ECG- Recognising Common Abnormalities Flashcards

1
Q

What are causes of abnormal rhythms? (2)

A

Abnormal impulse formation

Abnormal conduction (AV block)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Where in the heart can rhythms arise from and what are they called?

A

SAN-supraventricular rhythms
Atrium- supraventricular rhythms
AV node- supraventricular rhythms
Ventricle- ventricular rhythms

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What are the features of supraventricular rhythms?

A
  • conducted into and within ventricles by His-Purkinje system
  • normal ventricular depolarisation
  • normal (narrow QRS) complexes
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

What are the features of a ventricular rhythm?

A
  • from foci in ventricle
  • conduction not via usual His-purkinje system
  • depolarisation takes longer
  • wide and bizarre QRS complex (different foci produce different shapes)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What are the 3 types of ventricular rhythms?

A

Ventricular premature beats
Ventricular tachycardia
Ventricular fibrillation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is atrial fibrillations?

A

Random impulses causing chaotic depolarisation- cardiac cannot contract in a coordinated way

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What effect does atrial fibrillation have on CO?

A

It drops it slightly

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

What is the ECG of atrial fibrillations?

A

No p waves just wavy baseline (atria quiver rather than contract)

Narrow QRS at irregular intervals

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What happens to the heart rate and pulse rate during atrial fibrillations?

A

They become irregularly irregular

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Why would blood thinners be given to those who have atrial fibrillations?

A

Clots can form in the striatum because there is static blood this can cause strokes etc

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What is AV conduction blocks?

A

Delay/failure of conduction of impulses from atrium to ventricles via AV node and bundle of His

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are the two main causes of AV conduction block?

A

Acute MI - full recovery within a few days

Degenerative changes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What are the 3 types of heart blood?

A

First degree block

Second degree heart block: type 1 and type 2

Third degree heart block (complete heart block)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What happens in complete heart block?

A

A pacemaker in the ventricle will take over (ventricular escape rhythm)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

What is first degree heart block defined by?

A

Slow conduction in AV node and His Bundle

Normal P wave
Prolonged PR interval (>5 small squares)
Normal QRS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

What is type 1, 2nd degree heart block?

A

Progressive lengthening of PR interval until one P is not conducted (this allows time for AVN to recover) then cycle begins again

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What is type 2, 2nd degree heart block?

A

PR interval normal
Sudden non-conduction of a beat (dropped beat without warning- ie no QRS)

High risk of progression to complete heart block

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

What is 3d degree heart block?

A

Atrial depolarisation normal but no impulses are conducted to ventricles

Ventricular pacemaker takes over-wide QRS (ventricular escape rhythm)

No relationship between the P and the QRS wave of an ECG

19
Q

What are the problems with 3rd degree heart block?

A

Heart rate is too slow- 30/40
Too slow to maintain BP and perfusion
Urgent pacemaker insertion required

Possible for someone to go into asystole

20
Q

What are the features of ventricular ectopic beats?

A

Impulses not spread via the His-purkinje system

Much slower depolarisation of ventricles

Wide QRS complex, different in shape to usual

21
Q

What is ventricular tachycardia?

A

Run of 3 or more consecutive ventricular ectopics

Broad complex tachycardia

22
Q

Why is persistence ventricular tachycardia so dangerous?

A

Because the person is at high risk of ventricular fibrillation

They will need to be electrically shocked back into rhythm

23
Q

What is ventricular fibrillation?

A

Abnormal, chaotic, fast, ventricular depolarisation

Caused by impulses form numerous ectopic site in ventricular muscle

No co-ordinated contraction causing ventricles to quiver

24
Q

What effect doe VF have on CO?

A

No cardiac output, cardiac arrest

25
Ischameia and MIs can effect any part of the heart, how can you tell where the damage is?
Changes seen in leads facing affected area. Look at which ECG lead is abnormal
26
What region of tissue in the heart is most vulnerable from lack of perfusion?
Sub-endocardial region
27
What changes will you see in an ECG lead facing an area of ischaemic cardiac tissue?
ST segment depression Or T wave inversion Due to abnormal current during repolarisation *ischaemic ECG changes may only be seen during exercise but if severe reduction of lumen: ischaemic changes at rest
28
What is STEMI?
ST segment elevation myocardial infarction Due to complete occlusion of lumen by thrombus Occurs when muscle injury extends 'full thickness' from endocardium to epicardium
29
What defines a pathological Q wave?
Wider than 1 small square (longer than 0.04 seconds) Deeper than 2 small squares (Depth more than 1/4 of the height of the subsequent R wave
30
What effect does hyperkalaemia have on the heart?
RMP less negative which inactivates some voltage gates Na+ channels - heart is less excitable
31
What are the ECG changes seen with hyperkalaemia?
High T wave Prolonged PR interval, depressed ST segment P wave absent Ventricular fibrillation
32
What ECG changes are seen in hypokalaemia?
Low T wave
33
What is the cardiac axis?
The average direction of spread of ventricular depolarisation Usually Downwards and left (-30 to +90 degrees)
34
What is left axis deviation?
When overall direction of ventricular depolarisation is upwards and to the left (less than -30 degrees)
35
What is left axis deviation associated with?
Conduction block of anterior branch of left bundle Inferior MI Left ventricular hypertrophy
36
What is right axis deviation?
When overall direction of ventricular depolarisation is downwards and to the right (more than +90 degrees)
37
What is right axis deviation associated with?
Right ventricular hypertrophy
38
How can you spot left axis deviation?
Look at leads I and aVF (or III) If QRS is upright in lead I an inverted in aVF (or in III), it is LAD
39
How can you spot righ axis deviation?
Look at leads I and aVF/III If QRS is inverted in lead I and upright in lead III (aVF) then it is RAD
40
In an ECG of complete heart block what its he relationship between the P waves and the QRS complex?
No relationship between them because the atria and ventricle are being stimulated by different pacemakers
41
What might have happened to cause a widened QRS complex?
Problems with ventricle wall, bundle branch block, ischaemia, drugs, metabolic abnormalities, hypokalaemia
42
What do pathological Q waves suggest?
Indicates an area of muscle necrosis following ischaemia
43
What does ST depression suggest?
Reduced perfusion to the myocardial muscle
44
If a p wave can be seen on an ECG, it is possible for someone to be having AF?
No