ectopy and abbarency Flashcards
Premature atrial contraction
Rate: determine underlined rate. Rhythm: irregular. P wave: Present, may be different w/ PAC. P:QRS ratio is 1:1 PR-interval: Normal, may vary /w PAC. QRS width- below 120ms.
An abnormal (non-sinus) P wave is followed by a QRS complex.
The P wave typically has a different morphology and axis to the sinus P waves.
The abnormal P wave may be hidden in the preceding T wave, producing a “peaked” or “camel hump” appearance — if this is not appreciated the PAC may be mistaken for a PJC.
PACS arising close to the AV node (“low atrial” ectopics) activate the atria retrogradely, producing an inverted P wave with a relatively short PR interval ≥ 120 ms (PR interval < 120 ms is classified as a PJC).
PACs that reach the SA node may depolarise it, causing the SA node to “reset” — this results in a longer-than-normal interval before the next sinus beat arrives (“post-extrasystolic pause”). Unlike with PVCs, this pause is not equal to double the preceding RR interval (i.e. not a “full compensatory pause”).
PACs arriving early in the cycle may be conducted aberrantly, usually with a RBBB morphology (as the right bundle branch has a longer refractory period than the left). They can be differentiated from PVCs by the presence of a preceding P wave.
Similarly, PACs arriving very early in the cycle may not be conducted to the ventricles at all. In this case, you will see an abnormal P wave that is not followed by a QRS complex (“blocked PAC”). It is usually followed by a compensatory pause as the sinus node resets.
rate
PJC
Rate: determine underlined rhythm. Rhythm: irregular. P wave: none, antegrade, or retrograde. P:QRS ratio is 1:1 or retrograde PR-interval: none, short or retrograde QRS width- below 120ms.
arrow QRS complex, either (1) without a preceding P wave or
(2) preceded by an abnormal P wave with a PR interval of < 120 ms (these “retrograde” P wave are usually inverted in leads II, III and aVF).
Occurs sooner than would be expected for the next sinus impulse.
Followed by a compensatory pause.
PJCs that arrive early in the cycle may be conducted aberrantly, most commonly with a RBBB morphology.
PVC
Rate: determine underlined rhythm. Rhythm: irregular. P wave: none with PVC P:QRS ratio is none PR-interval: none QRS width- above 120ms WIDE.
**Compensatory pause is seen.
Broad QRS complex (≥ 120 ms) with abnormal morphology.
Premature — i.e. occurs earlier than would be expected for the next sinus impulse.
Discordant ST segment and T wave changes.
Usually followed by a full compensatory pause.
Retrograde capture of the atria may or may not occur.
PVCs may be either:
Unifocal — Arising from a single ectopic focus; each PVC is identical.
Multifocal — Arising from two or more ectopic foci; multiple QRS morphologies
PVCs often occur in repeating patterns:
Bigeminy — every other beat is a PVC. Trigeminy — every third beat is a PVC. Quadrigeminy — every fourth beat is a PVC. Couplet — two consecutive PVCs. Triplet — three consecutive PVCs.
Escape beats- junctional escape beat
Rate: determine underlined rhythm. Rhythm: irregular. P wave: none, antegrade or retrograde P:QRS ratio is 1:1 or retrograde PR-interval: none, short or retrograde QRS width- below 120ms.
ventricular escape beat
Rate: determine underlined rhythm. Rhythm: irregular. P wave: none with VEB P:QRS ratio is 1:1 none PR-interval: none QRS width- wide
Ashman phenomenon
Ashman phenomenon, also known as Ashman beats, describes a particular type of wide QRS complex, often seen isolated that is typically seen in atrial fibrillation.
** It is more often misinterpreted as a premature ventricular complex.
Ashman beats are described as wide complex QRS complexes that follow a short R-R interval preceded by a long R-R interval.[3] This wide QRS complex typically has a right bundle branch block morphology and represents an aberrantly conducted complex that originates above the AV node, rather than a complex that originates in either the right or left ventricle.