Burns Flashcards
Name the 6 categories used to classify burns
Fire, scalds, contact, chemical, electrical, and radiation.
Describe a superficial burn
Minor epithelial damage of the epidermis exists. Redness, tenderness & pain are common. No blisters. Heals after several days without scarring.
Describe a superficial partial thickness burn
Injury to the epidermis & superficial dermis. Ruptured weeping blistered which are erythematous and painful. Heals 1-3 weeks without scarring.
Describe deep partial thickness burns
Injury to the epidermis and deeper dermis with some viable dermis remaining. Heals spontaneously 3-4 weeks.
Describe full thickness burns
Injury to epidermis and entire dermis. Typically white, black or brown. Eschar is leathery and insensate. Does NOT heal spontaneously.
List and describe Jackson’s burn zones.
Coagulation - cenral zone. Dead or dying cells from coagulation necrosis and absent blood flow.
Stasis - intermediate zone. Appears to have intact circulation initially then circulation of superficial ceases and by third day zone becomes white as superficial dermis is avadcular and necrotic.
Hyperaemia - outer zone. Redzone, blanches on pressure. Healing present by 7th day.
How is the extent of a burn estimated?
Rule of nines. 9% - head and neck, 9% - each upper extremity, 18% - each anterior and posterior portion of trunk, 18% - each lower extremity and 1% - perineum and genitalia. Palm area = approx 1% TBSA.
What TBSA of a burn results in SIRS?
20% TBSA or more results in release of cytokines and other mediators into the systemic circulation causing SIRS.
Why does hypovolaemia occur in burns patients?
Vessels in burned tissues have increased vascular permeability which causes an extravasation of fluids into burned tissue.
Describe cardiovascular clinical manifestations in burns patients.
Fluid shifts, decreased cardiac output, increased systemic vascular resistance, hypotension, tachycardia, decreased pulses, prolonged capillary refill, myocardial depression.
Describe pulmonary clinical manifestations in burns patients.
Release of vasoconstrictive agents, decreased ability to oxygenate, airway oedema, singed nasal hairs, soot tinged sputum, change in voice, coughing, droolong, rapid or laboured breathing, crackles, stridor.
Describe renal clinical manifestations in burns patients.
Decreased glomerular filtrtion rate due to hypovolaemia and redistribution of blood flow to vital organs, oliguria, increased specific gravity, acute tubular necrosis.
Describe gastrointestinal clinical manifestations in burns patients.
Impaired gastric mobility and paralytic ileus due to inflammatory response, stress ulcers may result from stress response leading to Curling’s ulcer.
What is a Curling’s ulcer?
An acute peptic ulcer of the duodenum. Occurs when reduced plasma volume leads to ischaemia and cell necrosis of the gastric mucosa.
Describe the metabolic clinical manifestations in burns patients.
Increased secretion of catecholamines, cortisol, and glucagons to support tissue repair, hypermetabolism, protein wasting, weight loss.