Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

EPI PQ > Clostridium > Flashcards

Clostridium Flashcards

1
Q

Most clostridia have low invasive capacity

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Spores of clostridia are generally very resistant against heat

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

The habitat of clostridia is the gut and the soil

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Clostridia are obligate aerobic bacteria

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

Clostridium perfringens is an obligate pathogenic bacterium

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

Clostridium perfringens can produce main and auxillary toxins

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

Extracellular enzymes and toxins are virulence factors of clostridia

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

There are no vaccines for the prevention of diseases caused by clostridia

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

There are no vaccines for the prevention of diseases caused by clostridia

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Clostridium bacteria is not in the environment, because it cannot tolerate oxygen

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Clostridium spreads usually rapid in a herd

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Clostridium spread mostly with insecticides

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Clostridium difficile can be treated with metronidazole

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Clostridium difficile is seen in foal and piglets

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Many Clostridium species have flagella

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Clostridium species are only found in the subtropics

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Clostridium can cause severe contagious diseases

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Clostridium are obligate pathogens

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Anaculture or anatoxin vaccines are used for the prevention of malignant oedema

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Cl. chauvoei is the agent of malignant oedema

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Lesions of malignant oedema are mainly seen in the large muscles

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Malignant oedema is generally a consequence of wound infection

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Malignant oedema is generally a consequence of wound infection

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Movement difficulties are frequently seen in the case of malignant oedema

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Clostridium novyi can cause malignant oedema

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
26
Q

Malignant oedema can be diagnosed based on clinical signs

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
27
Q

Malignant oedema is caused due to wound infection

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
28
Q

Malignant oedema is only in ruminants

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
29
Q

Malignant oedema, one of the clinical signs is lameness/movement problems

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
30
Q

Malignant oedema, attenuated vaccine for prevention

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
31
Q

Clostridium channel is the agent of malignant oedema

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
32
Q

Malignant oedema is generally a consequence of a wound infection

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
33
Q

Clostridium histolyticum can cause malignant oedema

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
34
Q

Clostridium histolyticum can cause malignant oedema

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
35
Q

Clostridium histolyticum can cause malignant oedema

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
36
Q

Agents of malignant oedema can be detected by bacterium culture

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
37
Q

There are no vaccines for the prevention of malignant oedema.

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
38
Q

Malignant oedema occurs in ruminants and pigs

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
39
Q

Malignant oedema is an acute fatal disease

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
40
Q

Malignant oedema can be treated with antibiotics

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
41
Q

Malignant oedema can occur in any warm-blooded animal.

