Dysrhythmias Flashcards

1
Q

normal electrical conductivity of heart

A
Rate 60-100
Regular rhythm
P waves: upright and round, one before every QRS, regular rhythm
PR interval 0.12-0.20 sec
QRS: <0.12 sec
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2
Q

sinus arrythmia

A

normal
variability in HR 60-100bpm
common in young people
HR fluctuates with respiration or Autonomic nervous system

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3
Q

what is a dysrhythmia

A

abnormality of cardiac rhythm

problems with impulse generation or conduction

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4
Q

what causes dysrhythmia’s?

A

inappropriate automaticity

triggered activity re-entry

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5
Q

what are the different types of sinus rhythms?

A
sinus rhythmmia
sinus arrhythmia
sinus tachycardia
sinus bradycardia
paroxysmal supraventricular tachycardia
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6
Q

sinus bradycardia

A

SA node
regular rate less than 60
normal rhythm
normal PR (.12-.20) and QRS interval (< 0.12)

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7
Q

causes of sinus brady

A
hyperkalemia
vagal response
digoxin toxicity
late hypoxia
meds
MI
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8
Q

manifestations of sinus brady

A
lightheadedness/ dizziness with exertion
easily tired
syncope or near syncope
dyspnea
chest pain/discomfort
confusion
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9
Q

treatment of symptomatic bradycardia

A

atropine: anticholenergic

if drug not effective: pacemaker

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10
Q

sinus tachycardia

A
SA node
HR: 100-150
regular rhythm
p wave similar or partially hidden
normal PR (.12-.20)interval and QRS (<0.12)
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11
Q

causes of tachycardia

A
catecholamines: (exercise, pain, strong emotions)
fever
fluid volume deficit
meds (epi, albuterol, beta agonists)
substances (caffeine, nicotine, cocaine)
hypoxia
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12
Q

treatment for sinus tachycardia

A

based on cause
hypovolemia= fluids
fever= anti-pyretic
pain= analgesics

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13
Q

paroxysmal supraventricular tachycardia (PSVT)

A
caused by re-entry phenomenon
begins and ends suddenly
"feel like my heart is racing"
HR 150-250
AV node
usually no P wave if present, abnormal
QRS looks normal (<0.12)
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14
Q

causes of PSVT

A
over exertion
emotional stress
stimulants
digitalis toxicity
rheumatic heart disease
CAD
Wolff- Parkinson-White
right sided heart failure ( Cor pulmonale)
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15
Q

clinical signs and symptoms of PSVT

A
palpitations
chest pain
fatigue
lightheaded/dizziness
dyspnea
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16
Q

premature atrial contractions (PACs)

A
early P waves that look different
normal PR interval (12-20)
QRS does follow PAC
usually not a problem but frequent PAC's cause pt high risk for dysrhythmia's
check electrolytes may need O2
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17
Q

two types of atrial dysrhythmias

A

atrial flutter

atrial fibrillation

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18
Q

atrial flutter

A
AV node, overrides SA node
re-entry impulse that is repetitive and cyclic
Atrial pulse Rate >250 bpm,
regular atrial rhythm
ventricular rate is slower
P was "SAWTOOTH"
QRS narrow
may be 2:1, 3:1, or 4:1 ( ex: 2 p wave per 1 QRS complex)
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19
Q

causes of atrial flutter

A
CHD
cardiomyopathy
heart valve disease
congenital heart disease
inflammation of heart (myocarditis)
high BP
overactive thyroid, lung disease
electrolytes
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20
Q

atrial fibrillation

A

multiple irritable spots in atria (all sending signals)
irregularly irregular (atrial and ventricular)
HR 100-175
no identifiable P wave
fibrillation waves

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21
Q

clinical manifestations of Atrial Fibrilation

A
palpitations
heart racing
fatigue
dizziness
chest discomfort
sob
may be asymptomatic
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22
Q

complications and causes of Afib

A
electrolytes
hypoxia
cardiovascular disease
decreased cardiac output
heart failure
embolus/ stroke
23
Q

untreated a-fib doubles your risk of heart-related ____, and causes a 4-5 times greater risk of ____

A

death, stroke

24
Q

treatment of Afib

A

beta blockers, CCB, digitalis, amiodarone
stroke prevention: anticoagulants, antiplatelets
ablation
cardioversion

25
types of ventricular dysrhytmias
PVC's VTACH VFIB
26
PVCs
contraction coming from ectopic focus in ventricles PVC's come earlier than QRS and doesn't follow normal rhythm or p-wave wide and distorted QRS ( >.12) no atrial contraction, no P wave
27
causes of PVC's
``` stimulants electrolytes hypoxia fever exercise emotional stress CVD ```
28
treatment of PVC's
treat cause
29
three sub-types of PVC's
bigeminy trigeminy quadrigeminy
30
VTACH
3 or more PVC's together seriously decreases cardiac output ectopic focus within the ventricles takes control and fires repeatedly, NO atrial contractions DEADLY RHYTHM
31
VTACH presentations
Rate: 150-200, usually regular | no p-wave evident, PR measurable
32
VTACH associated with
MI, CAD, significant electrolyte abnormalities, HF, drug toxicity
33
treatment of VTACH
ACLS depends on pulse, pt symptomatic very quickly unless converts back to rhythm may need anti-dysrhythmic (beta blocker or CCB) electrolyte replacement
34
VFIB
irregular wave forms of varying shapes and sizes ventricles are QUIVERING NO CARDIAC OUTPUT
35
cardiac muscle cells have
automacity excitability conductivity contractility
36
automacity
ability to generate electrical impulse
37
excitability
ability of cell to interpret impulse
38
conductivity
ability to receive and conduct signal
39
contractility
ability to shorten in respond to impulse. "squeezing"
40
depolarization
light switch turning on, occurs during systole causing squeezing
41
repolarization
makes cells ramp up
42
atrial depolarization
P wave
43
ventricular depolarization
QRS complex
44
ventricular repolarization
T wave
45
atrial repolarization
do not typically see
46
what does potassium-ATP pump cause
causes action potentials
47
SA node
stimulates atria to contract P wave atrial contraction
48
AV node
makes ventricular go off | After P wave and before QRS
49
perkinjie fibers
LT ventricular contractions
50
what causes dysrhythmias?
inappropriate automaticity triggered activity re-entry
51
inappropriate automaticity
a cell initiates action potential when its not supposed to
52
triggered activity
an extra impulse generated during or just after repolarization
53
re-entry
cardiac impulse in one part of the heart contributes to depolarization after main impulse has finished
54
wolfe-parkinson-white
precipitates PSVT, r/t other rhythm abnormalities