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IBMS > diseases of oral mucosa > Flashcards

diseases of oral mucosa Flashcards

1
Q

draw the classification of oral mucosa lesions according to clinical appearance

A
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2
Q

what ulcers are associated iSight viral infections

A

multiple ulcers

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3
Q

what are white lesions usually NOT caused by

A

no viral or immunological fungal cause

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4
Q

what re infective and non infective lesions caused by

A

infective
- viral and fungi and bacteria
non infecitve- reaction/truama/ immunological,cancerous

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5
Q

descrive herpes simplex

A

multiple vesicles that burst in ucler
0 infective viral lesion
- most common virus
- coldsore on lips

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6
Q

describe the pathology of herpes simplex

A
  • Lymphtocyes invade oral epitehlium causing inflammation
  • intraepithelial vesicle formed of fluid
  • when vesicle reaches certain size it bursts to produce ulcer
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7
Q

how is herpes simplex diagnosed

A

Diagnosed by swab
Intracellular inclusion bodies- viral bodies present

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8
Q

Describe oral thrush

A
  • candida
    affect newly born children or old people -
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9
Q

who does oral thrush affect

A
  • newly born children or elderly people
  • immunosuppressant due to high dose of anitbitoaitcs
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10
Q

what are the clinical symptoms of oral thrush

A

tongue, throat , white lesion
when you remove it , it will leave a raw bleeding surface

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11
Q

describe the pathology of candida

A
  • candida hype penetrate eptiehul layer
  • intrapeitehial abscess formed due to degeneration of epitehoul layer due to infection
    infllamtry infiltration of lymphocytes underneath
  • disrupts plate and affect wall tissues
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12
Q

name 3 types of non infective ulcers

A

aphthous ulcer
traumatic ucler
squamous cell carnoam

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13
Q

what is the cause of recurrent authors ulcer

A

unknown cause, stress, Crohns disease, nutritional deficiencies such as vitamin B deficiencies

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14
Q

what is the cause of traumatic ulcers

A

aused by dentist ,LA, rare , heal within 10 days to 2 weeks- well defined but ragged border
Both have loss of surface epithelial and inflammation in lamina propria or connectivity tissues

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15
Q

what is the symptoms and cause of malignant ulcers

A

non healing ulcer , inflammation- underlying
Basement membrane is ruptured
epithelial islands that have grown into the connective tissue - sign of cancerous lesions

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16
Q

what non infective ulcer does this show

A

apthourse and traumatic ulcer
- loss of surface epithelial and inflammation in lamina propria

17
Q

what non infective ulcer does this image show

A

squamous cell carcinoma

18
Q

name some white an d red lesions

A
19
Q

what is dysplasia

A

away form normal shape and size

20
Q

what is hyperplasia

A

overgrowth of epithelium

21
Q

what is acanthosis -

A

growth of prickle cell layer

22
Q

what Is hyperkeratosis

A
  • whiteness - keratin block epithelial - overkertainisaiton - white leison
    Protective measure
23
Q

what feature is only present in white cell lesions

A

hyperkeratosis

24
Q

dsiitngusih between histopahtoglogy

A
25
Q

name 3 immunoligcla lesions

A
26
Q

describe lcihen plants

A

LICHEN PLANUS - t lymphocyte mediated inflammatory disease - CD4 and CD8 most common cells -
Lichenoid caused by drugs but not lichen planus -
65% females above 40
White strain on surface of mucosa
Small ulcers
Sometimes in form of plaque
To differentiate from others - the lichen planus happens laterally on buccal mucosa and on doral of tongue and rarely in gingiva
Respond clinically to corticosteroids
PATHOLOGICALLY THEY HAVE - very well known appearance
Rete pegs are narrowed in form of saw
Saw tooth tapered at end
Characterised by thin band of lymphocytes - t lymphocytes and CD8
White lesions characterised by hyperkeratosiss na dlymphocytic ifnirliatiosn
If lesion is atrophic - flattening epithelium and no rete pegs and destruction of basal cell layer and smaller band of infiltration aouion band
It is a premalignant lesion which can turn malignant

27
Q

describe pathology of lichen planus

A

PATHOLOGICALLY THEY HAVE - very well known appearance
Rete pegs are narrowed in form of saw
Saw tooth tapered at end
Characterised by thin band of lymphocytes - t lymphocytes and CD8
White lesions characterised by hyperkeratosiss na dlymphocytic ifnirliatiosn
If lesion is atrophic - flattening epithelium and no rete pegs and destruction of basal cell layer and smaller band of infiltration aouion band
It is a premalignant lesion which can turn malignant

28
Q
A
29
Q

describe systemic lupus

A

mmunologicla /autoimmune disease connected to RA or liver
Only skin and oral mucosa are affected
Similar to lichen planus
Orla lesions present in 20% of the cases
Preceded by other manifestations first
Differentiating factor
Unilateral
Lesions cna be white or red or ulceraitve
30% of cases sjdroegn syndrome is associated or developed - affects salivary glands
Irregular e;epithelium atrophy
Some inflammatory infiltrations

30
Q

describe the pathology of lupus

A

Thickening of BM, inflammatory infiltration band, degeneration of the epithelial cells

31
Q

describer erythema multiform

A

necrosis of prickle cell layer leading to intra epithelial bulla
- no Clea cruise
- 3-4 weeks with more lesion developing
- recurrent in month and years
- associated with ocular lesion - lead to loss of site

32
Q

list two immunological disorders

A
33
Q

describe the histopathology of pemphigus vulgars

A
  • Form vesicles that have clear fluid and bulla with clear vesicles -
  • Immunological - antibodies against substance
    Call quill detach from each other due to attack of intracellular semantic substance called acantholysis - prickle cell layer will loose connection with each other and form intraepithelial vesicles above the basal cell layer layer forming bulla
    Forging
    clefting above the basal cell layer forming intraepithelial vesicles that can lead to ulcers, acantholysis of the prickle cell layer ( loss of intercellular adherence), inflammatory infiltration in the underlying c.t.
    Binding antibodies to cells can be seen by fluorescence

IBMS (6 decks)

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