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YEAR 3 & 5 CORE CONDITIONS > Ear and Eye (ENT 2) > Flashcards

Ear and Eye (ENT 2) Flashcards

1
Q

What are the four categories of dizziness?

A

Vertigo
Presyncope
Disequilibrium
Light headedness

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2
Q

What is vertgio? How may this be described?

A

Imbalance of tonic vestibular signal

False sense of motion / spinning sensation

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3
Q

What is disequilibirum?

A

Symmetric vestibular loss, proprioceptive loss, cerebellar damage

Off balance / wobbly

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4
Q

what is presyncope?

A

Diffusely diminished blood flow / blood

Feeling of losing consciousness / blacking out

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5
Q

What is psychogenic?

A

Abnormality in the integration of afferent signal by CNS

Vague sumptoms
Possibly feeling disconnected with the environment

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6
Q

What may cause disequilibrium?

A

Cerebellar damage
Basal ganglia lesion
Cerebellar damage

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7
Q

What may cause presyncope?

A

Orthostatic hypotension
Hypoglycaemia
Cardiac arrhythmia

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8
Q

List some peripheral causes of vertigo

A 
Benign paroxsymal positional vertigo (BPPV)
Vestibular neuritis
Meniere disease
Otoscleroris
Lavrynthitis
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9
Q

List some central causes of vertigo

A 
Vestibular migraine
CV disease
Cerebellopontine angle and posterior fossa meningiomas
MS
Acoustic neuroma
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10
Q

How does BPPV present?

A

Attacks of sudden rotational vertigo lasting >30s are provoked by head turning

Other otological symptoms rare

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11
Q

How is BPPV diagnosed?

A

Establish important negatives:
No persistent vertigo
No speech, visual, motor or sensory problems
No tinnitus, headache, ataxia, facial numbness, dysphagia

Hallpike test +ve

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12
Q

What is an acoustic neuroma?

A

Usually painless benign subarachnoid tumours that cause problems by local pressure, and then behave as SOL

Rare = 1 / 100,000 / year

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13
Q

Where do acoustic neuromas usually arise?

A

Superior vestibular Schwann cell layer

Sometimes called vestibular schwannoma

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14
Q

How do acoustic neuromas present?

A

Progressive ipsilatural tinnitus +/- sensorineural deafness (cochlear nerve vompression)
Disequilibrium common
Vertigo rare
Trigeminal compression above tumour may give numb face

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15
Q

What investigations are done for an acoustic neuroma?

A

MRI for all those with unilateral tinnitus / deafness

Key differential = meningioma

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16
Q

What is the management of an acoustic neuroma?

A

Surgery is difficult and not often needed eg if elderly

Methods of preserving hearing and facial nerve eg stereotactic radiosurgery

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17
Q

What is tinnitus?

A

Perception of sound in the ears or head where no external source of the sound exists

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18
Q

How can tinnitis be divided?

A

Objective (audible to examiner) - rare

Subjective (audible only to pt)

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19
Q

What can cause objective tinnitis?

A

1) Vascular disorders = Pulsatile vibratory sounds
AV malformations, glomus tumours

2) High output cardiac states = Pulsatile sounds
Pagets, hyperthyroid, anaemia

3) Myoclonus of palatal of stapedius / tensor tympani muscles = audible click
4) Patulous eustachian tube = prolonged opening, causing abnormal sound transmission to ear

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20
Q

What can cause subjective tinnitis?

A

Most commonly associated with disorders causing sensorineural hearing loss eg presbycusis, noise induced hearing loss, Menieres (unilateral)

Conductive deafness less common eg impacted wax, otosclerosis

Otitis media with effusion 
Thyroid dysfunction
DM
MS
Acoustic neuroma
Anxiety
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21
Q

List some ototoxic drugs?

A

Cause bilateral tinnitis with associated hearing loss

Cisplatin and aminoglycosides = permanent hearing loss

Aspirin, NSAIDs, quinine, macrolides and loop diuretics are associated with tinnitus and reversible hearing loss

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22
Q

List some aminoglycosides

A 
Gentamicin
Tobramycin
Plazomicin
Streptomycin
Neomycin
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23
Q

What is the pathophysiology of tinnitis

A

Poorly understood

Spontaneous otoacoustic emissions
Altered or increased spontaneous activity of auditory nerve
Inappropriate feedback pathways in auditory-limbic system

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24
Q

What tests can be done for tinnitis?

A

Audiometry
Tympanogram
MRI for unilateral tinnitus to exclude acoustic neuroma

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25
Q

What is the treatment of tinnitus?

