Dermatology Flashcards
Ulcer
Causes
Vascular (chronic venous insufficiency, arterial, mixed, vasculitis)
Mechanical (pressure, friction, shear, trauma)
Neuropathic Surgical, malignancy, infection
Chronic Venous Incompetence
Signs
Oedema Staining
Lipodermatosclerosis
Lower 1/3 of the leg
Painless
Irregular shape
Exudate
Inverted champagne bottle legs
Arterial ulcers
Clinical Presentation
Claudication, rest pain
Lower ABI
Weak/absent pulses
Sluggish capillary refill
Regular, punched out appearance
Below ankles to toes
Chronic Venous Incompetence
Treatment
Graduated compression bandages
Address factors that delay wound healing - nutrition, smoking, exercise, prolonged standing
Surgery
Arterial ulcers
Management
Improve blood flow through angioplasty, stenting, by-pass grafting
Often requires amputation
Neuropathic ulcers
Painless
Bony prominence or area of pressure
Good blood supply for healing
Ischaemic ulcers
Painful
Not essentially on pressure areas
Poor blood supply will negatively affect healing
Burns dressings
Hydrogels
Hydrocolloid dressings
Contraindicated on foot ulcers in patients with diabetes or PAD
Red scaly rashes DDx
FLAWED PINS
Fungal
LP
Acne
Warts
Eczema
Drug reactions
Psoriasis, P. Rosea
Infections
Neoplasia
Seborrhoea keratoses, solar keratoses, seborrhoeic dermatitis
Non-itchy rash DDx
Neoplasia
Erythema Multiforme (target lesions - drug reaction)
Pityriasis lichenoides chronica (hypersensitivity reaction to EBV or parvovirus)
Sarcoid
Acne
Impetigo (school sores - Staph/Strep)
Itchy rash (5/10) DDx
Acute - P. Rosea, Lupus
Chronic fluctuating - psoriasis, tinea, subaceous dermatitis
Chronic persistent - mycosis fungoides
Itchy rash (10/10) DDx
Eczema
Dermatitis Herpetiformis
Lichen Planus (autoimmune)
Scabies
Eczema
Vague border
Erythematous base
Puffy surface, scale, wet crusts, erosions, exudative lesions
Vesicles, papules
Contact dermatitis
Eczema
Irritant contact dermatitis
Allergic contact dermatitis
Scabies
Burrows - grey, C/S shaped, black dot at one end
Papules - scattered, red, small, monomorphous
Vesicles - infants, palmar, plantar, occular
Nodules - penile, buttocks, scrotum, axillae
Poor symmetry
Site - below chin line in adults, penis, nipple
Actinic keratoses
Hyperkeratotic adherent scale
Skin coloured, erythematous, brown
Rough
Distributed around maximally sun exposed sites
Bowen’s disease
Skin cancer
Evenly coloured, well demarcated, adherent scale
Distributed around sun exposed sites
Asymettrical
BCC
Skin cancer
Risk factors - fair skin, sunlight exposure, age >40, previous BCC
Common sites - head and neck, trunk, limbs
Scabies
Keratoacanthoma
Risk factors - male, >50yo, sun exposure, fair skin
Signs - rapid evolution nodule, central keratotic plug, firm fleshy, skin coloured or red, may resolve spontaenously
SCC Signs
Crusty, scaling, tender nodule
Inflamed, may bleed
Grows over weeks to months
Poorly defined
SCC Sites
Head and neck
Limbs
Trunk
Ephelides
Freckles
Solar lentigines
Hypermelanosis (incraesed melanocytes)
Fair skin, >60yo
Induced by UVR, phototherapy
Surface scaling usually absent. Usually multiple.
Reticulated pigment under dermatoscope
Seborrhoeic keratoses
Stuck on appearance
Lusterless
Dermatofibroma
Common benign fibrous skin lesion
May arise at site of minor trauma such as insect bite
Often on arms and legs.
Firm, asymptomatic and persistent
May be pink, yellow-brown or dark. “Button hole” sign if it is squeezed forming a dimple
Melanoma
Risk factors
UVL
Skin phenotype
Family history
History of non-melanoma skin cancer
Melanocytic naevi (1/3 associated)
Previous melanoma
Melanoma
Subtypes
Superficial spreading (70%)
Nodular (15%)
Acral lentiginous (10%)
Lentigo MM (5%)
Desmoplastic
Melanoma
Clinical features
ABCDE
Asymmetry
Border irregularity
Colour variegation
Diameter >6mm
Enlargement over months
Atopic eczema
Mutations of the filaggrin gene. Leads to dry skin caused by a decrease of fatty acids in the epidermis
Associated with asthma and hayfever, more common in Melbourne and winter (with heaters)
Family history of atopic eczema
Allergens only responsible in 10% - food in infants, house dust mite/pet fur in children/adults.
Most childhood cases improve with age but will always have “sensitive skin”. Advise against nursing, hair-dressing, mechanic
Eczema
Appearance
Red, dry, scaly, itchy
“Water colours”
Can blister, be lichenified, weep or be infected/crusted
Atopic Eczema
Clinical Features
Infant - face
Childhood/adulthood - flexural (cubital/popliteal fossa), neck, wrists, ankles, eyelids, nipples, hands
Infected Eczema
Organisms
Staphylococcus aureus (most common)
Herpes simplex virus
Seborrhoeic Dermatitis
Type of eczema “Butterfly rash” - scalp, top of eyebrows, top of nose and chest
Malassezia species involved