CP Exam 3: CHF Flashcards

1
Q

What is congestive heart failure?

A

inability of heart to supply the organs and tissues of the body with sufficient amount of blood

most commonly leading to dyspnea and fatigue

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2
Q

What is the major concept behind CHF?

A

you have a decreased CO that is less than the normal of 4 ml/min

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3
Q

What is the more accepted AHA/ACC stages of CHF?

A

A. pt at high risk for CHF but no structural damage

B. structural damage but no sx

C. Structural damage and DOE like sx but controlled by meds

D. advanced disease requiring in hospital assistance, VAD or cardiac transplant

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4
Q

What is NYHA stages of CHF?

A

Stage 1: no fn. limitations or sx w/ normal activity

2: pt w/ slight sx with ordinary fnxl , comfortable at rest
3: marked limitation w/ any activity, only comfortable at rest
4: any activity brings discomfort and pt has sx at rest

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5
Q

What is the most common etiology of CHF?

A

MI/CAD

not common but can also be idiopathic

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6
Q

What is the basics of systolic CHF?

A

inability of the heart to squeeze out

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7
Q

What are the four areas that can cause systolic HF?

A
  1. preload- EDV and fiber length prior to contraction
  2. afterload- resistance of flow to ventricular ejection
  3. contractility- contractile strength of myocardium
  4. chronotropy- rate of contraction, ability to maintain HR
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8
Q

What is a hallmark characteristic of SHF?

A

a dilation or over stretching heart of the left ventricle leading to less contractile force (length tension curve)

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9
Q

What does this lead to?

A

this leads to increased EDV and poor contractility

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10
Q

What does EF have to be below to be considered SHF?

A

below 45%

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11
Q

As a result of increased EDV where does the back up blood go into?

A
  1. Lungs (w/left sided)- which decreases BP in systolic, Sx include-fine crackles, DOE, pink sputum, orthopnea
  2. Periphery- (right sided)- peripheral edema, jugular venous distension, acites
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12
Q

What else can SHF lead to?

A
  1. increased LV pressure- S3 sound

2. decreased resting and exercising SBP

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13
Q

What can also happen as a result to SHF that creates a vicious cycle?

A

baroreceptors at nephron sense low BP and activate RAAS which leads to more water retention, this will then increase preload further stretching the heart and worsening SHF

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14
Q

What is diastolic heart failure characterized by?

A

LV is stiff, decreased compliance of the ventricle

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15
Q

What happens as a result of a stiff LV?

A

inability of ventricle to relax and decrease diastole, decreases space for volume in LV

this leads to loss of SV and CO

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16
Q

What are the sx for DHF?

A

same as SHF but harder to diagnose

17
Q

What is EF with DHF?

A

EF is preserved b/c you are getting less volume in but you are getting less volume out so percentage stays the same

18
Q

What are characteristics of left CHF?

A

pressure backs up towards lungs, orthopnea, dyspnea, exercise intolerance, LVEF affected only with systolic

19
Q

In left sided CHF what happens with exercise?

A

CO may be flat or dropping with increasing activity

20
Q

What are characteristics of right CHF?

A

pressure backs up toward periphery, JVD, peripheral edema, ascities, dyspnea, same exercise tolerance, RVEF affected

21
Q

What is B-type Natriuretic peptide? BNP

A

amino acid that is leaked out of ventricles or atriums when muscle fibers are overstretched in both SHF and DHF

22
Q

What is the lowest and highest BNP normal values?

A

below 100 no HF, above 900 severe HF

need to be above 400-500 to have HF

23
Q

What is orthopnea?

A

due to back flow of blood and fluid into alveoli pt is unable to lay flat b/c laying flat causes increased pulm capillary pressure

measured in pillows

24
Q

Where will blood back up into first in RCHF?

A

juglar vein and liver, portal HTN

25
Q

Why can LCHF cause peripheral edema?

A

lack of blood flow will trigger receptors to activate RAAS and will lead to retained fluid

26
Q

What is a clinically significant number for weight gain?

A

more than 2 pounds in a day

27
Q

What is very important to remember about pts with peripheral edema in CHF?

A

do not massage this fluid, use meds and diuresis to relieve sx, also don’t elevate legs above heart

pitting edema measurements used

28
Q

How does MI/CAD cause CHF?

A

damage to myocardium impairs contractility, this and remolding leads to dilation of heart, leads to increased EDV and stretching

29
Q

How can uncontrolled HTN lead to CHF?

A

leads to LV hypertrophy, overtime the pressure leads to EDV, leading to poor contractility

30
Q

What type of left sided valve dysfunction causes SHF?

A
  1. aortic stenosis- similar to HTN
  2. aortic insufficiency- increased EDV due to back flow
  3. mitral valve insufficiency- inability to push EDV out efficiently
31
Q

What is tx of CHF?

A
  1. usually lifestyle mods- decrease salt intake, nutrition big factor- not making prepared foods
  2. meds- diuretics, ACE I, ARB, BB, digoxin
  3. PT
  4. surgical options
32
Q

How does the valsalva maneuver affect CHF?

A

when you take a deep breath in it increases intra thoracic pressure, this unloads left side of heart decreasing EDV resulting in a more favorable length tension curve leading to flat BP response

33
Q

How is this diff in pts with non CHF?

A

normally this would increase BP then reduce it

34
Q

How does CHF affect skeletal muscle?

A

due to consistent lack of blood flow it will decrease fiber diameter, number of mitochondria, cell acidosis

leading to impaired strength/endurance or deconditioning

35
Q

What is important to remember about interval training with CHF pts?

A

they may never progress out of this and that is okay

36
Q

Why do CHF need IMT?

A

even in normal MIP it can decrease DOE and help endurance

duration- 15-30 mins per day at intensity of 15-60% max MIP for 2-3 months