A

Treat any underlying cause
Explain - it common and usually improves with time via habituation
Hearing aids - if hearing loss >35db, improves perception of background noise and makes tinnitus less apparent
Psychological support eg sound therapy (radio / fan use to mask sound)
CBT

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26
Q

What is glaucoma?

A

Group of eye diseases associated with acute or chronic destruction of the optic nerve with or without increased intraocular pressure (IOP)

Aqueous humor is produced by the ciliary body on the iris, flows from the posterior chamber through the pupil into the anterior chamber, and then drains back into the venous system via the trabecular meshwork in the angle of the anterior chamber.
- Any process that can disrupt the flow can lead to optic nerve damage

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27
Q

What are the two main types of glaucoma?

A

Open angle (90%) = slowly progressive, initially often asymptomatic, but leads to peripheral vision loss over time (peripheral to central)

Acute angle closure glaucoma

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28
Q

What is acute angle closure glaucoma?

A

Angle of anterior chamber narrows narrows acutely causing a sudden rise in IOP to >30mmHg (normal range is 15-20)

Pupil becomes fixed and dilated and axonal death occurs

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29
Q

What is primary vs secondary acute angle closure glaucoma?

A 
Primary = anatomical predisposition
Secondary = pathological processes eg traumatic haemorrhage pushing the posterior chamber forwards
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30
Q

What factors predispose acute angle closure glaucoma?

A

Hypermetropia (long-sightedness)
Pupillary dilatation
Lens growth associated with age (typically 40-60yr)
Asian

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31
Q

How does acute angle closure glaucoma present?

A

severe pain: may be ocular or headache
decreased visual acuity
symptoms worse with mydriasis (e.g. watching TV in a dark room)
hard, red-eye
haloes around lights
semi-dilated non-reacting pupil
corneal oedema results in dull or hazy cornea
systemic upset may be seen, such as nausea and vomiting and even abdominal pain

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32
Q

What is the management of acute angle closure glaucoma?

A

Urgent referral to opthalmologist for gonioscopy - angle between iris and cornea checked (drainage angle)

Triad of:

1) Beta blockers to suppress aqueous humour production eg timolol 0.5%
2) Pilocarpine 2-4% drops/2hrs - pupillary constriction opens trabecular mesh work and increases drainage
3) 500mg IV acetazolamide stat then 250mg/8hr PO/IV - reducing aqueous humour formation

Admit to monitor IOP

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33
Q

What is done once IOP is controlled in acute angle closure glaucoma?

A

Peripheral iridectomy - a piece of iris is removed (at ‘12 o clock’) in both eyes to allow aqueous to flow

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34
Q

What are some complications of acute angle closure glaucoma?

A

Visual loss
Central retinal artery or vein occlusions
Repeated episodes

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35
Q

What is the management of open angle glaucoma?

A

Prostaglandin eye drops eg latanoprost 50mcg/ML / travoplost

Laser trabeculopasty
Surgical trabeculectomy

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36
Q

What is a cataract?

A

Any opacity in the lens

Leading cause of blindness

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37
Q

How common are cataracts?

A

75% of >65yr
20% of 45-65yr

Occur earlier in DM

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38
Q

What should be investigated when a cataract is found?

A

Fasting plasma glucose
Steroid use
High myopia - nearsightedness
Dystrophia myotonica

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39
Q

What is noted on examination of cataracts?

A

Loss of red reflex

NB in immature cataracts it may still be present

40
Q

How are cataracts classified?

A

Nuclear
Cortical
Posteriorsubcapsular

41
Q

Describe nuclear cataracts

A

Affects centre of lens - affects the lens refractive index and can cause more myopia (nearsightedness) at first or temporarily improve reading

With time, lens turns more yellow / brown and clouds vision

Common in old age

42
Q

Describe cortical cataracts

A

Affects the edges of the lens - wedge shaped opacities which have milder effects on vision

43
Q

Describe posterior subcapsular cataracts

A

Progress faster and cause the classic glare from bright sunlight and lights whilst driving at night, even when visual acuity is only mildly affected

Small, opaque area area at the back of the lens

44
Q

How do cataracts present?

A

Blurred vision

Unilateral = often unnoticed but loss of steropsis affects distance judgement

Bilateral = gradual painless loss of vision (frequent changes of glasses as refraction changes)
+/- dazzle esp in sunlight
+/- monocular diplopia (double vision resolves when affected eye covered)

Difficulty driving at night and haloes around street lights

45
Q

How may cataracts present in children?

A

Squint
White pupil
Nystagmus / amblyopia

46
Q

What is stereopsis?

