Acute Lameness Flashcards

1
Q

What to say on first contact over the phone ?

A
  • Don’t move horse until vet assessment
  • If haemorrhage -> pressure bandage
    -> If foreign body say wether to remove or leave in situ

(if nail at risk of being pushed further into foot => remove but note direction)
if embedded -> leave in foot

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2
Q

How to do the initial assessment for acute lameness?

A
  • CE - mm colour, Hr, RR, neuro status
  • History -> trauma, sendduen onset, when seen last normal, any wounds ?
  • Sedate if safe to prevent further injury
  • If fract -> bandage/splint to prevent worsening
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3
Q

What further asessment should you do?

A
  • Full hx & CE
  • Plapation / hood testers
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4
Q

What are we looking for on palpation?

A

➢ Swelling/Joint effusion
➢ Wound
➢ Elevated digital pulses?
➢ Solar penetration?
➢ Hoof tester examination*******
➢ Pain on palpation /manipulation
➢ Reduced range of motion
➢ Crepitus
➢ Asymmetry

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5
Q

What are the main 8 causes of Acute severe lameness?

A
  1. Solar abscess/penetration
  2. Laminitis
  3. Cellulitis/lymphangitis
  4. Tendonitis/desmitis
  5. Synovial infection
  6. Fracture
  7. Tendon laceration/rupture
  8. Myopathy
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6
Q

Which diagnostic test would you choose for each condition?

A
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7
Q

HOW to TRANSPORT horses with MSK injury?

A
  • Minimise walking distance
  • Confine horse for leaning
  • Sufficient head/ neck mov to aid balance
  • Slings in ambulance if want to reduce load on all limbs
  • Unload carefully & slowly
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8
Q

Which direction fo transport horse with hind vs front limb issue?

A

-> Forward for fract of HL
-> Backward for fract of FL

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9
Q

What should you ALWAYs try to rule out in acute single limb lameness?

A

Solar abscess/ Nail bind

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10
Q

What is the clinical presentation for nail bind/ solar abscess?

A

Progressive/acute unilateral lameness
Increased digital pulses
Increased heat in foot
+ve hoof tester examination
+/- softening of coronary band/heel bulbs
+/- cellulitis of distal limb

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11
Q

What relevant history fo solar abscess/ nail bind case?

A

Recent farriery (nail bind)
Duration
Any objects removed from the foot
History of laminitis
Tetanus status

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12
Q

What particular test for nail bind?

A

Hoof tester over individual nails can identify painful one

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13
Q

How to manage solar abscess? (pt 1)

A
  • Remove shoe
  • Pare foor & identify tract
  • Follow tract & open abscess (black brown/yellow fluid)
  • Admin of anti-tet & toxoid
  • Hot tub
  • Iodine / epsom salts
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14
Q

How to manage solar abscess? (pt 2)

A

-> Apply poultice to draw out infection
➢ Animalintex most commonly used
➢ Apply hot and wet while abscess draining
➢ Switch to dry poultice when discharge stops
➢ See video for refresher on poulticing technique

NSAIDS

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15
Q

Are ABs indicated for uncomplicated abscesses ?

A

NO

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16
Q

Describe solar bruising?

A
  • Caused by blunt trauma
  • Haemorrhage and inflammation
  • Horses with thin soles/poor conformation
  • Unshod horses
  • Shod horses- shoeing overdue/shoes too small
  • Corn formation – ‘Seat of corn’
  • Can lead to abscess formation
  • May need hoof hardener/pads/hoof boots
  • Treat as abscess – see below
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17
Q

What is the classic presentation of laminitis?

A
  • Acute onset multi-limb lameness
  • Shifting weight/rocking on heels
  • Reluctant to walk with short choppy gait
    → worse on tight turns
  • ‘Bounding’ digital pulses
  • Heat and pain on hoof testers
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18
Q

What are the 3 clinical categories of laminitis?

A
  • Endocrinopathic (80%) -> EMS, PPID
  • Inflamamtory (systemic dx, grain overload, RFM)
  • Traumatic
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19
Q

How to treat Laminitis?

A
  • Strict rest and support the foot
  • Soft, deep bed
  • +/- remove shoes
  • Sole and frog supports
  • Pain relief and suppress inflammation
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20
Q

What pain releif woudl u use in laminitis?

A
  • Phenylbutazone 4.4 mg/kg IV/PO for 24-28 hrs
    2.2 mg/kg PO BID after 48 hrs

Additional pain relief?
* Paracetamol – not licensed
* Gabapentin – not licensed
* Opioid analgesia if severe

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21
Q

What is an ongoing tx for laminitis?

