Colorectal surgery: Small Bowel Obstruction Flashcards

1
Q

Describe a way of classifying bowel obstructions [3]
Name three suggestions for the each of the above [9]

A

Extramural: block bowel from outside
- Adhesions (congenital or aquired)
- Hernia
- Volvulus (caecal; sigmoid; small bowel)
- Compression from lymph nodes

Intramural: blockage from within the wall
- Tumours (adenocarcinoma, GISTs, lymphoma, leiomyosarcoma)
- Strictures bowel has narrowed due to: diverticular, ischaemic, IBD, post op,
- Intussusecption (wall of bowel moves into itself & blocks itself, most commonly at terminal ileum and caecum)

Intraluminal: within the wall
- Gall stones
- Bezoar
- Foreign body
- Meconium

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2
Q

Where does most of bowel obstruction occur? [1]
Why is this clinically significant? [1]

A

Small bowel: 75%; more likely to cause vomiting (& pain - colicky)

Large bowel: 25%: more likely to cause distension and absolute constipation (& pain - colicky)

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3
Q

What is the imaging that is the investigation of choice for bowel obstruction? [1]
State other imaging used [1]

A

CT: imaging of choice;
XR

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4
Q

What is gastrograffin AXR? [1]
When is its use indicated? [1]

A

Gastrograffin:
* Water soluble contrast for small bowel adhesive obstruction.
* If gastrograffin has passed into colon then suggests that will resolve (if not then surgery is indicated)

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5
Q

The “big three” causes account for around 90% of cases of bowel obstruction.

What are they? [3]
Where are they found (small or large bowel) [3]

A

Adhesions: small bowel
Hernias: small bowel
Malignancy: large bowel

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6
Q

Describe the pathophysiology of small bowel obstruction [5]

A
  • Passage of food, fluids and gas, through the intestines becomes blocked
  • Obstruction results in a build up of gas and faecal matter proximal to the obstruction
  • This causes back-pressure: resulting in vomiting and dilatation of the intestines proximal to the obstruction.
  • When there is an obstruction, and fluid cannot reach the colon, it cannot be reabsorbed: as a result, there is fluid loss from the intravascular space into the gastrointestinal tract leading to hypovolaemia & shock
  • The higher up the intestine the obstruction, the greater the fluid losses as there is less bowel over which the fluid can be reabsorbed.
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7
Q

How does bowel obstruction lead to bowel ischaemia, infarction, necrosis, and perforation? [3]

A
  • With time, bowel wall oedema forms
  • This compresses the intestinal veins and lymphatics, reducing the venous drainage of the bowel
  • As this occurs, it compresses intestinal arterioles and capillaries
  • This prevents arterial perfusion to the bowel wall.
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8
Q

What is the most common cause of SBO? [1]

A

Adhesions

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9
Q

What’s a pneumonic for remembering the causes of small bowel obstruction?

A

HANG IVs”

Hernias 2%
Adhesions (from previous surgery with formation of intra abdominal adhesions, commonly colorectal and gynaecological surgery)
Neoplasms (malignant, benign, primary or secondary) (5%)
Gallstone ileus
Intussusception
Volvulus
Strictures (eg Crohn’s disease (6%), ischaemia)

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10
Q

State 4 causes of intestinal adhesions that could contribute to the formation of bowel obstruction [4]

A

Abdominal or pelvic surgery (particularly open surgery)
Peritonitis
Abdominal or pelvic infections (e.g., pelvic inflammatory disease)
Endometriosis

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11
Q

What specifically leads to the colicky pain experienced in SBO? [2]

A

dilation of the intestine
AND
the continuing intestinal peristalsis

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12
Q

Describe the difference in presentation of more proximal and more distal bowel obstructions [2]

A

In more proximal bowel obstructions:
- patients tend to present earlier
- abdominal pain and vomiting are the predominant symptoms.

In distal small bowel obstructions:
patients usually present after 2-3 days of abdominal pain.
- The predominant symptoms are abdominal distension and constipation.

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13
Q

Describe bowel sounds of SBO [3]

A
  • Initially high pitched (tinkling)
  • Increased frequency
  • In delayed presentations, bowel sounds can be reduced due to secondary ileus
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14
Q

Describe presentation of SBO [4]

A
  • Absence of passing flatus (90%) or stool (80%)
  • Abdominal pain (90%): Ranges in severity; Often cramping in nature; Intermittent every 3-4 minutes.
  • Nausea and vomiting (80%)
  • May be bilious
  • Abdominal distension (65%)
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15
Q

Describe the relationship between abdominal pain and vomiting in SBO? [1]

A
  • Abdominal pain often precedes vomiting (constant pain may indicate bowel ischaemia)
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16
Q

Describe the nature of vomit of SBO [1]

A

green bilious vomiting

17
Q

Explain what VBG readings would you suspect with a patient with bowel obstruction? [2]

A

Metabolic alkalosis due to vomiting stomach acid

18
Q

What size small bowel would indicate it is dilated? [1]

A

greater than 3 cm

19
Q

How can you tell from the AXR if the small bowel is involved? [2]

A

Valvulae conniventes
Normally more central

20
Q

Describe the non-operative treatment for SBO [3]

A

Drip and Suck:

  • Nil by mouth
  • IV fluids to hydrate the patient and correct electrolyte imbalances
  • NG tube with free drainage to allow stomach contents to freely drain and reduce the risk of vomiting and aspiration
21
Q

What are the indications for surgery for SBO? [2]

A

Bowel compromise (e.g. ischaemia, perforation, necrosis), generally occurring in complete bowel obstructions

Surgically correctable causes (e.g. volvulus, incarcerated hernia, gallstone ileus, foreign body ingestion, tumour)

22
Q

How would you surgically treat SBO? [4]

A
  • Exploratory surgery in patients with an unclear underlying cause
  • Adhesiolysis to treat adhesions
  • Hernia repair
  • Emergency resection of the obstructing tumour
23
Q

What are 4 key emergency risks of SBO? [4]

A

Hypovolaemic shock due to fluid stuck in the bowel rather than the intravascular space (third-spacing)
Bowel ischaemia
Bowel perforation
Sepsis

24
Q

If surgery is indicated for SBO, patients should be given antibiotic prophylaxis of which antibiotics? [3]

A

cefoxitin, or ampicillin plus gentamicin