Communication Disorders Flashcards

1
Q

what does intact communication rely upon

A

Interaction between
Language
Cognition
Speech

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2
Q

which hemisphere is dominant for language

A

Everyone will say left cerebral hemisphere is dominant for language
That said, recent research suggests both hemispheres play some role

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3
Q

language definition

A

The ability to understand and produce conventional code of symbols and the rules for using them
Note: production includes speaking, writing, signing/gesturing

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4
Q

is aphasia a language or speech disorder

A

Language, because they can affect any or all components of language – and speech is only one component of language

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5
Q

broca’s aphasia

A

Non-fluent expressive aphasia

Lesion would be in the posterior inferior left frontal lobe

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6
Q

wernicke’s aphasia

A

Fluent receptive aphasia

Lesion would be in the posterior superior left temporal lobe

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7
Q

global aphasia

A

Non-fluent

Lesion would be in the left MCA territory of the frontal, temporal, AND parietal lobes

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8
Q

conduction aphasia

A

Fluent
Lesion would be in the left superior temporal region with incomplete damage to Wernicke’s area and the inferior parietal lobe especially the supramarginal gyrus

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9
Q

artery that supplies all of the areas that produce aphasia

A

Middle cerebral artery (MCA)

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10
Q

special role of the angular gyrus

A

This is a region at the junction of the temporal, parietal, and occipital lobes important for processes involved in reading and writing

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11
Q

cognitive linguistic impairments in patients with brain injury/demential/cog disorders

A
Disorganization 
Disorientation
Attention deficits 
Amnesia 
Impaired judgement
Decreased processing speed 
Deficits in problem solving 
Reduced word fluency 
Impaired naming 
Impaired reading 
Verbal paraphasia – the substitution of a word with a nonword that preserves at least half of the segments and/or number of syllables of the intended word
Anomia – inability to recall names of everyday objects 
Echolalia – meaningless repetition of another person’s spoken words 
Decreased speech rate 
Decreased comprehension
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12
Q

non-linguistic communication impairments in patients with brain injury/demential/cog disorders

A

Gestures
Facial expressions
Body language

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13
Q

speech apraxia

A

Impairment of the capacity to PLAN sensorimotor commands for the positioning and movement of muscles for the volitional production of speech
Can occur without significant weakness or neuromuscular slowness, and in the absence of disturbances of conscious thought or language

Characterized by:
Difficulty with volitional movement of articulators
Difficulty sequencing the sounds in syllables in words
Difficulty imitating speech sounds and movements
Inconsistent errors
Slow rate of speech
Production of automatic speech
Oral groping

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14
Q

2 most likely lesion locations that cause speech apraxia

A

Supplementary area – medial

Premotor – lateral

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15
Q

dysarthria

A

Impairment of movement due to abnormal neuromuscular execution that may effect the speed, strength, range, timing, or accuracy of speech

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16
Q

does dysarthria impair comprehension

A

There are no comprehension problems – this is purely a motor speech production problem

Therefore, this is a speech disorder not a language disorder

17
Q

localization/pathophysiology that may produce spastic UMN dysarthria

A

corticobulbar lesion

18
Q

localization/pathophysiology that may produce hypokinetic dysarthria

A

lesion in basal ganglia circuit – seen with Parkinson’s

19
Q

localization/pathophysiology that may produce hyperkinetic dysarthria

A

lesion in basal ganglia circuit – seen in Huntington’s

20
Q

localization/pathophysiology that may produce ataxic dysarthria

A

lesion in cerebellar control circuit in the spinocerebellum

21
Q

localization/pathophysiology that may produce flaccid LMN dysarthria

A

lesion to CN VII, IX, X, XII or their nuclei