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Resp. Physio > Control of Breathing > Flashcards

Control of Breathing Flashcards

1
Q

بسم الله الرحمن الرحيم
عليك توكلنا يا رب المؤمنين يا حي يا قيوم توكلنا علي الحي الذي لايموت سبحانه وتعالي علوا كبيرا عما يصفون

control of respiration is ?

A

Nerual
chemical
nonchemical

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2
Q

Respiartory centers

A

in brain stem
___________________

Medullary:
Ventral respiratory group VRG
Dorsal respiratory group DRG

Pontine:
Pnumotaxic
Apneustic

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3
Q

Dorsal repiratory group of medullary cenetrs role ?

A

contain inspiratory neurons firing to the bodies of the respiratory inspiratory nerves

like body of nerve fibers of phrenic c345 in the spinal cord casuing its firing and contraction of diapharagm
Relaxation occur when the firing of the DRG inspiratoy neuron cease

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4
Q

VRG is inder overirde control of the DRG?

A

In case of deep inspiration DRG stimulate VRG inspiratory neurons > deep inspiratoin active

in case of expiration DRG Stimulate VRG expiratory neurons > active expiration

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5
Q

Pre Botzinger complex

A

Neurons induce selft ryhtmic action optentials affecting The DRG and VRG

It is the cause of rhytmic stimulaiton of inspiratory neurons of DRG
RESPONSIBLE FOR RHYTHYM OF Ventilation

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6
Q

Injury of DRG and VRG not abolish respiartion

A

As neurons of Pre Botzinger Complex Cause ? rhymic stimulation of phrenic nerve of diapharagm

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7
Q

Pnemotaxic ?>

A

Pneumotaxix and vagus inhibits the apnestic and the DRG

causing limited Inspiration and not prolonged :-

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8
Q

In case of
pnemotaxix injury
vagal injury

pnemotaxic and vagal injury?

A

1- prolonged inspiration
2-also
3-prolonged inspiration and less epxiration apneusis casue apnustic center take the lead

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9
Q

Hering-Bruerer reflex ?

A

Strtching of lung during inspiration as in exercise casue the stopping inspiration to prevent over inflation

Stretch of lung > stretch receptor > vagal impulses inhibiting the DRG > stop inspiration and prevent over inflation of the lung

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10
Q

chemoreceptore location

A

central medulla

peripheral : Aortic bodies and carotid bodies in the bifurcation of aortic arch common carotid

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11
Q

when arterial blood pressure below 60 mmHg?

A

peripheral chemorececptors stimulated stimulating the medullary respiartory centre causing ?
increased ventilations as comesnation

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12
Q

does anemia and CO poisoning affect chemoreceptor?

A

No as its only respond to low PO2 not the content of O2

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13
Q

the most important regulator of ventilation durin rest?

A

PCO2

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14
Q

pco2 as a regulator?

A

central mainly by increased H+ induced by high Pco2 and this leads to stimulation of the medullary resp centre

peripheral chemoreceprs weak sstimulation add effect

in highest levels leads above 70-80 depression of the respiratory centre alos the brain as a whole
decreased ventilation

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15
Q

Chronic lung disease

A

low o2 stimulate peipheral
High o2 stmiulate centre
net effect increase of the ventilation

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16
Q

Renal compensation in casr of acidosis

A

high reuptake of HCO3 - CROSSING BBB netralizing the H+ normal srimulation of central chemoreceptoir

17
Q

Exercises and diabeta metabolic acidosis leads to ?

A

high H+ stimulation of peripheral chemoreceptor only as it not cross BBB
so increased respiratory centre stimulation and ventilatopn expiring c02 reutung ph to normal

18
Q

Vomintig and reparatd alkalosis in repsiartion contorl “/

A

decrease H+ DEPRSSING THE PERPHERAL CHEMO AND THE RESPIATORT INCERASING CO2 AND H+ TO NORMAL

19
Q

DIRECT INHIBTION OF CENTRAL CHEMORECEPTOR ?

A

PO2 < 60

PCO2 < 70-80

20
Q

HYPOXIC DIRIVE IN ?

A

HIgh altitude

Chroinc lung disesase low senestivity to co2

high level pco2 narcosis

21
Q

non chemical regulation of respiartiopn

A

Higher centers?
CL
Cortex
limbic sytesm hypothalamus

Lung
HCB

Hering berure
Cough and sneezing reflex
Pulmonary chemoreflex

others? 
MAG
-Mechanoreceptors
-Artertial barorecptors
-Gut
22
Q

Why breaking pint occur during voluntary apnea?

A

This is due to stimulation of central chemoreceptors override the cereberal cortex voulunary inhinotiry effect

23
Q

prolong the duration of v apnea?

A

hyperventilation befroe

100 o2 breathing

24
Q

Limbic sytem and hypothalamus ?

A

Strong emotions and pain increase symp in hypothalamus

and the high temp inc the stimulation of resp centres

25
Q

Pulmonary chemo reflex

A

J receptor stimulation during inflation of lung during edema and congestion leads to apnea followed by reapid brathing bradycardia hypoteniosin DUE TO

(Capsacian )intake in hot chili peppers

26
Q

Coughing

Sneezing

A

Forced expiration againts a closed glottis

Forced expiration againt contiunously oepend glottis

27
Q

Abrupt increase in ventilation durin exercise due to ?

A

Pshuchi stimuli

Proprioceptors

28
Q

Gradual increase in ventilation during exercise due to ?

A 
Body temperature
K level and peripheral chemorecpetors
Fluctations of O2 and CO2
Sensitviy of respriatory centre to CO2
Sympathatic stimulation increased ventialtion
29
Q

The main reporatory stimulant during Vigorus exercise ?

A

Lactic acidosis due to anaerobic glycolysis

as the PO2 increased and PCO2 decreased During hyperventilation

30
Q

Ventilation is stimulated after exercise by?

A

Lactic acidemia

31
Q

O2 used during muscle recovery in ?

A
  • myoglobin saturation
  • atp and creatinine
  • removal of lactic acid in 80 > glycogen 20 > h2o and co2
32
Q

Mountain skciKness comp

A

Headace, fatigubility ,drowisness
Nausea ,Vomiting, Loss of appetite
Tachycardia ,tachypnea

33
Q

Compensatory mechanisms for mountain sickness

A

High ventilation and pulmnary flow and surface area of diffusion
increased erythropoeten and RBCS
2,3DPG Right shift and O2 release
increased capillaries at tissue levels
Corrected alkalosis by decresad excrestion of H and increasd OF HCO3

34
Q

ايه مشاكل الوسائل التعويضية عند المرتفعات ؟

A

انها بتزود لزوجة الدم لزيادة كرات الدم الحمراء فيزادا شغل القلب عشان يعرف يضخ كويس

35
Q

تم بحمد الله

الحمدلله الذي خلق السماوات والأرض وجعل الظلمات والنور ثم الذين كفروا بربهم يعدلون

A

Resp. Physio (3 decks)

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