbness, and tingling in shoulder region down to fingers
Limited ROM
Bicep weakness, numbness in thumbs and index fingers
Symptoms vary dependent on which nerve roots
Unilateral symptoms, unless central herniation
Associated symptoms of herniation
Frequent headaches, pain originating in paraspinal muscles radiating down upper extremity
Prognosis of herniation
Should improve over time and symptoms become less intense following manual therapy
Stenosis
Narrowing of spaces in spine which can compress spinal cord
Age groups affected by sternosis
50+ due to osteoarthritic symptoms beginning leading to changes in spinal structures