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
42
Q

Once an area is infected with gas gangrene re-occurrence is common

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
43
Q

Malignant oedema cannot occur in swine

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
44
Q

Malignant oedema usually develop following an endogenous infection

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
45
Q

Malignant oedema is well treated with long-term antibiotics therapy

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
46
Q

Malignant oedema can be treated with polymyxin

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
47
Q

Malignant oedema can be well treated with antibiotics over a long period

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
48
Q

Is gas gangrene (malignant oedema) a regional illness

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
49
Q

The lesions of malignant oedema are mainly seen in the lungs

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
50
Q

Blackleg is caused by Clostridium septicum

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
51
Q

Lesions of blackleg are mainly seen on the claws

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
52
Q

Lameness is a clinical sign of blackleg

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
53
Q

Blackleg is a frequent disease in pigs

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
54
Q

Generally attenuated vaccines are used for the prevention of blackleg

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
55
Q

Anaculture or anatoxin vaccines are used for the prevention of blackleg

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
56
Q

Blackleg occurs only in tropical and subtropical countries

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
57
Q

Generally attenuated vaccines are used for the prevention of blackleg

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
58
Q

Blackleg generally occurs in endemic form

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
59
Q

Blackleg occurs most frequently in pigs

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
60
Q

Blackleg is a gas gangrene disease

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
61
Q

Blackleg is generally endogenous in sheep

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
62
Q

Blackleg is generally endogenous in cattle

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
63
Q

Movement disorders and lameness can be clinical signs of Blackleg

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
64
Q

Clostridium chauvoei can produce acids and gas from carbohydrates

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
65
Q

Blackleg occurs mainly in ruminants

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
66
Q

Oedema is a typical clinical sign of blackleg

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
67
Q

Live vaccines are used for the prevention of blackleg

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
68
Q

Blackleg infects ovine through wounds

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
69
Q

In Blackleg disease we use attenuated vaccine

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
70
Q

In the case of sheep, blackleg is generally consequence of a wound infection

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
71
Q

Oedema in the muscles is a typical clinical sign of blackleg

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
72
Q

Attenuated vaccines are used for the prevention of blackleg

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
73
Q

Blackleg is caused by Clostridium chauvoei

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
74
Q

Severe diarrhoea is the main clinical sign of blackleg

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
75
Q

Blackleg is caused by Clostridium septicum

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
76
Q

Blackleg occurs in cattle and sheep.

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
77
Q

Blackleg occurs in cattle and sheep.

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
78
Q

Attenuated vaccines are used for the prevention of blackleg

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
79
Q

Blackleg disease occurs only in ruminants

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
80
Q

Blackleg can usually be treated with antibiotics successfully

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
81
Q

Blackleg in cattle is mainly endogenous between 6 months-3 years old

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
82
Q

The disease caused by Clostridium chauvoei occurs mainly in cattle and sheep

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
83
Q

The disease caused by Clostridium chauvoei is primarily the result of endogenous infection
in cattle.

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
84
Q

Blackleg has four toxins

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
85
Q

Blackleg can be prevented by using vaccine

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
86
Q

We use neomycin and polymyxin to treat disease caused by Clostridium chauvoei

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
87
Q

Blackleg in cattle is mainly endogenous between 2 months-2 years old

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
88
Q

Blackleg in bovine is caused by wound infections

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
89
Q

Blackleg in bovine is caused by wound infections

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
90
Q

Oedema in the wall of the abomasum and duodenum are postmortem lesions of bradsot

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
91
Q

Bradsot is caused by Clostridium chauvoei

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
92
Q

Bradsot is caused by Clostridium chauvoei

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
93
Q

Soil contaminated frozen feed is a frequent predisposing factor of bradsot

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
94
Q

Frozen food is a predisposing factor of bradsot

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
95
Q

Bradsot occurs mainly late autumn and winter

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
96
Q

Overeating can predispose the animals to bradsot

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
97
Q

Thickening of and oedema in the stomach wall are typical lesions of bradsot

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
98
Q

Aminoglycosides are successfully used for treatment in the case of bradsot

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
99
Q

Bradsot is mainly seen in late autumn and winter

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
100
Q

Bradsot is mainly seen in late autumn and winter

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
101
Q

Severe pneumonia is a typical clinical sign of bradsot

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
102
Q

Bradsot has a very fast course

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
103
Q

Bradsot occurs only in suckling lambs

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
104
Q

Bradsot is typically a chronic disease

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
105
Q

Bradsot is common in the summer out on the pasture

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
106
Q

Bradsot is an acute disease resulting in sudden death in many cases

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
107
Q

We can use anaculture strain vaccine against Bradsot

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
108
Q

Bradsot causes oedema of the legs and necrosis

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
109
Q

Post mortem lesions of bradsot can be seen in the stomach (rennet)

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
110
Q

Koves disease is an indicator disease

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
111
Q

CSF is a predisposing factor of koves disease

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
112
Q

Koves disease can be seen in pigs

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
113
Q

Koves disease is caused by Clostridium chavoei

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
114
Q

Infectious necrotic hepatitis is mainly seen in pigs

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
115
Q

Infectious necrotic hepatitis can be prevented by using anatoxin vaccines

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
116
Q

Liver fluke can predispose animals to infectious necrotic hepatitis

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
117
Q

In sheep, Clostridium septicum causes necrotic liver infection

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
118
Q

Infectious necrotic hepatitis causes inflammation and necrotic nodules in the liver

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
119
Q

There is no vaccine to prevent infectious necrotic hepatitis

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
120
Q

Infectious necrotic hepatitis is caused by Clostridium septicum

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
121
Q

Infectious necrotic hepatitis is mainly seen in suckling lambs

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
122
Q

Parasite infection is a frequent predisposing effect of infectious necrotic hepatitis

