Arrhythmias Flashcards
1
Q
EKG features of normal sinus rhythm
A
- a normal P wave (atrial depolarization), PR interval (resistance to conduction at the AV node), QRS complex (ventricular depolarization), and a T wave (ventricular repolarization).
- Normal sinus rate in adults is 60-100 bpm.
- Normal PR interval is 0.12-0.20 seconds.
- Widening of the QRS, preceded by a normal P wave and a normal PR interval→rhythm is still sinus.
- Rate and regularity of rhythm may vary slightly with respiration.
2
Q
Sinus tachycardia definition/common conditions
A
- regular, fast heart rate (>100 bpm)
- commonly occurs during exercise or emotional stress
- no treatment is generally needed, but in patients with coronary artery disease the increased cardiac oxygen demand may precipitate angina
- Sinus tachycardia is associated with hyperthyroidism.
- If treatment is needed, a beta-blocker is usually effective.
3
Q
Sinus bradycardia definition/common conditions
A
- regular, slow heart rate (<60 bpm)
- common in normal individuals, especially athletes and requires no treatment
- Sinus bradycardia may produce syncope during intense vagal activation as in fainting for which atropine is an effective tx
- Often occurs with small inferior wall infarctions that increase vagal tone.
- Can cause syncope, lightheadedness or fatigue in elderly patients with age-related dysfunction→sick sinus syndrome. Treatment may require placement of an electronic pacemaker.
4
Q
Atrial rhythms definition
A
- rhythms originating in the atria
- e.g. atrial fibrillation or atrial flutter
5
Q
Junctional (nodal) rhythms definition
A
- Regular, narrow (normal) QRS complex with no antecedent P waves.
- region surrounding the AV node = “junction” and rhythms originating there are called junctional rhythms.
- They may be either slow or fast. They are a regular rhythm usually with narrow QRS complexes.
- P waves are often not seen because they are buried within the QRS complex or they may occur very shortly before or after the QRS.
- P waves that are seen often inverted because they are conducted upward from the AV node rather than downward from the SA node.
6
Q
Ventricular rhythm
A
- rhythms originating in the ventricle
- e.g. ventricular tachycardia or ventricular fibrillation
7
Q
Causes, EKG features of first-degree AV block
A
- Causes: drug-induced (beta-blockers, some calcium blockers, digitalis), conduction system disease.
- EKG features: PR interval is greater than 0.2 seconds (one large block on the EKG).
- This is a benign condition that can proceed to more serious types of block.
8
Q
Causes, EKG features of second-degree AV block
A
- Causes: conduction system disease, high vagal tone, excessive effects of drugs.
- EKG features: Some P waves conduct normally to ventricles but others do not. Patterns vary.
- If the rate is too slow to slow to support cardiac output adequately, syncope or confusion may occur requiring a pacemaker.
9
Q
Causes, EKG features of third-degree AV block
A
- Causes: av node or junctional failure with aging, infarct or disruption during cardiac surgery—rarely caused by drugs.
- EKG features: Both Ps and QRSs show regular rhythm, but they are at different rates. With P rate > QRS rate.
- May cause syncope or sudden death. Usually requires a pacemaker.
10
Q
Causes, EKG features, clinical manifestations, and tx of atrial fibrillation
A
- EKG features: irregularly irregular ventricular rhythm. No p waves.
- Causes: NI subjects, aging, post-operative, heart disease, hyperthyroidism
- Clinical manifestations: rapid heart rate (syncope, ischemia, heart failure), loss of atrial kick (heart failure), atrial thrombi (embolic stroke).
- Treatment: anticoagulation, rate control with drugs, cardioversion—electrical or drugs, ablation.
11
Q
Causes, EKG features, clinical manifestations, and tx of atrial flutter
A
- EKG features: P waves (flutter waves) at rate of 240-320 bpm. Pulse may be regular or irregular. Ventricular rates vary widely—typically rapid if untreated.
- Complications: atrial flutter has some risk of embolic stroke due to clot in the left atrium, and may result in rapid ventricular rates that are poorly tolerated.
- Treatment: anticoagulation, rate control with drugs, cardioversion, ablation.
12
Q
Causes, EKG features, clinical manifestations, and tx of ventricular tachycardia
A
- EKG features: repetitive wide-abnormal QRS complexes, no p-wave
- Causes: fibrosis, infiltrate, dilation, long path length permitting reentry.
- Clinical manifestations: very bad. Abnormal ventricular contraction
- Treatment: emergency defibrillation.
13
Q
Causes, EKG features, clinical manifestations, and tx of ventricular fibrillation
A
- EKG features: abnormal, abnormal, abnormal—all noise, no p waves, no QRS complexes, no T waves.
- Causes: can progress from ventricular tachycardia. Heart failure.
- Clinical manifestations: very bad. No ventricular contraction.
- Treatment: immediate emergency defibrillation.