DOD Flashcards
Physis matures too rapidly
Contracted tendons
Physis becomes inflammed
Physistis
Physis has abnormal mechanical pressures
ALD
Epiphysis has abnormal ossification
OCD
What are some etiologic factors that can contribue to DOD?
- genetic predisposition/rapid growth
- high energy (carbs)
- high Ca and DE, high P
- trace minerals, Cu & Zn (excess or imbalance)
- endocrine imbalances (TH, Insulin)
- trauma
What is the most significant skeletal disorder of growing horses?
Osteochondrosis
- OCD
- SBC/SBL
*more common in certain jts, and certain sites within joints
- young horses, often fast growing
- jt effusion
- variable lameness
- often bilateral involvement
- Lateral or medial trochlear ridge
Clinical signs of OCD (stifle)
How can we treat OCD of the stifle?
- prevention:
- look at nutrition! - Sx
- debride/inject/pin lesion at arthoscopy, favorable prognosis - conservative
- rest, re-radiograph
How can we treat OCD of the Hock?
- prevention
- nutrition! - Sx
- debride
- favorable px - conservation
- rest, re-rad
- small lesions without effusion
What is Idiopathic Bog Spavin? Tx?
Tarsocrural effusion WITHOUT OCD
Tx: drain, inject, bandage
What are the clinical signs of SBC in stifle? Where do they occur
Minimal effusion and variable lameness
Almost always MEDIAL FEMORAL CONDYLE
Flexural Deformities (contracted tendons)
What forms are there? what jts are involved?
Congenital or acquired
Acquired is often assoc’d with rapid growth, and pain
Acquired forms usually involved DIP jt or fetlock jt
At what age are Contracted Tendons seen for DIP and MCP jts?
DIP = 4 weeks to 4 months
MCP= yearlings
Valgus vs Vargus
VaLgus = lateral
Vargus = medial
Congenital/perinatal causes of ALD
- Incomplete ossification
2. Ligament laxity