A
  • Ertuglifozon/Canagliflozin -> Sodium-glucose cotransporter 2 inhibitors (SGLT2i)
  • Dietary restriction
  • Low quality or soaked hay
  • Stop concentrates
  • Balancer should be given (vitamin and mineral supplement)
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22
Q

What else can we do for ongoign tx of laminitis?

A

REMEDIAL FARRIERY
- Reduce breakover
- Frog support
- Heart bar shoes
- Wooden shoes/ clogs

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23
Q

Solar panatrations are usually?

A

nails embedded in sole

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24
Q

What sensitive structures may or may not get involved in solar penetrations that we need to be aware of?

A

➢ Deep digital flexor tendon
➢ Distal Interphalangeal Joint
➢ Navicular Bursa
➢ Digital Flexor Tendon Sheath
➢ Navicular bone
➢ Pedal bone

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25
Q

What should we look for with solar penetration ?

A
  • Effusion of DIP/ DFTS -> Pare area aroudn tract - find haemorrhage, follow tract -> leakage? ; sterile probe in tract
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26
Q

What diagnostics might we wanna do for solar penetration?

A
  • Xray of foot -> ideally object in situ +/- probe or +/- contrast in tract
  • MRI
  • Synoviocentesis of DIP, Nb, DFTS
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27
Q

What meds to give for solar penetration?

A
  • Broad ABs (penicillin/ gentamicin)
  • NSAIDS
    +/- tetanus
28
Q

IF synovial structures are involved what to do?

A

arthroscopic debridement and lavage

29
Q

Whta does prognosis depend on?

A

What structure involved & duration from penetration to sx

30
Q

Pathogenesis of septic arthritis?

A
  • Same as in small animals
  • Inflammatory response
  • Cartilage destruction – extends to subchondral bone
  • PAIN and swelling of infected join
  • Ultimately leads to degenerative OA
31
Q

Tx for septic arthritis?

A
  • Joint lavage ASAP
  • Check tetanus status
32
Q

Septic arthritis causes in adult vs foals?

A

Adults-> laceration near joint or Iatrogenic
Foals -> Wound or haematogenous spread

33
Q

Presentation of septic arthritis?

A

➢ Acute onset/progressive severe lameness
➢ If synovial structure is draining → evidence of synovial fluid and variable lameness
➢ +/-Wound close to/overlying synovial structure
➢ Heat, pain and swelling (joint effusion/tendon sheath effusion)

34
Q

How should we do our CE in septic arthritis case?

A
  • Assess location & depth of wound
  • Clip hair to visualise
  • Effusion of synovial structures or evidence of cellulitis
  • synovial leakage
    -> prep aseptically & evaluate with sterile gloved finger
35
Q

If unlikely to be septic arthritis what to do?

A
  • Anti-microbials?
  • Tetanus?
  • Pain relief?-> ongoign admin avoided as it can mask worsening lameness indicating sepsis
36
Q

What is it is likely septic arthritis?

A
  • confirm with synoviocentesis (DONT do through cellulitic skin)
  • Sample collected & sent for: cytology, C & S
  • Pressure test -> distent joint with saline -> see communication
  • Contrast study
37
Q

What is abnormal synovial fluid like?

A

➢Serosanguinous/turbid/reduced viscosity
➢High white blood cells and total protein
➢>90 % Neutrophils

38
Q

Tx for septic arthritis?

A
  • Joint lavage (large vol polyionic solution)
  • Arthroscopy ‘Godl standard’ +/_ synovial biopsy?
  • Through and through needle lavage
  • Systemic ABs (broad but ideally base don C & S)
  • Intra-articular AB
  • Intravenous regional perfusion
  • Nsaid
39
Q

Prognosis for septic arthritis?

A
  • Early diagnosis and aggressive management achieve best outcome
  • Affected by concurrent damage – worsens prognosis
  • Osteomyelitis
  • Soft tissue damage
40
Q

When might septic arthritis caes not present as NWB lame?

A

if joint open and draining

41
Q

What is Synovitis? (Joint flare)

A

-Occurs after chemical tx/ anaesthesia (steroids, LA ..)
- Low incidence

42
Q

when does joint flare happen?

A

3-24h post injection

43
Q

Prevention, dx & tx fo joitn flare?

