Conduction blocks Flashcards

1
Q

What is first degree heart block

A

occurs where there is delayed atrioventricular conduction through the AV node

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

ECG for first degree heart block

A

pr interval is greater than 0.2 seconds
5 small or 1 big square

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is second degree heart block

A

where some of the atrial impulses do not make it through the AV node to the ventricles. This means that there are instances where p waves do not lead to QRS complexes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

names of the three types of second degree heart block

A

Mobitz1
Mobitz2
2:1 BLOCK

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What happens in mobitz type 1

A
  • atrial impulses becomes gradually weaker until it does not pass through the AV node.
  • After failing to stimulate a ventricular contraction the atrial impulse returns to being strong
  • cycle repeats
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

ecg for mobitz 1

A
  • increasing PR interval until the P wave no longer conducts to ventricles
  • This culminates in absent QRS complex after a P wave.
  • This cycle repeats itself.
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

what occurs in mobitz type 2

A
  • intermitted failure or interruption of AV conduction
  • Normal pr intervals
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

ecg for mobitz type 2

A
  • pr intervals are the same
  • missing qrs complexes
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what is 2:1 block

A

2 p waves for each qrs complex , every second p wave is not a strong enough atrial pressure to stimulate a QRS complex

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

what is third degree heart block

A
  • COMPLETE HEART BLOCK
  • no observable relationship between p waves and QRS complexes
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

treatment for stable av node blocks

A

observation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

treatment for unstable or risk of asystole

A
  • first line- atropine iv 500mcg
  • no improvement:
  • atropine repeated up to 6 doses
  • other inotropes
  • transcutaneous cardiac pacing
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What occurs in left bundle branch block

A

right ventricle contracts before the left

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

define bundle branch block

A

a block in the conduction of one of the bundle branches , so the ventricles don’t receive impulses at the same time

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

describe normal cardiac conduction

A
  • The sino-atrial node acts as the initial pacemaker
  • Depolarisation reaches the atrioventricular node
  • Impulses travel simultaneously down the bundle of His via the left and right bundle branches. The septum is depolarised from the left.
  • Both the left and right ventricular walls are depolarised simultaneously
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

diagnostic criteria for RBBB

A
  • QRS- > 120 ms
  • RSR pattern in V1-V3
  • Wide , slurred S wave in lateral leads
17
Q

Explain MaRroW for RBBB

A

Complexes in V1 resemble M and V6 resembles W

18
Q

Describe the steps in right bundle branch block

A
  • The sino-atrial node acts as the initial pacemaker
  • Depolarisation reaches the atrioventricular node
  • Depolarisation through the bundle of His occurs only via the left bundle branch. The left branch still depolarises the septum as normal.
  • The left ventricular wall depolarises as normal.
  • The right ventricular walls are eventually depolarised by the left bundle branch, this occurs by a slower, less efficient pathway.
19
Q

Diagnostics for LBBB are

A
  • QRS duration > 120ms (3 small squares)
  • Dominant S wave in V1
  • Broad, monophasic R wave in lateral leads – I, aVL, V5-V6
  • Absence of Q waves in lateral leads
  • Prolonged R wave > 60ms in leads V5-V6
20
Q

WiLliaM mnemonic for LBBB

A

Complexes in v1 resemble W
Complexes in v6 RESEMBLE m

21
Q

Why might LBBB occur

A
  • conduction system degeneration
  • myocardial pathologies
  • after cardiac procedures
  • STEMI - rare
22
Q

What does the left bundle branch split into

A

anterior and posterior fasicles

23
Q

anterior fasicle block may cause ….?

A

left axis deviation

24
Q

posterior fasicle deviation causes …. r

A

right axis deviation

25
A systolic murmur occurs ...
after S1
26
A diastolic murmur occurs ....
after s2
27
name the two systolic murmurs ASMR
Aortic stenosis mitral regurgitation
28
name the two diastolic murmurs ARMS
Aortic regurgiation mitral stenosis
29
What can cause first degree heart block
- High vagal tone (e.g. athletes) - Acute inferior MI - Electrolyte abnormalities (e.g. hyperkalaemia) - Drugs: NHP-CCBs, beta-blockers, digoxin, cholinesterase inhibitors
30
Causes of mobitz type 2
- Infarction: particularly anterior MI which damages the bundle branches - Surgery: mitral valve repair or septal ablation - Inflammatory/autoimmune: rheumatic heart disease, SLE, systemic sclerosis, myocarditis - Fibrosis: Lenegre's disease - Infiltration: sarcoidosis, haemochromatosis, amyloidosis - Medication: beta-blockers, calcium channel blockers, Digoxin,
31
Definitive management of mobitz type 2
Permanent pacemaker
32
Clinical features of third degree heart block
Patients may present with syncope or cardiac arrest. ECG shows severe bradycardia and dissociation between the P waves and the QRS complexes.