A

Depth perception due to the slightly different perspectives that each individual eye has

47
Q

What is amblyopia?

A

aka lazy eye

Decreased visual acuity

48
Q

What is the management of cataracts?

A

Mydriatic drops (dilate pupils) / sunglasses can help

Surgery if large effect on ADL or cannot read number plate from 20m (and a driver)

Treat congenital cataracts surgically ASAP to prevent amblyopia - must be corrected within latent period of visual development (1st 6 weeks of life)

49
Q

What surgery is offered for cataracts?

A

Day case using LA with small incision

Lens removed by phacoemulsion - broken up by US then aspirated into a cannula

Incision enlarged and an artificial lens (eg perspex, acrylic or silicon) implanted

Abx and anti-inflammatory drops used 3-6wks post op

2% get serious complications

50
Q

How can cataracts be prevented?

A

Sunglasses
Decrease oxidative stress eg with antioxidants
Stop smoking / alcohol

51
Q

What are some causes of cataracts?

A

Congenital <1% eg TORCH esp rubella

Acquired 99% eg age, DM, renal insufficiency, wilson’s, drugs induced eg glucocorticoids or anticholinesterases, sunlight, infections

52
Q

What is keratitis?

A

Corneal inflammation
Identified by a white area on the cornea indicating a collection of white cells in corneal tissue

Keratoconjunctivitis = conjunctivitis with associated corneal involvemet

53
Q

What is a corneal abrasion? How may it present?

A

An epithelial breach
Causing pain, photophobia +/- decreased vision

Non infective corneal ulcers eg accidental scratches / contact lenses

Infective

54
Q

What are some infective causes of corneal abrasion?

A 
Bacterial (beware pseudomonas - may progress rapidly)
Herpetic
Fungal - candida, aspergillus
Protozoal = acanthamoeba
Basculitis eg in RA
55
Q

How can a corneal lesion be identified?

A

Use fluorescein drops and blue light on a slit lamp

Corneal lesions stain green - drops are orange and become yellow on contact with eye

Always invert the eye lid to look for FB

56
Q

How can a simple corneal abrasion be managed (non infective)?

A

Chlorampenicol ointment

+/- cycloplegia (relax ciliary muscle relieving pain)

57
Q

What is a corneal abrasion vs an ulcer?

How is corneal ulcer managed?

A

Abrasion = scratch to epithelium but does not go through the Bowman’s layer underneath (an ulcer does)

Refer to specialist
Remove contact lenses
Diagnostic smear / gram stain and scrape

58
Q

What causes dendritic corneal ulcers?

A

HSV type 1

59
Q

How do dendritic ulcers present?

A

Pain
Photophobia
Watering

60
Q

What is the management of dendritic ulcers?

A

Aciclovir 3% eye ointment 5 x daily

Corneal transplants for those with significant visual impairment due to scarring

61
Q

What is the management of corneal ulcers?

A

Until cultures known, alternative chloramphenicol drops (for gram +ve) with ofloxacin (or gram -ve) drops

or
0.3% cefuroxime drops with gentamicin drops

admit in DM, immunosuppressed or pt won’t manage drops

Steroid drops can be added once recovery has begun

62
Q

How are ofloxacin drops given?

A

Up to one drop every 15min

63
Q

What are some risks of steroid eye drops?

A

Associated with increasing IOP (which can be risky in glaucoma pt) and cataract formation

Can potentially prolong the course of viral conjunctivitis by increasing the latency of adenovirus

Careful not to miss corneal ulcer as steroids can propagate the ulcer causing corneal melt and blindness

64
Q

What is a stye?

A

General term for inflammatory lid swelling, could be:

Hordeolum externum
Hordeolum internum
Chalazion
Meibomian cyst

65
Q

What is a hordeolum externum?

A

Abscess or infection, usually staph, in a lash follicle

These can also involve the glands of Moll (sweat glands) and of Zeis (sebum glands attached directly to lash follicles)

Can cause much inflammation

66
Q

How is hordeolum externum treated?

A

Apply hot compress for 5-10 mins several times each day until the stye resolves

67
Q

What is a hordeolum internum?

A

An abscess of the Meibomiam glands

Open onto conjunctiva and cause less local reaction but leave a residual swelling = chalazion or a meibomiam cyst when they subside

68
Q

Which styes point inwards and outwards?

A

Hordeolum externum = ‘outwards’

Hordeolum internum = ‘inwards’

69
Q

What may cause vision to decrease in hordeolum externum?

A

Corneal flattening (rare)

70
Q

How are residual swellings managed in hordeolum internum?

