CVA Flashcards
FAST
Face Arms Speech Time
What is a stroke?
Supply of blood to brain is suddenly disrupted. Two types haemorrhagic (bleed in or around brain). Ischemic (blocked bf to the brain)
Signs & Symptoms of Stroke
Hemiparesis,mono paresis, or quadriparesis. Hemisensory deficits, visual field deficits, dysarthria, ataxia, facial droop, vertigo,aphasia, sudden decrease in level of consciousness
Sub arachnoid hemorrhage presentation
Sudden onset headache, nausea,vomiting, signs of meningitis (neck stiffness, photophobia) decrease level of consciousness.
Intracerebral hemorrhage presentations
More insidious, focal neurological signs, fluctuating levels of consciousness
Describe ischaemic stroke
Obstruction of an artery leading to or in the brain preventing oxygenated blood and nutrients from reaching parts of the brain that the artery feeds
Name the 2 the types of ischemic strokes
Large vessel thrombosis (most common, occurs in large arteries). Small vessel disease (lacunar infarction -deep brain infarct). Occurs when BF is blocked to a very small arterial vessel.
Embolic ischaemic stroke
A blood clot that forms in one area of the body and travels through the bloods stream where it may lodge. Can be fat globules, air bubbles, or bits and pieces of atherosclerotic plaque such as lipid debris that have detached from an artery wall or cardiac source.
Nurses role in stroke management
Neuro obs, assistance with mobility, falls prevention,info & support, do, trachy management, pt centred care, continence,skin integrity, pain control, incontinence, swallowing hydration
Ischemic stroke
Main mechanisms= thrombosis, embolism, and systemic hypo perfusion. The underlying mechanism can often be deduced by the size of the artery affected. Large arteries = carotid, vertebral and basilar. Small arteries =perforator arteries from MCA, verterbral and basilar - ischemia in these vessels give rise to lacunar strokes.
Borderzone Infarcts
Ischemic- sometimes known as watershed infarcts. Typically occur at boundaries of arterial territories. Occur due to hypo perfusion either following a period of systemi hypotension (cardiac arrest or intraoperative) or from complete occlusion of a large artery with minimal compensatory collateral flow
Primary and secondary prevention in TIA and stroke
Anti platelet meds -aspirin, copied ogres, or comb of aspirin and dipyrimade, anti coags- warfarin. BP management -ACE inhibitors in comb with diuretics. Lifestyle changes- diet and exercise. Cholesterol lowering therapy
Cerebral oedema
Common complication of large multi lobe strokes. Peaks 3-5 days and is only a problem in the first 24 hours in young stroke pts. Young people have no cerebral atrophy and thus no room to accommodate a swelling brain. Clinical signs= change in consciousness, worsening neurological deficits! new pupillary changes or changes in respiratory patterns .
Midline shift
Shift of the brain past it’s centre line. Commonly associated with a distortion in the brain stem, failure of the pupils to constrict in response to light. Often associated with a high ICP that can be deadly.
Herniation
Coning. When pt has a neurological injury or a space occupying mass within the skill (such as a tumour, swelling, excess CSF or bleed ) the pressure in the cranium may rise= brain tissue displaced to an area of low pressure. When the pressure increases the brain tissue herniates (get pushed down). This can cause alterations in the functions of neurons. Clinical symptoms = hemiplegia, dilated pupils and restlessness. If pressure persists brain may be pushed down through foramen ovale, which may lead to a basic loss of cardio respiratory function,