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
123
Q

Parasite infection is a frequent predisposing effect of infectious necrotic hepatitis

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
124
Q

Parasite infection is a frequent predisposing effect of infectious necrotic hepatitis

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
125
Q

Infectious necrotic hepatitis is caused by Clostridium septicum

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
126
Q

Infectious necrotic hepatitis is caused by Clostridium novyi

A

T

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
127
Q

Infectious necrotic hepatitis is spread by tick

A

F

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
128
Q

Infectious necrotic hepatitis is caused by Clostridium novyi type B

A

T

129
Q

Infectious necrotic hepatitis is caused by Clostridium novyi type B

A

T

130
Q

Infectious necrotic hepatitis can be transmitted by liver flukes

A

F

131
Q

Infectious necrotic hepatitis occurs mostly in young sheep

A

F

132
Q

There is intravascular haemolysis in the case of bacillary haemoglobinuria

A

T

133
Q

Bacillary haemoglobinuria is caused by Clostridium haemolyticum

A

T

134
Q

There are no vaccines for the prevention of bacillary hemoglobinuria

A

F

135
Q

Phospholipase C is a virulence factor of the agent of bacillary hemoglobinuria

A

T

136
Q

Bacillary haemoglobinuria is mainly seen in cattle

A

T

137
Q

Bacillary haemoglobinuria is caused by Clostridium septicum

A

F

138
Q

Jaundice and anaemia are important clinical signs of bacillary hemoglobinuria

A

T

139
Q

Red urine is a typical clinical sign of bacillary hemoglobinuria

A

T

140
Q

Bacillary hemoglobinuria is a slow, chronic disease

A

F

141
Q

Bacillary hemoglobinuria can frequently be seen in horses

A

F

142
Q

Clostridium novyi is the causative agent of bacillary hemoglobinuria.

A

F

143
Q

Bacillary hemoglobinuria causes severe haemorrhages

A

T

144
Q

Bacillary hemoglobinuria are caused by infection from the soil

A

T

145
Q

Lamb dysentery occurs in a week old animal

A

T

146
Q

Isolation of the agent from the gut gives aetiologic diagnosis of lamb dysentery

A

F

147
Q

Isolation of Cl. perfringens from the gut confirms the diagnosis of lamb dysentery

A

F

148
Q

Lesions of lamb dysentery are generally seen in the large intestine

A

F

149
Q

Lesions of lamb dysentery can be seen in the small intestine

A

T

150
Q

Lamb dysentery is caused by Clostridium perfringens

A

T

151
Q

Lambs have to be vaccinated with anatoxin vaccine in order to prevent lamb dysentery

A

F

152
Q

Lambs have to be vaccinated with attenuated vaccine in order to prevent lamb dysentery

A

F

153
Q

Pregnant ewes have to be vaccinated in order to prevent lamb dysentery

A

T

154
Q

Haemorrhagic diarrhoea is a clinical sign of lamb dysentery

A

T

155
Q

Lamb dysentery can be seen in lambs around weaning

A

F

156
Q

Lamb dysentery is found in 3-4-week-old lambs

A

F

157
Q

Pathological lesions of Lamb dysentery starts in the colon

A

F

158
Q

We can culture the pathogen of Lamb dysentery from the intestines

A

T

159
Q

Lamb dysentery is caused by Clostridium dysenteriae

A

F

160
Q

Lamb dysentery can be seen in lambs after weaning

A

F

161
Q

There is no vaccine for the prevention of lamb dysentery

A

F

162
Q

Lamb dysentery occurs in 2-6 weeks old lambs

A

F

163
Q

For diagnosis of lamb dysentery, the pathogen should be cultured from the intestine

A

T

164
Q

Pathological symptoms of lamb dysentery can be found in the large intestines

A

F

165
Q

Lamb dysentery can be prevented by vaccinating pregnant ewes

A

T

166
Q

Lamb dysentery can be successfully treated with penicillin when clinical signs appear

A

F

167
Q

Lamb dysentery occurs in a week-old animal.

