ECG abnormalities Flashcards

1
Q

what are the supraventricular rhythms

A

rhythms arising above the ventricle so either from the sinus node, the atrium or the AV node

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2
Q

true or false: supraventricular nodes give a wider QRS complex

A

false - they have a normal, narrow complex as there is still normal ventricular depolarisation

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3
Q

what happens to the QRS complex in ventricular rhythms

A

it widens as the depolarisation doesn’t conduct through the usual His-Purkinje system and so takes longer

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4
Q

what is atrial fibrillation

A

where there are multiple atrial impulses and so chaotic atrial depolarisation

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5
Q

what does an ECG look like in atrial fibrillation

A

no p waves, wavy baseline, normal QRS complexes

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6
Q

true or false - during atrial fibrillation all of the impulses arriving at the AVN are conducted to the ventricles

A

false - due to the refractory period of the AVN not all impulses will be conducted

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7
Q

how often are impulses conducted to the ventricles in atrial fibrillation

A

at irregular intervals

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8
Q

what happens to the pulse and heart rate in atrial fibrillation

A

they are irregular

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9
Q

what is heart block/AV conduction block

A

where they is a delay or failure to conduct impulses from the atria to the ventricles via the AVN and bundle of His

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10
Q

what causes heart block

A
  • myocardial infarction (e.g. lack of blood supplying the AVN node prevents it from working)
  • degenerative changes
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11
Q

how many types of heart block are there

A

3

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12
Q

what is first degree heart block

A

where there are prolonged PR intervals which last longer than 5 small squares (0.20 seconds) due to slow conduction

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13
Q

what does an ECG look like in primary heart block

A

normal p wave
normal QRS
increased PR interval

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14
Q

what is Mobitz type 1 second degree heart block

A

where the length of the PR interval progressively increases until a QRS complex cant be conducted

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15
Q

what is Mobitz type 2 secondary heart block

A

where the PR interval is normal and there is a sudden non-conduction of an impulse to the ventricles

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16
Q

what is 3rd degree heart block

A

where atrial depolarisation is normal but no impulses are conducted to the ventricles
the ventricular pacemaker must then takeover

17
Q

what rhythm does the ventricular pacemaker give

A

the ventricular escape rhythm which is very slow and gives a wide QRS complex

18
Q

why is a pacemaker urgently required in 3rd degree heart block

A

because the heart rate set up by the ventricular escape rhythm is too slow (30-40bpm) to maintain BP and perfusion

19
Q

what is an ECG of 3rd degree heart block like

A

no relationship between p waves and QRS
RR intervals much slower
PP intervals constant

20
Q

what are ventricular ectopic beats

A

when there is an opportunistic beat coming from the ventricles

21
Q

how are ventricular ectopic beats different from the escape rhythm

A

the ectopic beat is a one off beat that does not occur due to impulse delays

22
Q

what does the QRS complex look like in a ventricular ectopic beat

A

wider

23
Q

what is it called if there are more than 3 consecutive ventricular ectopic beats

A

ventricular tachycardia which gives a high risk of ventricular fibrillation

24
Q

what needs to be given during ventricular tachycardia

A

electrical shock

25
Q

what is ventricular fibrillation

A

where there is abnormal, chaotic ventricular depolarisation with impulses originating from numerous ectopic sites in the ventricles

26
Q

what happens to the cardiac output in ventricular fibrillation

A

there is no cardiac output as theres no coordinated contraction leading to cardiac arrest

27
Q

where in the heart is the most vulnerable to reduced perfusion

A

the sub endocardial muscle as it is the furthest away to the coronary arteries

28
Q

when are ischaemic ECG changes seen

A

during exercise

or at rest when theres severe narrowing of the artery lumen

29
Q

what changes are seen on an ECG during exercise and why

A
  • ST depression
  • T wave inversion
    due to abnormal repolarisation
30
Q

what changes does a myocardial infarction have on an ECG during a complete occlusion of an artery lumen

A

elevated ST segment

31
Q

what happens to the ECG weeks after an STEMI

A

the ST and T are normal

persistent Q wave

32
Q

what does the persistent Q wave indicate

A

muscle necrosis

33
Q

describe the pathological Q waves seen in a MI

A

1 small square wide
2 small squares deep
more than 1/4 of the height of the R wave

34
Q

what happens to the resting membrane potential in hyperkalaemia and hypokalaemia

A
  • hyper = less negative (more depolarised)

- hypo = more negative (hyper polarised)

35
Q

what happens to the excitability of the heart as the hyperkalamia worsens

A

It becomes less excitable so theres a prolonged PR interval

36
Q

what happens to the T wave in hyper and hypokalaemia

A
hyper = tall and pointed 
hypo = low