Clinical Treatment of Arrhythmias Flashcards

1
Q

Sinus bradycardia

A
  • slow HR
  • <60 bmp
  • does not necessarily indicate pathology
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2
Q

Sinus arrest

A
  • lack of sinus node discharge

- absence of p wave and absence

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3
Q

Bady-tachy

A

intermittent episodes of slow and fast rates form SA node or atria

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4
Q

Chronotropic incompetence

A
  • HR should normally respond smoothly to excercise/stress

- unable to reach max HR smoothly and unable to maintain

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5
Q

First-degree AV block

A
  • no actual block
  • AV conduction is delayed
  • long PR interval (>.2 secs)
  • primarily block occurring w/in AV node
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6
Q

Second-degree AV block - Mobitz I

A
  • not every p wave transmits to ventricular contraction

- progressive prolongation of PR interival until ventricular beat is drop

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7
Q

Locations w/in conduction system where problems can d\occur

A
  • @ sinus node
  • @ AV node
  • infranodal (below AV node)
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8
Q

Second-degree AV block - Mobitz II

A
  • no progressive prolongation – suddenly dropped QRS wave
  • regular PR interval
  • block primarily occurs below AV node: w/in His, bundles, or purkinje
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9
Q

Third-degree AV block

A
  • no impulse conduction from the atria to the ventricles

- p wave

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10
Q

Indications for treating bradyarrhythmias

A
  • patient is symptomatic and arrythymia at from any point in conduction system is detected
  • when the rhythm is infranodal (below the AV node)
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11
Q

Steps in treatment of bradyarrhythmias

A
  1. Find and treat reversible causes: i.e. ischemia/infarct, hypothyroidism, neurologic causes, lyme disease
  2. Stop offending meds
  3. Acute tx: beta-agonists (dopamine or isoproterenol), transcutaneous pacing, venous pacing
  4. Long term: permanent pacemaker
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12
Q

Tachyarrhythmias

A
  • Above ventricle=supraventricular tachycardias (SVTs)

- @ ventricle=ventricular tachycardia, ventricular fibrillation

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13
Q

Supraventricular tachycardia types

A
  • regular (consistent QRS intervals w/P waves) vs. irregular
  • regular: sinus tachycardia
  • irregular: atrial fibrillation (no discrete Ps); multifocal atrial tachycardia (3+ Ps); atrial flutter (flutter waves)
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14
Q

Irregular supraventricular tachycardia tx

A

if unstable –> shock

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15
Q

Regular supraventricular tachycardia tx

A
  • give adenosine
  • adenosine may slow down rhythm to help dx arrythmia
  • adenosine may also slow down AV node and actually treat/terminate tachycardia
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16
Q

Atrial fibrillation approach

A
  • 5 C’s:
  • Cause: reverse
  • Control rate
  • antiCoagulation –> @ risk for stroke, must prevent clots
  • Control rhythm
  • Cure?: ablation
17
Q

Common causes of AF

A
  • hypertension
  • mitral valve disease
  • alcohol
  • cardiomyopathy
  • hyperthyroidism
  • lone AF
  • cardiac surgery
18
Q

Immediate Treatment of Atrial fibrillation

A
  • cardiovert (shock) if close to hemodynamic collapse

- control rate if

19
Q

Rhythm control in Atrial fibrillation

A
  • pharmalogical: less successful, but does not require sedation –> Class IC (flecainide, prpafenone) and Class II anti-arrhythmics (ibutilide, amiodarone, dofetilide, sotalol)
  • electrical
20
Q

Maintenance rhythm control in AF

A
  • can use meds at lower doses
  • Class IC agents: contraindicated inCAd and structural heart disease
  • Class III agents: amiodarone, sotalol, dofetilide, dronedarone
21
Q

Rate control in AF

A
  • medications: beta blockers, digoxin, verapamil, diltiazem, amiodarone as rate-control in decompensated HF
  • Digoxin=not good during exercise
  • Beta-blockers and Ca channel blockers control HR during exercise
22
Q

Rhythm control: catheter ablation

A

-surgical technique that cauterizes atrium to prevent triggers from pulmonary veins from getting into atrium

23
Q

Atrial flutter treatment approaches

A
  • similar to Afib
  • can be more difficult to control rate or rhythm
  • catheter ablation more successful than meds (95% cure rate)
24
Q

Other SVTs (3)

A
  • AV nodal reentrant tachycardia
  • accessory pathway-mediated tachycardias: abnormal connection between atrium and ventricle
  • focal atrial tachycardias: least common, abnormal focus w/increased automaticity
25
Q

Treatment approaches to other SVTs

A
  • individualized tx

- catheter ablation

26
Q

Ventricular Tachyarrhythmias characteristics

A
  • “more Vs than As” = more QRS waves than P waves
  • wide complex tachycardia usually indicates ventricular origin
  • CAD = 90% of time it is VT
27
Q

Acute tx in stable Vtach

A
  • meds: amiodarone, lidocaine, procainamide

- treat underlying causes

28
Q

Acute tx in unstable Vtach

A
  • shock
  • treat underlying causes
  • meds
29
Q

Approach to Vtach w/out structural heart disease

A
  • usually idiopathic: focal arrythmogenic trigger
  • usually “benign”
  • meds or ablation
  • defribillator rarely necessary
30
Q

Approach to Vtach w/ structural heart disease

A
  • treat underlying causes
  • risk stratify
  • defribillator + meds/ablation
31
Q

Reasons for defribrillator in Vtach

A
  • secondary prevention: pt who has already had sudden cardiac arrest due to VT/VF w/out a reversible cause
  • primary prevention:pt w/out previous cardiac arrest but is at significant risk