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EPI HARDCORE > Clostridia hardcore > Flashcards

Clostridia hardcore Flashcards

1
Q

Clostridium perfringens can produce main and auxillary toxins

A

T

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2
Q

Clostridium difficile can be treated with metronidazole

A

T
Enterotoxaemia of young animals foals and piglets

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3
Q

Many Clostridium species have flagella

A

FOnly clostridium dificcile has peritrichous flagellaeje giafto troi metronitazoli :)

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4
Q

Lesions of malignant oedema are mainly seen in the large muscles

A

T

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5
Q

Malignant oedema is only in ruminants

A

Fmainly horses also mammals and birds

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6
Q

Malignant oedema occurs in ruminants and pigs

A

TMammals and birds

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7
Q

Malignant oedema is an acute fatal disease

A

TAcute for sure 1-2days ip fatal yes because a lot of pathology

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8
Q

Malignant oedema can be treated with antibiotics

A

Ftoo late

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9
Q

Malignant oedema can occur in any warm-blooded animal

A

F

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10
Q

Once an area is infected with gas gangrene re-occurrence is common

A

T

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11
Q

Malignant oedema is well treated with long-term antibiotics therapy

A

F

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12
Q

gas gangrene (malignant oedema) is a regional illness

A

FWorldwide

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13
Q

Blackleg generally occurs in endemic

A

TEndemic regions

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14
Q

Blackleg is generally endogenous in sheep

A

FSwce

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15
Q

If antibiotics are applied after appearance of the clinical signs of blackleg, treatment is generally successful

A

F

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16
Q

Blackleg disease occurs only in ruminants

A

FRarely other species

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17
Q

Blackleg can usually be treated with antibiotics successfully

A

F

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18
Q

Blackleg in cattle is mainly endogenous between 6 months-3 years old

A

F
2m-2y

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19
Q

Blackleg in cattle is mainly endogenous between 2 months-2 years old

A

Tswce

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20
Q

Blackleg in bovine is caused by wound infections

A

F

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21
Q

Oedema in the wall of the abomasum and duodenum are postmortem lesions of bradsot

A

Trennet = abomasum

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22
Q

Bradsot occurs mainly in tropical and subtropical countries

A

Fnorth europe kriada frozen potato and turnip

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23
Q

Köves disease is an indicator disease

A

TIndicator of a primary disease theat caused ulceration haemorrhages on the git.

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24
Q

Infectious necrotic hepatitis is found worldwide

A

T

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25
Q

Infectious necrotic hepatitis occurs mostly in young sheep

A

F
Mainly sheep also cattle1-4 years old? is this young

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26
Q

Bacillary haemoglobinuria is mainly seen in cattle

A

TBlack disease SCBac haemo CS

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27
Q

Bacillary haemoglobinuria is a slow, chronic disease

A

Fsudden onset all histolytic clostridia and (enterotoxaemic clostridia?)

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28
Q

Bacillary hemoglobinuria causes severe haemorrhages

A

T

29
Q

Bacillary hemoglobinuria are caused by infection from the soil

A

Tmainly endogenous but also infection from the soil

30
Q

Isolation of the agent from the gut gives aetiologic diagnosis of lamb dysentery

A

F

31
Q

Isolation of Cl. perfringens from the gut confirms the diagnosis of lamb dysentery

A

F

32
Q

Lambs have to be vaccinated with anatoxin vaccine in order to prevent lamb dysentery

A

FEwes

33
Q

For diagnosis of lamb dysentery, the pathogen should be cultured from the intestine

A

T
lamb dysentery no bacterium isolation from the gut but microscopy culture is okay

34
Q

Lamb dysentery can be successfully treated with penicillin when clinical signs appear

A

F

35
Q

Struck is a slow disease of older sheep

A

FFast course sudden onset

36
Q

Struck is a worldwide common disease with great economic impact

A

F

37
Q

Infectious necrotic enteritis of piglets occurs in the first 1-2 weeks of life

A

T2-4d so 1st week

38
Q

Pigs showing clinical signs of enterotoxaemia have to be treated with antibiotics immediately

A

FToo latePenicillins to sows

39
Q

Necrosis of gut epithelium is a postmortem lesion of pig enterotoxaemia

A

T

40
Q

Pig enterotoxaemia has to be diagnosed by detecting antibodies in the piglets

A

Fdirect detection

41
Q

Pig enterotoxaemia causes abdominal contractions in sows

A

F

42
Q

Mesenteric lymph node is congested in case of pig enterotoxaemia

A

F

43
Q

Clostridium enterotoxaemia can be cultured from mesenteric lymph nodes or gut

A

T

44
Q

Enteritis in piglets cannot be diagnosed by post-mortem, only by bacteriology

A

F

45
Q

Necrotic enteritis of piglets cannot be diagnosed by isolating the agent from the gut

A

TEpidi en na evris c.perfringes C pou kami jalla disease??culture smear microscopy no isolation

46
Q

The toxin of the agent of pulpy kidney disease is sensitive to trypsin

A

TD(α, ε=trypsin act)

47
Q

Neurological signs are typical in the case of pulpy kidney disease

A

T

48
Q

Ulcerative enteritis can occur in 4-12-week-old chickens

A

Tdo not confuse with Necrotic enteritis of Chicken caused by C. perfringes ABroiler 2-5w, turkeys 7-12w

49
Q

Ulcerative enteritis of poultry is generally prevented with vaccination

A

F

50
Q

Lesions of ulcerative enteritis are mostly seen in the small intestines

A

TFirst small intestine then upper large intestine

51
Q

Lesions of necrotic enteritis of chicken are typically occur in the large intestine

A

Fjejunum and ileum =SI

52
Q

Necrotic enteritis mostly occurs in chicken

A

TThus the name necrotic enteritis of chickenAlso turkeys

53
Q

Waterfowl are not susceptible to necrotic enteritis

A

FThey are

54
Q

Gangrenous dermatitis causes muscle oedema

A

T

55
Q

The clinical signs of tetanus are inducible

A

T

56
Q

The agent of tetanus causes septicaemia

A

FThe bacterium never enters the bloodstream. Only the toxins released after the autolysis of the vegetative bacterium. Necrotic anaerobic conditions at entry site initiate spore germination.

57
Q

Clostridium tetani produced endotoxin

A

FEndotoxins= membrane compounds of gram negative bacteriaExotoxins = are proteinoid substances released

58
Q

Tetanus can be prevented with vaccines containing inactivated bacteria

A

Finactivated toxoid = Anatoxin

59
Q

Tetanus can only develop after deep wounds

A

F Not deep only, even navel infection, teeth of foals, and ear tagging

60
Q

The paralysis usually starts at the place of the wound

A

FSpasms begin from the head

61
Q

Dogs have high resistance to tetanus

A

Thave natural resistance to the toxin

62
Q

Clostridium tetani produces neurotoxins

A

T

63
Q

Clostridium botulinum cannot tolerate air at all

A

T

64
Q

In Hungary, botulism is seen most commonly in birds

A

T

65
Q

Clostridium botulinum spores are extremely resistant to heat

A

T

66
Q

In Hungary, botulism occurs in winter and early spring

A

Fwarmer climate summer

67
Q

Clostridium botulinum can produce toxin, some of which are activated by proteases

A

T

68
Q

Botulism is seen mainly during summer

A

T

69
Q

Spasms are the typical clinical sign of botulism

A

F

EPI HARDCORE (12 decks)

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