A

T

168
Q

Lamb dysentery can be diagnosed by culturing the bacteria

A

T

169
Q

Newborn lambs have to be vaccinated in order to prevent lamb dysentery.

A

F

170
Q

Toxoid vaccines can be used in the prevention of the disease

A

T

171
Q

Infection of lamb by secretion in the milk

A

F

172
Q

Lamb dysentery occurs in 1-2 weeks old lambs.

A

T

173
Q

Struck is caused by Clostridium perfringens

A

T

174
Q

Overeating is a predisposing factor of struck

A

T

175
Q

Struck can be seen mainly in lambs younger than 2 weeks

A

F

176
Q

Struck is an acute disease in horses

A

F

177
Q

Struck is a zoonotic disease

A

F

178
Q

Struck is a slow disease of older sheep

A

F

179
Q

Struck is a worldwide common disease with great economic impact

A

F

180
Q

Infectious necrotic enteritis of piglets occurs in the first 1-2 weeks of life

A

T

181
Q

The lesions of Infectious necrotic enteritis of piglets can be seen typically in the large
intestine

A

F

182
Q

Maternal protection is important in the case of Infectious necrotic enteritis of piglets

A

T

183
Q

There is no vaccination for the prevention of Infectious necrotic enteritis of piglets

A

F

184
Q

Pig enterotoxaemia can be prevented by vaccinating the pregnant sows.

A

T

185
Q

Pig enterotoxaemia is caused by Clostridium perfringens C

A

T

186
Q

Pig enterotoxaemia is caused by Clostridium perfringens C

A

F

187
Q

Lesions of pig enterotoxaemia can be seen in the small intestine

A

T

188
Q

Lesions of pig enterotoxaemia can be seen in the large intestine

A

F

189
Q

Lesions of pig enterotoxaemia can be seen in the large intestine

A

T

190
Q

Lesions of pig enterotoxaemia can be seen in the large intestine

A

T

191
Q

Pig enterotoxaemia can be generally seen in weaned piglets

A

F

192
Q

Necrosis of gut epithelium is a postmortem lesion of pig enterotoxaemia

A

T

193
Q

Clostridium enterotoxaemia of piglets is caused by C. perfringens

A

T

194
Q

Clostridium enterotoxaemia of piglets is more frequent in the case of first farrowing Sows

A

T

195
Q

Clostridium perfringens C causes infectious necrotic enteritis of piglets

A

T

196
Q

Infectious necrotic enteritis of piglets occurs in piglets after weaning

A

F

197
Q

The lesions of infectious necrotic enteritis of piglets can be seen generally in the small
intestine

A

T

198
Q

Infectious necrotic enteritis of piglets can be prevented by vaccinating the pregnant sows

A

T

199
Q

Necrotic enteritis of piglets is seen in piglets around weaning

A

F

200
Q

Necrotic enteritis of piglets can be prevented by vaccination the sow with anatoxin

A

T

201
Q

Pig enterotoxaemia has to be diagnosed by detecting antibodies in the piglets

A

F

202
Q

Pig enterotoxaemia causes abdominal contractions in sows

A

F

203
Q

Mesenteric lymph node is congested in case of pig enterotoxaemia

A

F

204
Q

Clostridium enterotoxaemia can be cultured from mesenteric lymph nodes or gut

A

T

205
Q

Enteritis in piglets are caused by Clostridium perfringens D

A

F

206
Q

Enteritis in piglets can be avoided by anatoxin vaccination

A

T

207
Q

Enteritis in piglets cannot be diagnosed by post-mortem, only by bacteriology

A

F

208
Q

Pig enterotoxaemia is caused by β-toxin production in 1st week of life

A

T

209
Q

Pig enterotoxaemia can cause a high mortality

A

T

210
Q

Necrotic enteritis of piglets cannot be diagnosed by isolating the agent from the gut

A

F

211
Q

Enterotoxaemia is mainly seen in piglets after weaning

A

F

212
Q

Pig enterotoxaemia is not present in Europe

A

F

213
Q

Pig enterotoxaemia cannot be prevented by using vaccines

A

F

214
Q

Pulpy kidney disease is caused by Clostridium perf. D

A

T

215
Q

Overeating is a predisposing factor to pulpy kidney disease

A

T

216
Q

The toxin of the agent of pulpy kidney disease is sensitive to trypsin

A

F

217
Q

Pulpy kidney disease is caused by Clostridium perfringens D

A

T

218
Q

Pulpy Kidney Diseases is caused by Clostridium chauvoei.

