Corticomotor Flashcards

1
Q

What is the Origin of the corticospinal tract?

A

60% frontal lobe areas 4,6,8

40 % somatosnsory complex

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2
Q

Where does the corticospinal tract terminate?

A

Sensory in dorsal horn

Motor onto monosynaptic connections/ interneurons.

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3
Q

What is the general function of the cortico spinal tract?

A

Control of distal muscles as well as proximal flexors.

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4
Q

Where does the corticonuclear tract originate?

A

Originates in the primary motor cortex (4,6,8)

Sensory and motor to face.

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5
Q

Where does the rubrospinal tract originate?

A

Red nucleus

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6
Q

How would a lesion to the lateral corticospinal tract present?

A

No spasticity
Weakness of arm with slight hyperreflexia
Babinskis sign would be present

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7
Q

What broadmans area is correlated to the motor cortex?

A

Broadmans area 4

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8
Q

What are the functions of the motor cortex?

A

Controls muscles for movement.

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9
Q

What effect would a lesion to the motor cortex show?

A

Weakness (Paresis) NOT PARALYSIS
Spactisity can occur if other areas also damaged.
Persistant hypotonia
Can cause permanent deficit in hand control.

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10
Q

Which broadmans areas are associated with the premotor cortex?

A

Areas 6,8

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11
Q

What is the function of the premotor cortex?

A

Visual guided movement
Reaching/grasping
Mirror neurons here!

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12
Q

Lesions of premotor cortex?

A

Apraxia = Inability to initiate certain tasks.

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13
Q

What function does the supplementary motor area have?

A

Planning/preparing for motor acts

Coordinating posture and vol. movements.

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14
Q

What is seen in a lesion to the supplementary motor area?

A

Inability for complex 2 handed tasks.

Damage to puntamen in parkinsons can cause difficulty with initiating.

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15
Q

Which broadmans areas are associated with the posterior parietal cortex?

A

Broadmans areas 5,7

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16
Q

What is the function of the posterior parietal cortex?

A

Sends information from the sensory and motor cortices to the cerebellum via pontine nuclei.

17
Q

What occurs in posterior parietal cortex lesions?

A

Contralateral neglect syndrome (non dominant hemi-lesion)

Aprraxia (dominant hemi)

18
Q

What broadmans areas are related to the frontal eye fields?

A

Broadmans area 8

19
Q

Lesion to Frontal eye field

A

Pt cannot voluntarily direct eyes away from stimulus.

20
Q

An infarction to the anterior cerebral artery bilaterally would present in which way?

A

Paraplegia affecting lower extremities sparing the face and hands.
Incontinence
Abulic and motor aphasia.

Frontal lobe changes: Personality change and contralateral grip reflex.

21
Q

How will Brown Sequard syndrome resent?

A

Hemisection of the spinal cord:

1) Loss of ipsilateral motor control.
2) Loss of ipsilateral vibration, and proprioception.
3) Contralateral loss of pain and temp sensation 1-2 segments below lesion. (Spinothalamic)
4) Loss of all ipsilateral sensation with flaccid paralysis. at the level of the lesion.

22
Q

What is crossed paralysis?

A

Seen in some brainstem lesions. Lesion occurs before the motor neurons have crossed affecting the contralateral side.

23
Q

Which fibers would be affected in a lesion to the genu of the internal capsule?

A

Corticonuclear fibers.

24
Q

Which fibers would be affected in a lesion to the posterior limb of the internal capsule?

A

Corticospinal fibers

25
Q

What signs would be seen in an infarct to the lenticulostriate arteries?

A

Pure UMN motor signs contralateral to the lesion.