A

Incision and curettage under LA

71
Q

What is lid inflammation called?

A

Blepharitis

72
Q

What can cause blepharitis?

A

Staph infection
Seborrhoeic dermatitis
Rosacea

73
Q

How does blepharitis present?

A 
Burning feeling
Itching red margins
Crusting = staph infection
Gunking = secretions from Meibomian glands
Loss of eyelashes
Frothy tears
74
Q

What is the management of blepharitis?

A

Good eyelid hygiene - use a cotton bud and baby shampoo diluted 1:10 with warm water and clean along the lid margins twice a day

In children with blepharokeratitis, consider oral erythromycin too

75
Q

What is an entropion?

A

Lid inturning

76
Q

What commonly causes an entropion?

A

Degeneration of lower (rarely upper) lid fascial attachments and their muscles

Rare <40yr

Inturned eyelashes irritate the cornea

77
Q

What is the management of entropion?

A

Taping the lower eyelids to the cheek or botulum toxin injection to lower lid gives temporary relief
More lasting relief = surgery

78
Q

What is an ectropion?

A

Lower lid eversion causing eye irritation, watering +/- exposure keratitis

79
Q

What is ectropion associated with?

A

Older age

Facial palsy

80
Q

What is the management of an ectropion?

A

Plastic surgery may correct deformity

If facial palsy is the cause, consider surgical correction with an implant in upper lid to aid closure

81
Q

What can cause a red eye?

A 
Subconjunctival haemorrhage
Inflammation of the sclera - Scleritis and episcleritis
Uveitis
Anterior uveitis
Acute angle closure glaucoma
Conjunctivitis
82
Q

Describe episcleritis vs scleritis

A

Episcleritis = common, freq self limiting, usually benign

Scleritis = rare, very painful, sight threatening sequelae, strong association to systemic disease

83
Q

Who commonly gets episcleritis?

A

70% women

84
Q

What is the cause of episcleritis?

A

Mostly idiopathic

Minority of cases associated with underlying disease eg RA
Can be bacterial / viral infections or drug reaction (eg bisphosphonates)

85
Q

How does episcleritis present?

A 
Acute onset
Mild eye pain / irritation
Watering
Eye redness
Unilateral in 70%

NO VISION LOSS

86
Q

What are the two types of episcleritis?

A

Simple / diffuse = most common

Nodular / focal = occurs in those with underlying systemic disease, prolonged irritation

87
Q

How can episcleritis and scleritis be distinguished on examination?

A

Vasoconstricting eye drops eg phenylephrine 2.5% or 10%

Test whether blood vessels blanch, if they do = superficial vessels meaning episcleritis

If they do not, vessels are deeper = slceritis

88
Q

What is the management of episcleritis?

A

Symptomatic - cold compresses, eye lubrication

If persistent - NSAIDS / topical steroids

Treat underlying conditions

89
Q

What is the prognosis of episcleritis?

A

Usually spontaneous regression after 1-2 weeks

Can recur every 1-3 months

Rarely progresses to reduce visual acuity - important to rule out other causes that do need treatment eg scleritis, keratitis

90
Q

What is scleritis?

A

Generalised inflammation of the sclera itself with oedema to the conjunctiva, sclera thinning and vasculitic changes

91
Q

Describe the sclera in relation to the cornea

A

Cornea covers the iris and front of the eye is continuous with the sclera

92
Q

What is the cause of scleritis?

A 
60% associated with underlying disorders:
RA
SLE
Granulomatosis with polyangitis
Polymyositis
Gout
93
Q

What are the different types of scleritis?

A

Anterior (90%)
Posterior
Necrotising - can cause perforation of sclera

94
Q

How does scleritis present?

A

Constant severe dull ache which ‘bores’ into the eye
Ocular movements painful as the muscles insert onto the sclera
Eye redness
Headache
Photophobia +/- vision loss

95
Q

How is scleritis investigated?

A 
US for posterior scleritis
Orbital CT / MRI 
Systemic disease work up:
- RF = RA
- ANA = SLE
- ANCA = AI vasculitis
- HLA typing
96
Q

How is scleritis managed?

A

Depends on subtype

Non necrotising anterior = oral NSAIDS +/- PO high dose prednisolone

Posterior scleritis or any evidence of necrotising = systemic steroids

If no response to steroids = systemic immunosuppressives eg azathioprine or methotrexate

Scleral transplantation if impending perforation

97
Q

What complications are associated with scleritis?

A

Cataracts
Corneal changes
Glaucoma
Permanent loss of vision

YEAR 3 & 5 CORE CONDITIONS (30 decks)

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