Dysrhythmias and poor performance Flashcards

1
Q

Equien HR?

A

Resting: 2-42bpm
Can raise to 240 in exercising thoroughbred
Trot up to 140 in normal horse

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2
Q

when are dysrrhymias often seen?

A

during cool down when parasympathetic tone is highest

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3
Q

Common types of dysrhythmia?

A
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4
Q

When to investigate dysrhythmias?

A
  • Vetting – always listen at rest prior to examination, straight after exercise and
    during cooldown.
  • Heart rate or rhythm disturbance is detected by auscultation (often as part of a
    routine clinical examination)
  • Relevant clinical signs of cardiac disease (CHF, oedema, cough)
  • Client concerns
  • Poor performance
  • Collapse
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5
Q

How do we invesitgate?

A

ECG

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6
Q

How do we do ECG diagnostic?

A
  • Exercising ECg often to decide if horse is at risk
  • need to reach max exercise depending on breed and rq work
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7
Q

What does a normal equine ECG look like?

A

o P wave: large atria, often bifid
o QRS Complex: variation common
o T wave: changes during exercise
o PR interval
o RR interval

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8
Q

DESCRIBE 2nd ° Av block

A

o Common in fit/athletic horses at rest
o Due to high vagal tone
o Disappear with exercise/excitement or atropine administration

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9
Q

What primary cause of dysrhythmias?

A

isolated electrical disturbance

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10
Q

2ary causes of dysrhythmias?

A
  • Structural heart disease
  • Metabolic and endocrine disorders
  • Systemic inflammation
  • Hypotension, haemorrhage, anaemia and ischaemia
  • Autonomic influences
  • Toxicosis
  • Drugs
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11
Q

Describe Atrial Fibrillation

A

o The most common cardiac dysrhythmia in horses
o Can be associated with poor performance and exercise intolerance
o Caused by irregular depolarization of the atrial myocytes

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12
Q

Clinical signs of AFib?

A
  • Irregularly irregular rhythm -> intermittent long pauses with rapid runs
  • Maybe no CLs
  • Exercise intolerance (without cardiac structure change Afib will only affect performance in intense exercise)
  • Signs of CHF
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13
Q

What are the two classifications of Atrial fibrillation?

A
  • Lone atrial fibrillation
  • Atrial fibrillation with concurrent cardiac dx
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14
Q

Describe lone atrial fibrillation?

A
  • Irregular rhythm without evidence of cardiac disease
  • Young athletic horses (4.9% of racehorses in Hong Kong)
  • Sudden onset exercise intolerance
  • Often develops during/immediately afterstrenuous exercise
  • Revert to normal rhythm within 24h - 48h with no treatment
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15
Q

Describe Atrial fib with concurrent dx?

A
  • Elderly horses with loud murmurs of MR
  • Moderate to severe resting tachycardia with dysrhythmia: 50 bpm – 100 bpm
  • Clinical signs of CHF: exercise intolerance, dyspnoea, oedema, weight loss, collapse
  • Need further investigation and treatment
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16
Q

Diagnosis of A fib?

A
  • PE & Hx
  • Irregular heartbeat & pulse - ECg -> definitive diagnosis
  • Echocardiography eval of underlying dx)
  • Exercise ECG
17
Q

Afib on Echo?

A
  • Assess any regurg present?
  • Assess structural abn
  • Most likely atrial dilation

-> if structural abn -> tx likely unsuccessful

18
Q

Exercising ECg for Afib - do we need to convert?

A
  • If severe tachycardia present during exercise they are unsafe to ride as they
    are an increased risk of sudden cardiac death
  • Assess for any ventricular tachycardia
  • Must be done prior to commencing any treatment
19
Q

Prognosis for Afib?

A
  • HR >60 considered a poor prognostic marker
  • Inc atrial flutter rate inc lieklihood of recurrence
  • Atrial enlargement => poor prognostic marker
  • Duration. foA-fib matters (remodelling of atria!)
  • Lone A fib or recent onset = better prognosis
20
Q

Treatement for Afib?

A
  • conversion to normal sinus rhythm-> Exmaple: Transvenous electrical cardioversion (TVEC)
21
Q

Describe TVEC?

A
  • Widely performed in referral
  • Cardioversion catheters placed in pulm artery and right atrium
  • Synchronised electrical shock under GA
  • Sotalol used prior to and after TVEC
22
Q

Medical tx - Afib?

A

-> Quinidine sulphate
-> Amiodarone

23
Q

Describe quinidie?

A
  • Class IA anti-arrhythmic drug
  • PO administration via NGT
  • Repeat q2h until the rhythm converts to NSR
  • Only applied in horses with no systemic illness
  • Side effects: colic, diarrhea, tachycardia, urticaria, oedema of nasal mucosa, ventricular arrhythmias
24
Q

Describe Amiodarone?

A
  • Class III anti-arrhythmic drug
  • IV CRI administration
25
Q

What tx for cases showing signs of CHF at time of diagnosis?

A

o Supportive care before conversion trial
o Treatments to relieve signs of CHF: diuretics (e.g. furosemide)
o Improve ventricular response to fibrillating atria (e.g. digoxin)
o Conversion to NSR
o Poor prognosis though

26
Q

Summarise approach to A fib?

27
Q

DESCRIBE POST-NSR CONVERSION MONITORING?

A
  • Echo & 24h holter post NSd conversion
  • Anti-arrythmic drug (solatol) to prevent recurrence
  • Rest -> 4 weeks + periodical auscultation
28
Q

What causes primary tachyarrythmias?

A

Myocardial dx -> infectious, immune-M, toxic, nutritional, neoplastic ..

29
Q

Tachyarrythmais can also be 2ary to what factors?

A
  • Associated with non-cardiac causes
  • Hypoxia (e.g. dynamic airway obstruction in athletic horses)
  • Endotoxemia,septicemia, SIRS
  • Electrolytes and/or acid-base imbalance
30
Q

Describe Atrial PRemature contractions and wether to worry about them

A
  • On auscultation there will be premature beats interrupting the NSR
  • Rarely cause performance issues on their own

Biggest concern:
* Ability to incite a-fibrillation or a-flutter

31
Q

What is Atrial premature contraction associated with?

A

o Often associated with primary myocardial disease
o Ectopic premature atrial activation
o Abnormal P with normal QRS-T

32
Q

What abnormalities to note with premature atrial contractions?

A
  • Change in P wave morphology
  • QRS often normal BUT
  • QRS can be wide and biazrre
33
Q

Describe VPCs?

A
  • On auscultation there will be premature beats interrupting the NSR
  • Normally a compensatory pause following a VPC
  • The abnormal beat will often sound loud
  • Wide and bizarre QRS complex
34
Q

What are VPCs associated with and what will we see?

A

o Often associated with secondary to other causes
o Abnormal QRS-T
o The more complex and variable the VPCs, the higher the risk

35
Q

Management for tachyarrythmias?

A

o Primary goal istreating the underlying cause
- Often requires steroidal therapy
- Any infections treat those

o Anti-arrhythmic drugs
* Lidocaine and magnesium frequently used
* Beta-blockers can help
* Can consider digoxin if life threatening

36
Q

Prognosis for tachyA?

A
  • If lone VPC that is obliterated during exercise risk is small
  • If occurring just after exercise can be associated with parasympathetic
    tone and normal
  • If it occurs in exercise then serious concern
  • Only consenting adults should ride these horses
37
Q

Should. urest VPC?

A

yes at least >6months

38
Q

Describe high degree heart blocks?

A

o High grade 2º AV block or 3º AV block can result in syncope and collapse
o More frequently seen in donkeys and mules
o Management: pacemaker