cv leik Flashcards
incidental finding on CXR, may show widend mediastinam and obliteration of the aortic knob
thoracic aortic dissection
apical impulse
5th ics mid clavicular line left sternal border
LVH (severe) and cardiomyopathy can displace more than 3cm, it is larger and more prominent
3rd trimester preggers S3 during preggers is OK, located slightly up on the left side of the chest
de ox blood path
sean is a total victor passes prominantly
Superiror vena cava Inferior vena cava Atrium Tricuspid Vent Pulmonic Pulm artery
ox blood path
after my vistory aspireing always
pulm veins from lungs then
Atrium Mitral Vent Aortic valve Aorta
motivated apples
Mitral Aortic
Tricuspid pulmonic
AV valves semilunar
S1 systole Motivated
lub
closure of mitral and tricuspid valves
(AV valves have 3 leaflets
S2 diastole apples
dub
closure of aortic and pulmonic valves
two leafs
S3 sound
equals heart failure
early diastole also called ventricular gallup or
S3 gallup
kentuky
always abnormal if after 35 cept preggers, can be normal in kids
The third heart sound is caused by a sudden deceleration of blood flow into the left ventricle from the left atrium
s4 heart sound
increased resistance from stiff left ventricle usually indicates LVH
can be normal in some elderly
late diastole
atrial gallop
sounds like tennessee
BEST HEARD AT THE APEX OR APICAL AREA USING THE BELL OF THE STETHESCOPE
bell is for
low tones like extra heart sounds
mitral stenosis
diaphragm is for
mid to high pitich such as lung sounds
Mitral regurg
aortic stenosis
benign variants
split S2- PULMONIC AREA 2nd ICS appears at inspiration dissapears at experiation
s4 in elderly- if no S and S of heart or valve disease, it can be normal
Systolic murmurs
Mitral Regurg
Aortic Stenosis
Systolic murmurs
diastolic murmurs
Mitral
Stenosis
Aortic
Regurg
Diastole
mitral area
lower anatomically
apex, or apical area
5 ICS 8-9 cm form the midsternal line slightly medial to the midclavicular line
PMI or apical pulse is here
Aortic area
up top
2nd ACS at the BASE (anatomically higher)
S1 murmurs MR Ass
holosystolic, pan systolic, early mid or late systolic,
louder and radiate up to the neck
Mitral regurg
pan or holosystolic murmur
HEARD at the APEX 5th ICS
Radiates to axillia
LOUD BLOWING and high pithced (use diaphragm)
Aortic Stenosis
midsystolic ejection murmur
2nd ics R sternal border (careful with this one)
HARSH NOISY MURMUR, diaphragm
MS ard Diasotlic
diastolic murmurs are always bad
S2 MS-
low pitched- bell
heard best at apex 5th ics
also called “opening snap”
MS ARD
aortic regurg
high pitched diastolic
2nd ics RSB
high pitched blowing murmur - diaphragm
murmur grade
1
2
3
1 barely able to hear, can do only under p=optimal conditions
- mild to mod loud murmur
- loud murmur easily heard once the stethiscope is placed on chest
murmur grade
4
5
6
4 louder than three FIRST TIME THRILL IS PRESENT (think palpable murmur)
5 very loud murmur more obvious thrill, can hear with edge of scope off chest
- thrill is easily palpated, can hear with scope off chest
radiats to axila
mitral regurg
radiats to the neck
aortic stenosis
parox AF
terminates within 7 days but usually less than 24 hours, generally asymoptomatic
AF CC and treatment
fluttering heart, hypotension, pre or near syncopy,
tx is based on pt type and risk factors for stroke
CHADS VASC
0 is low 2 or more needs AC C-CHF H-HTN A age over 75 D- DM S- stroke or tia V- vascular disease A- 65-74 years S- female gender (higher risk)
AF labs after EKG
TSH, lytes, rena panel, BNP to R/O HF dig level if on digoxin 0.5-2 digibind for elevated
eccho to evaluate for valve problems that increase risk of stroke
if parox consider
24hr holter monitor
lifestyle for afib
avoid stims, caffeine, nicotine, decongestants and booze in some pts
AF - new
refer to cards
can cardiovert if stable in first 48 hours
or seek rate control
Rate control meds AF
BB
calcium channel blockers
Norvasc (amlodipine) Plendil (felodipine) DynaCirc (isradipine) Cardene (nicardipine) Procardia XL, Adalat (nifedipine) Cardizem, Dilacor, Tiazac, Diltia XL (diltiazem) Sular (Nisoldipine) Isoptin, Calan, Verelan, Covera-HS (verapamil)
or dig cardiac glycosides.
AF antiarythmics
amiodarone (cordarone) black box for pulmonary or liver damage, simvastatin with amio can cause rhabdo
Af anticoagulation
coumadin- vitamin K agonist only one reccomend for liver failure pt
baseline labs for coumadin
prothrombin time is 9.5-13.5 seconds.
INR below 1.1
aptt 30-40sec
platelet count 150,000 to 450,000
initial dose for coum-
5mg but frail or elderly above 70 take 2.5
full effect takes a few days 2-3
check INR- every 2-3 days untill theraputic for 2 consecutive checks
then weekly untill stable at between 2-3
then every 4 weeks when stable
for non vavlular AF consider
direct thrombin inibitors - xarelto, eliquis, no INR or diet restrictions
90% of warafin deaths are caused by
intracerebral hemmorhage
INR goal with a prostetic valve
2.5-3.5
INR less than 5 or 5-9 with no bleed
5-no vitamin K , sip next dose/adjust baseline dose, recheck once or twice a week while adjusting
5-9 hold one or 2 doses with 1 to 2.5mg of vitamin K monitor INR and lower maintnece dose of coum
WPW (more common in kids) delta wave Paroxsysmal SVT- peaked QRS with P waves CC and TX
like a-fib but with p waves, HR like 150 or 250
can be caused by dig tox, etoh, hyperthyroid, caffeine, drugs
EKG, if unstable may need cardioversion
vagal maneuvers
pulsus paradoxus
apical pulse can be heard but radial pulse cant be palpated, mesured with BP cuff and sterthoscope, imapired diastolic filling cause drop of sustolic pressure of more than 10mmhg
pulm cause- asthma or emphysema- increased positive pressure
cardiac- tamponade, pericarditis, effusion
anterior wall mi
Reciprocal ST segment depression in the inferior leads (II, III and aVF
st elevation in 3-4
BP measurement
manual is preferred- 2 or ore readings seperated by at least 2 mins should be averaged per visit
higher number determins BP stage
any change in PVR or CO has corrosponding effect on BP