Congestive Heart Failure Flashcards

1
Q

What is heart failure?

A

inability of the heart to maintain its function of pumping blood to the lungs and the rest of the body

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2
Q

What medical conditions increase afterload?

A

On left side: HTN, aortic stenosis, aortic coarctation. On right side: pulmonary embolism

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3
Q

What is cardiac arrest/asystole?

A

cessation of normal circulation of the blood due to failure of the heart to contract effectively. brain injury is likely if longer than 5 minutes.

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4
Q

How does failure of the compensatory mechanism of the Frank-Starling Law of the heart explain the pathophysiology of heart failure?

A

the ventricle is loaded with blood to the point where heart muscle contraction becomes less efficient due to reduced ability to cross-link actin and myosin filaments in over-stretched heart muscle

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5
Q

What is reduced space capacity?

A

loss of cardiac reserve or the ability of the heart to work harder during physical activity since it can no longer meet normal O2 demands.

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6
Q

How does increased HR lead to left sided heart failure?

A

Initially, this helps compensate by maintaining BP. This places further strain on the myocardium, increasing coronary perfusion requirements, which can lead to worsening of ischemic heart disease

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7
Q

How does hypertrophy lead to left sided heart failure?

A

terminally differentiated heart muscle fibres increase in size to improve contractility. This contributes to the increased stiffness and decreased ability to relax during diastole

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8
Q

How does enlargement of the ventricle lead to left sided heart failure?

A

contributes to the spherical shape of the failing heart. The increase in ventricular volume also causes a reduction in stroke volume due to mechanical and contractile inefficiency.

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9
Q

What might you find on physical exam of right sided heart failure?

A

pitting peripheral edema, ascites, hepatomegaly, increased JVP, and parasternal heave

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10
Q

What is the jugular venous pressure?

A

Best estimate of right artrium pressure and right side function. Identify highest point of pulsation and measure from highest point vertically to the sternal angle. Abnormal if > 3 cm

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11
Q

What happens as a result of backward failure of left-side failure?

A

tachypnea, rales/crackles w/pulmonary edema (fluid in alveoli), cyanosis in severe pulmonary edema, orthopnea, paroxysmal nocturnal dyspnea, cardiac asthma

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12
Q

What is the most common mechanism of damage that leads to systolic dysfunction?

A

ischemia causing infarction and scar formation. After myocardial infarction, dead myocytes are replaced by scar tissue, deleteriously affecting the function of the myocardium

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13
Q

Describe the New York Heart Association Classification of heart failure

A

Class I: no limitation and no symptoms from ordinary activities. Class II: slight, mild limitation of activity and sx. Class III: marked limitation of any activity; the patient is comfortable only at rest. Class IV: symptoms occur at rest.

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14
Q

Describe the American College of Cardiology/American Heart Association classification of heart failure

A

Stage A: Patients at high risk for developing HF in the future. Stage B: a structural heart disorder but no symptoms at any stage. Stage C: previous or current symptoms of heart failure w/an underlying structural heart problem, managed with medical treatment. Stage D: advanced disease

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15
Q

What stage of heart failure are beta blockers contraindicated?

A

stage IV

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16
Q

What is immediate treatment for acute decompensation?

A

ABC. combination of vasodilators such as nitroglycerin, diuretics such as furosemide, and possibly non invasive positive pressure ventilation (NIPPV).

17
Q

What causes diastolic dysfunction?

A

failure of the ventricle to adequately relax and typically denotes a stiffer ventricular wall. causes inadequate filling of the ventricle, decreased stroke volume, and elevated end diastolic pressures. 4th heart sound

18
Q

What is important to control with diastolic dysfunction?

A

Adequate rate control (slow down AV conduction). sudden bouts of tachycardia may result in flash pulmonary edema