EKG II- Other EKG Changes (Electrolytes, Hypothermia, Pericarditis, PE, Stroke) Flashcards
Hyperkalemia
T waves start to peak (ALL LEADS.)
- if QRS progressively widens and merges w/T wave–> V FIB.
- progressive evolution
- LEADS TO V-FIB if not corrected
Hypokalemia
- ST segment depression
- T wave flattening
- QT prolongation and U wave
U wave best seen in anterior leads
U wave is more prominent than T wave (pos deflection)
U wave after the T wave
Calcium- Hypocalcemia and Hypercalcemia
HYPO- prologned QT interval
(concerns for Torsades de Pointes)
HYPER- shortened QT interval
remember that HR inc as QT shortens, and HR dec as QT prolongs
Hypothermia
- sinus bradycardia
- ALL segments PROLONGED
- Osborn Wave: special ST segment elevation
- absrupt ascent with a plunge to baseline
J point is the exact end of QRS and start of ST segment–> in osborn wave there is a pos deflection at the J point
Pericarditis
- diffuse ST segment and T wave changes
- large effusion can cause low voltage adn electrical alternans (change in amplitude of QRS)
electrical alternans- alternating amplitudes in QRS complex
Acute Pulmonary Embolism
- Lead I: large S wave
- Lead III: deep Q wave
(S1Q3 pattern) - T wave inversion in II
S1Q3T3
S1Q3T3 is all 3
Central Nervous System events
CVA events- cerebrovascular events
- diffuse T wave inversions
- prominent U waves
- sinus bradycardia often seen
caused by CVA events (subarachnoid bleed or cerebral infarction)