A

F

219
Q

Pulpy kidney disease generally occurs in 1-2week old lambs

A

F

220
Q

Pulpy kidney disease can occur at any age

A

F

221
Q

Pulpy kidney disease of suckling lambs can be prevented by vaccinating pregnant ewes

A

F

222
Q

Sudden change the diet is a predisposing factor to pulpy kidney disease

A

T

223
Q

The toxin damages the endothelial cells in the case of pulpy kidney disease

A

T

224
Q

Neurological signs are typical in the case of pulpy kidney disease

A

T

225
Q

Isolation of the agent is necessary to the diagnosis of pulpy kidney disease

A

F

226
Q

Pulpy kidney disease is typically seen in lambs below 2 weeks of age

A

F

227
Q

Inactivated vaccines are used for the prevention of pulpy kidney disease

A

T

228
Q

Pulpy kidney disease is seen in piglets in the first week of life

A

F

229
Q

Pulpy kidney disease is a worldwide common disease.

A

T

230
Q

Enterotoxaemia of sheep is also called pulpy kidney disease.

A

T

231
Q

Pulpy kidney disease is caused by Clostridium perfringens D.

A

T

232
Q

Cattle are not susceptible to this disease

A

T

233
Q

Vaccination are possible against pulpy kidney disease

A

T

234
Q

Coccidiosis is a predisposing factor of ulcerative enteritis in poultry

A

T

235
Q

Ulcerative enteritis of chicken is caused by Clostridium colinum.Ulcerative enteritis of chicken is caused by Clostridium colinum.

A

T

236
Q

Ulcerative enteritis is frequently seen in day old chicken

A

F

237
Q

Ulcers sometimes covered with pseudomembranes are frequent post mortem lesions of
ulcerative enteritis of chicken

A

T

238
Q

Ulcerative enteritis can occur in 4-12-week-old chickens

A

T

239
Q

Clostridium perfringens is the causative agent of ulcerative enteritis in poultry

A

F

240
Q

Ulcerative enteritis of poultry is generally prevented with vaccination.

A

F

241
Q

Lesions of ulcerative enteritis are mostly seen in the small intestines

A

F

242
Q

Lesions of ulcerative enteritis are mostly seen in the small intestines

A

T

243
Q

Prevention of coccidiosis can help lower the incidence of ulcerative enteritis

A

T

244
Q

Coccidiosis is a predisposing factor of necrotic enteritis of chicken

A

T

245
Q

Foamy, brownish-red faeces is a clinical sign of necrotic enteritis of chicken

A

T

246
Q

Lesions of necrotic enteritis of chicken are typically occur in the large intestine

A

F

247
Q

Day-old chickens are widely vaccinated in order to prevent of necrotic enteritis

A

F

248
Q

Necrotic enteritis mostly occurs in chicken

A

T

249
Q

Waterfowl are not susceptible to necrotic enteritis

A

F

250
Q

Necrotic enteritis occurs in 1-3 weeks of age

A

F

251
Q

Tyzzer’s disease is caused by Clostridium piliforme

A

T

252
Q

Gangrenous dermatitis is caused by Clostridium septicum and Clostridium perfringens A

A

T

253
Q

Gangrenous dermatitis is caused by obligate pathogens

A

F

254
Q

Gangrenous dermatitis causes muscle oedema

A

T

255
Q

Vaccines are the primary way of prevention of gangrenous dermatitis

A

F

256
Q

Flaccid paralysis is a frequent clinical sign of tetanus

A

F

257
Q

The agent of tetanus is strictly anaerobic

A

T

258
Q

The agent of tetanus can enter the host through wounds

A

T

259
Q

Tetanus is only seen in horse

A

F

260
Q

Over-eating can predispose animals to Tetanus

A

F

261
Q

The agent of Tetanus needs oxygen to replicate

A

F

262
Q

Anatoxin vaccines are available for the prevention of tetanus

A

T

263
Q

Haemorrhages under the serous membranes and enlargement of parenchymal organs are
typical postmortem lesions of tetanus

A

F

264
Q

Spasms are typical clinical signs of tetanus

A

T

265
Q

Tetanus is a zoonosis

A

F

266
Q

Toxoid vaccines can be used for the prevention of tetanus.

