Concussion Flashcards
Primary predominant mechanism of concussions
Shear forces caused by rotational acceleration
Average concussive head impact - linear acceleration value
62.4 +/- 29.7 g force
Physiology - what happens after acceleration/deceleration injury occurs
Nerve (axon) is deformed (shearing and stretching motion)
Neurotransmitters are released and influx of ions - what happens after
binding of glutamate to excitatory amino acids receptor
What does calcium do during concussions recovery?
Impairs mitochondria and ATP production
Calcium accumulation can directly activate pathways leading to cell death
The metabolite normalization process after a concussive episode is an _______, rather _____ in the first 2 weeks, and ____ between 22 and 30 days
exponential phenomenon
slow
faster
What is a second impact syndrome?
Condition in which a person experiences a second head injury before the complete recovery of the initial injury
Cerebral herniation
Rapid pressure develop and deterioration and leading to death within 2-5 minutes
Normalization of NAA concentrations(in other words, ATP stores) may occur only after what
after the cerebral energy state has fully recovered
what preventive method is more important that neck strength?
Ability to actively engage neck muscles and resist linear and rotational acceleration is more important
Is the mouthguard a good prevention method?
Conflicting results
Is the helmet a good prevention method?
May reduce concussion severity and duration of symptoms as well as SRC odds if direct head impact
How do we define persisting post-concussive symptoms?
Symptoms lasting 2 weeks or longer for adults and 4 weeks or longer for children
Is there a gold standard for diagnosis of concussions?
NOPE, rely on symptoms report
Risk factors for concussion outcomes (protracted recovery)
- history of concussions
- history of migraines
- ADD/ADHD
- females
- younger
What acute symptom should signal a red flag and a possible longer recovery in our ax?
Dizziness
Does the SCOAT6 include blood pressure measurement?
YES
What are the 6 clinical trajectories of concussions?
Vestibular
Ocular-motor
Cognitive/fatigue
Post-traumatic migraine
Cervical
Anxiety/mood
S/S of cognitive/fatigue trajectory
fatigue
decreased energy levels
non-specific H/A
sleep disturbances
Common to see increase of symptoms at end of the day
Difficulties concentrating
Increase headache with cognitive activity
Cognitive/fatigue trajectory
What is the physiology behind the cognitive/fatigue trajectory?
Energy crisis because of the lack of ATP related to the mitochondria
Testing shows deficit in what, for cognitive/fatigue
Memory
Processing speed
Reaction time
S/S - vestibular trajectory
Dizziness, fogginess, nausea, and feeling of being detached, anxiety, and overstimulation in more complex environments
Increase in sx in busier, more stimulating environments
Rapid head or body mvt may increase sx
Vestibular trajectory
Testing shows deficit in what, for vestibular
Vestibular and ocular motor screening
S/S - ocular motor trajectory
localize, frontally based H/A, fatigue, distractibility, difficulties with visually based classes, pressure behind eyes, and difficulty with focus
Difficulties with extended time in front of computer screens or reading
Full days of work/school may increase sx vs decrease on weekends
Ocular motor trajectory