EKG- Electrolytes Flashcards

1
Q

What is flattening and/or inversion of T-waves which indicates no ventricle relaxation?

A

HYPOKalemia- <3.5 w/ see EKG changes

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2
Q

Less K will mean less repolarization, what else will show on EKG?

A

ST depression and prominent U-waves- BC ventricle most contract at some point

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3
Q

What is the junction of the QRS and beg of ST segment?

A

J point.

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4
Q

What are cause and result of hypokalemia?

A

Hydrochlorothiazides, Furosemide.
Ventricular arrhythmia risk.
Check K+ and MG labs

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5
Q
Ekg has the following. what electrolyte is causing these disturbances?
1. Peaked T waves 
2. Prolong PR 
3. Absent P- waves
4 Wide QRS.
A

HYPERkalemia- level 5.5, ventricle in long contraction

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6
Q

What is the MC cause of HYPERkalemia

A

chronic renal failure-

imbalance of aldosterone

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7
Q

What can lead to torsades?

A

HYPOKalemia no relax, irregular torsades

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8
Q

What electrolyte level can lead to V-tach?

A

HYPERkalemia, long relaxed sinusoidal rhythm

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9
Q

Which electrolyte imbalance is correctable?

A

HYPERKalemia

  1. Calcium, Saline-stabilize membrane
  2. Insulin- Lateral-shift K level
  3. Furosemide, Sodium Bicarb- excretors
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10
Q

What leads are HYPERkalemia seen and what else is seen?

A

MC precordial leads V1-V6.

  1. DEEP QRS V2
  2. LBBB-V1 or V6
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11
Q

Which RX drug is treatment and proarrhythmia in CVD?

A

Digoxin- treat HF and arrhythmias

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12
Q

How does Digoxin affect PRI?

A

INC AV nodal refractory period.
2. Pos. Ionotropic/contract and NEG conotrophic/ DEC HR.
LONG contraction, slower HR.

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13
Q

What are CP signs of toxicity w/ Digoxin?

A

HA, weak, seizure, drowsiness, AN, N/V yellow halo, palpitation, dec HR

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14
Q

What else should be monitored with Digoxin?

A

K levels- can also lower K

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15
Q

What will Digitalis toxicity show on EKG?

A
  1. Shorter QT
  2. SCOOP of ST-T-
  3. ST+T fused w/in scoop
  4. PRI prolonged
  5. Bradycardia
  6. U wave
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16
Q

What will the scoop look like?

A
  1. Hockey stick R mustache

2. lowercase h followed with U wave (hence HYPOKalemia risk)

17
Q

What are the QT prolongation drugs and T wave inversion/flattening?

A

Quinidine, Procainamide, disopyramide

18
Q

What is seen with hypothermia?

A
  1. Osborne J waves in precordial and inferior leads
    BUTTed up against Post QRS and b4 ST segment
  2. Bradycardia
19
Q

Why would hypercalcemia produce J waves?

A

Tetany. Hypothermia Ventricular frozen in time

20
Q

What shows the severity of hypothermia?

A

Ht of the J-wave

21
Q

How do you distinguish J waves from RBBB?

A

J waves curved seen in INF and precordial.

RBBB- sharp curves in V1 or V6

22
Q

What is GS for DX of PE?

A

CT scan.

ECG can show PE, but no dX

23
Q

Describe each ECG finding of a PE?

A
  1. Sinus tachy- faster to get blood to system d/t block
  2. RV strain seen in VI -V4, relate to R side of heart pulmonary circuit to lungs
  3. ST depression- ischemia tissue dying
  4. RBBB-right side struggling
24
Q

Which Leads are significant signs of PE?

A

S1- deep S WAVE IN LIMB-I R-side heart.
Q3- Q WAVE IN LIMB3-lung strain
T3- INVERTED T-WAVE IN LEAD 3- ventricular strain

25
Q

Who needs a pre op EKG?

A

PHM- CVD, CVA, comorbidity, CAD, arry, PAD, structure heart. Bleeding risk

26
Q

What are concerns for pre op EKG?

A
  1. Q Waves
  2. STEMI, NSTEMI-dep
    3 LVH
  3. QT long
  4. BBB
  5. Arrhythmias
27
Q

When is a nuclear stress test advised?

A

STABLE Angina DOE w/ TKR

28
Q

When is TM stress test advised?

A

STABLE Angina DOE, SOB.

29
Q

Ms. Niehaus has syncope, DOE but her EKG was NSR, what monitor is ideal for her?

A

Zio Patch-14, where outside clinic. Holter- 24-48h