A

T

267
Q

Dogs are resistant to tetanus

A

F

268
Q

The clinical signs of tetanus are inducible

A

T

269
Q

Tetanus toxin cleaves synaptobrevin

A

T

270
Q

For tetanus we use vaccines which contain toxoid

A

T

271
Q

Tetanus cannot be prevented with vaccination.

A

F

272
Q

Tetanus is caused by Clostridium tetani

A

T

273
Q

The agent of tetanus causes septicaemia.

A

F

274
Q

Tetanus can be diagnosed on the basis of post mortem lesions

A

F

275
Q

Clostridium tetani produced endotoxin

A

F

276
Q

C. tetani needs anaerobic conditions for propagation

A

T

277
Q

Dogs are susceptible to tetanus

A

T

278
Q

Tetanus can be prevented with vaccines containing inactivated bacteria

A

F

279
Q

Tetanus can cause spasms

A

T

280
Q

Horses are resistant to tetanus

A

F

281
Q

Tetanus can only develop after deep wounds

A

F

282
Q

Wounds can predispose to tetanus

A

T

283
Q

The paralysis usually starts at the place of the wound

A

F

284
Q

Clostridium tetani toxin is produced in the feed

A

F

285
Q

Horses are most sensitive to tetanus

A

T

286
Q

Tetanus can be prevented by anatoxin vaccination

A

T

287
Q

Tetanus causes rigid paralysis

A

T

288
Q

There is no vaccine for tetanus

A

F

289
Q

Dogs have high resistance to tetanus

A

T

290
Q

Dogs have high resistance to tetanus

A

T

291
Q

The toxin of clostridium botulinum causes flaccid paralysis

A

T

292
Q

Clostridium botulinum generally causes wound infection

A

F

293
Q

Focal necrosis in the liver is a typical post mortem lesion of Botulism

A

F

294
Q

The toxin of Clostridium botulinum has irreversible effect

A

T

295
Q

Botulism can be seen as a result of a wound infection

A

F

296
Q

Flaccid paralysis is the main clinical sign of botulism

A

T

297
Q

Birds are resistant to botulism

A

F

298
Q

Necrotic foci in the liver are typical post mortem lesions of botulism

A

F

299
Q

Generally wounds predispose animals to botulism

A

F

300
Q

The agent of botulism generally produces toxin at the site of entry

A

F

301
Q

Botulism is diagnosed on the basis of the typical post mortem lesions

A

F

302
Q

Clostridium botulinum can produce toxins outside the hosts.

A

T

303
Q

No characteristic post mortem lesions can be seen in the case of botulism

A

T

304
Q

Botulism doesn’t occur in Europe

A

F

305
Q

Clostridium botulinum cannot tolerate air at all

A

T

306
Q

Botulism usually develops following a wound infection

A

F

307
Q

Clostridium botulinum propagates in rotten materials.

A

T

308
Q

In Hungary, botulism is seen most commonly in birds

A

T

309
Q

In Hungary, botulism is seen most commonly in birds

A

T

310
Q

In Hungary, botulism occurs in winter and early spring.

A

F

311
Q

Botulism is eradicated in Europe

A

F

312
Q

Clostridium botulinum can produce toxin, some of which are activated by proteases

A

T

313
Q

Botulism is seen mainly during summer

A

T

314
Q

Spasms are the typical clinical sign of botulism

A

F

315
Q

Paralysis is the main sign of botulism

A

T

316
Q

Toxins of botulism are produced generally in the food.

A

T

317
Q

Botulism happen generally through wound infection

A

F

318
Q

Animals are mostly sensitive to C and D types of Clostridium botulinum.

A

T

EPI PQ (37 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2024 Bold Learning Solutions. Terms and Conditions