Cumulative Final Flashcards
need a fungal cultures for tinea capitis or unguium because
hard to treat, make sure you have the right organism
atopic triad
Eczema, asthma and hay fever (allergic rhinitis)
lichen simplex chronicus
due to constant itching of atopic dermatitis. scaly, well demarcated, rough plaques with exaggerated skin lines
pityriasis rosea presentation and treatment
herald patch, then 2 weeks–>salmon colored macules in trunk and upper extremities in “christmas tree” pattern. NO TREATMENT NEEDED. its very itchy though so maybe topical steroids, po antihistamines
what causes psoriasis
inflammatory/autoimmune (genetics). keratin hyperplasia (epidermal thickening/dermis is continually turning over) due to T cell activation
what are you going to see for tinea versicolor on diagnostics (which diagnostic too)
KOH: “spaghetti and meatballs” hyphae and spores. ALSO yellow/green on wood’s lamp
treat tinea versicolor
selenium sulfide lotion, oral fluconazole–>dont shower need to sweat it out to get to skin.
seborrheic dermatitis occurs where
high sebaceous oversecretion–>scalp, face, eyebrows, body folds. fungal?
treat psoriasis
-NEVER USE SYSTEMIC CORTICOSTEROID -phototherapy, high potency topical corticosteroid + Vitamin D analogs
oval, fawn/salmon-colored, scaly plaques with collarette scale
Pityriasis Rosea
seborrheic dermatitis treatment
-scalp: zinc pyrithione.selenium shampoo -facial and intertriginous: hydrocortisone
what is this

erythema multiforme: classic target lesion 3 concentric zones of color change, found acrally on hands and feet
benign, pruritic, TENSE blisters in flexural areas
bullous pemphigoid
pruritic, VIOLACEOUS, flat-topped papules with fine white streaks; mucosal lacy lesions of buccal/vaginal mucosa; on flexural surfaces and trunk. starts at the WRIST
lichen planus, can develop into SCC
burning or stinging (CNS); erythematous, dilated vessels on cheeks—telangiectasias; papules/pustules possible
rosacea, mc 30-50. avoid the triggers and treat with topical metronidazole or clindamycin
rosacea vs ance?
neurovascular component of rosacea and comedones are in acne and absent in rosacea
actinic keratosis

- prolonged sun exposure
“sandpaper pink macules/papules”
- Pre-malignant–>SCC
- treat with 5-FU/cryotherapy
–>can also get hypertrophic
Basal cell vs SCC
Basal: pearly/waxy/arborizing vessels/telangestasias. mc, local infiltrating, doesnt met
SCC: more likely to met, often preceded by actinic keratosis

seborrheic keratosis

“stuck on” benign lesion of elderly people with sun exposure–velvety/warty
extensor (elbows and knees) vs flexor (antecubital and knee fossa)
psoriasis–>extensor and nail pitting
atopic dermatitis–>flexor, atopic triad
impetigo causative agents and treatment
s aureus, GAS
mupirocen
erysipelas caused and DOC

group A streptococcus
(edematous hot raised circumscribed red area on *cheeks or leg)
Penicillin! (GAS)
Edematous erythematous warm spreading plaque. chills, fever, malaise, lymphadenopathy
cellulitis. have to have a break in the skin (ex tinea pedis). treat with antibiotics 7-10 days. s aureus GAS common offenders.





