A
  • Prevention - NSAIDs given at time of injection and for 3 days after
  • Diagnosis – synoviocentesis
  • Treatment – rest, NSAIDs, cryotherapy +/- corticosteroids
44
Q

What to be careful with in tx of joint synovitis?

A

Don’t mask signs of septic arthritis

45
Q

Describe Acute tendinitis/ Desmitis

A
  • Most commonly sustained during intense exercise
  • Flexor tendons/suspensory ligaments of forelimbs
  • Uni- or bilateral
  • Re-injury of previously injured tendon
46
Q

Clinical Presentation of Acute Tendonotis? Desmitis?

A
  • Localised heat, swelling, pain
  • Lameness
  • Often no wound/external trauma
47
Q

Other ddx for desmitis/ tendonitis?

A

Fracture
Bruising /stone in shoe

48
Q

INITIAL tx for acute tendonitis/ Desmitis?

A
  • Cold therapy
  • Nsaids
  • Steroids iv?
  • Bandage limb -> reduce swelling & infl ; RJ bandage + dorsal splint for severe injuries
  • US exam
49
Q

What diagnostics might we do for acute tendonitis/desmitis?

A
  • Regional anaesthesia?
  • Radiographs
  • US
  • MRI
50
Q

What is the complete TX for tendonitis/ desmitis?

A
  • NSAIDs +/- other anti-inflammatories
  • Cryotherapy
  • Bandaging
  • Box Rest
  • Remedial farriery
  • Treatment plan – see Chronic Lameness lecture
  • Physiotherapy
  • Re-evaluation
  • Rehabilitation plan
51
Q

Why do we see lymphangitis & cellulitis?

A
  • Inflammation that causes swelling, pain +/- lameness
  • Most commonly in hindlimbs
  • Compromised immune system
52
Q

T/F Lymphamgitis may not respond to conservative tx?

A

FALSe - it usually does!

53
Q

What does conservative tx involve of lymphangitis?

A
  • Cryotherapy – cold hosing/ice boots
  • Hand walking or turnout in clean/dry area
  • Can lead to cellulitis
54
Q

Describe cellulitis

A
  • Bacterial infection
  • Usually only one hindlimb
  • Variable lameness, but can be severe
  • Affects dermis and subcutaneous tissues
  • Bacteria enter body through a break in skin
55
Q

Signs of cellulitis?

A
  • Heat, pain and lameness
  • Pyrexia
  • Pitting oedema
  • May get fluid leakage/lethargy/anorexia if severe
56
Q

Txfor lymphangitis & cellulitis?

A
  • NSAID’s
  • Antimicrobials (ab stewardship)
  • Cryotherpay
  • +/- box rest or some exercise as appropriate
57
Q

CLs of tendon laceration, rupture/ dispalcement

A
  • Lameness-
    Maybe not - extensor tendon
    Will be - flexor tendons & suspensory ligament
  • Distress-> SDFT luxation
  • Postural abnormality
58
Q

What postural abnormality for which injury?

59
Q

Draw it

60
Q

What to do in case of tendon/lig rupture

A
  • REFER
    -Admin of NSAId, AMs? +/- sedation +/- IV fluids
  • Apply dressing
  • Provide limb support
  • Immediate euthanasia?
61
Q

What limb support for what rupture?

A

➢ Flexor tendon – align dorsal cortices RJB plus
splint or cast bandage
➢ Extensor tendon – RJB

62
Q

CLS of Fractures?

A

➢ Acute, severe lameness
➢ Local heat, pain, swelling
➢ Limb instability
➢ Crepitus
➢ Reduced range of motion
➢ Resentment of flexion/extension
➢ Severe distress

63
Q

Initial tx for fract?

A
  • Control haemorrhage
  • +/- sedation
  • NSAID analgesia
  • +/- Antimicrobials and tetanus prophylaxis
  • IV fluids
    then
    Radiography -> Immobilise -> Transport
64
Q

What aims of immobilisation?

A
  • Prevent further injury
  • Assist weight bearing
  • Relieve anxiety
  • Counteract forces that will displace the fracture
  • Joint above and below fracture
65
Q

What is exertional rhabdom?

A

‘tying up’ -> rule out pelvic fracture / laminitis

66
Q

Dx of ER?

A
  • Blood sample –
    CK/AST/urea/creatinine
  • Urine sample - myoglobin
  • Muscle biopsy
67
Q

Tx for ER?

A

Initial treatment
* Fluid therapy
* NSAIDs – care
* ACP
* Vit E Selenium

Ongoing management
* Diet
* Avoid stress
* Avoid rest days/turn out
* Dantrolene