Clinical neurosurgery lecture [12/01/20] Flashcards

1
Q

What is neurophobia?

A

Perceived complexity about neuroanatomy, neurological examination, multitude of rare disease

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2
Q

Three types of approaches to approaching a patient? Which is the modern approach?

A
  • Disease orientated approach [diagnose on spot from Sx, simplistic]
  • Localisation approach: take each patient Sx then draw vend diagram to get Dx
  • Clinical syndrome: story of Pt, only 20 or so clinical syndromes mostly anatomically based. Modern way.
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3
Q

Name the 20 neuro clinical syndromes [very hard]

A

[look at slide]

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4
Q

if in doubt with neuro syndrome, what should you do?

A

Scan!

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5
Q

Which junction type does myasthenic syndrome involve?

A

Neuromuscular junction

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6
Q

Which part of the nerve does a radiculopathy affect?

A

Nerve root

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7
Q

Which part of the body does vertebral pain affect?

A

The discs/ligaments of the spine

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8
Q

Part of the body does a myelopathy affect?

A

The spine

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9
Q

Which cell in spine does MND affect?

A

Anterior horn cell

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10
Q

Where does a parasellar syndrome grow in the body?

A

Pituitary tumour

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11
Q

What can cause meningeal irritation?

A

Pus/blood against it

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12
Q

Put simply, what is a stroke?

A

Any neurological deficit

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13
Q

Part of the brain does extrapyramidal Sx indicate effected?

A

Basal ganglia

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14
Q

What is somatisation?

A

Psychological

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15
Q

Next steps if patient unable to give a history

A
  • Collateral history

- ABC resus

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16
Q

Next steps if patient able to give a history?

A
  • do pain tool
  • do domain tool
  • pick up on ‘patient speak phrases’
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17
Q

Following steps after speaking to patient who is able to give history

A
  • hypothesis-based signs from framework headings

- core examination [incl. vital signs]

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18
Q

Final step in basic structure of neuro-assessment

A

Clinical syndrome secondary to [likely] underlying disease entity

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19
Q

Summarise basic neuro-assessment

A

[look up]

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20
Q

Reasons why patient may not be able to give a history

A

Patient confused, impaired level of consciousness, can;t speak language, tracheostomy, lower CN problems, struggling to speak, dysphasia, individuals who are deaf, special needs [sign language], ventilated people

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21
Q

What are the 5 vital signs?

A

Blood pressure, body temperature, pulse rate, respiratory rate, oxygen saturation

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22
Q

if something wrong with vital signs, what does this indicate?

A

A secondary brain problem is afoot

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23
Q

What is a primary brain problem?

A

A problem of the brain

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24
Q

Signs of an AAA

A

Pulse rate up, BP down, abdominal distention

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25
Q

Go through the GCS pointing system

A

[look at slide]

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26
Q

Name a way of doing a quick neuro examination

A

GCS!

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27
Q

Which examination should you do in ‘anal sphincter’ scenarios?

A

GCS! Happen about once monthly when you qualify.

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28
Q

Which drive present during anal sphincter moments? Describe it.

A

Sympathetic drive: rush adrenaline, logic out of window

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29
Q

Which part most important in the GCS?

A

Best motor response. Apply painful stimulus. Ensure doesn’t elicit reflex.

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30
Q

Best place to get a motor response?

A

Fingers back of jaw: won’t get bruising behind ears, can do jaw thrust easily from this position, squeeze same extent when can’t see hands

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31
Q

Another common place to do best motor response?

A

Supraorbital place

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32
Q

Which CN distribution does mastoid area for motor response sensation test?

A

Trigeminal nerve

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33
Q

Differentiate localising, flexing, and extending in the GCS

A

Localising: arm comes up to push away painful stimulus
Flexing: arm up but doesn’t push away
Extending: arm goes down and stays there

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34
Q

Which is better to use when presenting GCS, numbers or descriptors?

A

Descriptors! Less confusion.

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35
Q

When does GCS become a good predictor of outcome for patients?

A

If it is done an hour after the incident. Exceptions include if on tube and ventilation done on site [as anaesthetist has put pain medication down throat]

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36
Q

Exceptions to pt extending meaning person is not essentially dead?

A
  • Unless under hour car accident

- or something really simple like hydrocephalus in a young person

37
Q

What happen if person is flexing?

A

Then patient will likely do well

38
Q

What is withdrawal on the GCS and why isn’t it used now?

A

Squeezing finger for painful stimulus. Not used now as can cause reflex.

39
Q

How common is abnormal/spastic flexion?

A

Rare, pt will usually come down on flexion or extension if press long and hard enough

40
Q

What does lateralising signs mean?

A

Means a problem of one hemisphere compared to another. Don;t have to test for lateralising signs. Could be inattnetion, gaze paresis.

41
Q

What are lateralizing signs

A

Look up

42
Q

If patient has fixed dilated pupils, what could be a sign of?

A

Lesion 3rd nerve parasympathetic fibres

43
Q

Where does the 3rd nerve supply the brain from?

A

I think the midbrain

44
Q

Describe the course of the 3rd CN. Why does this lead to dilated pupils?

A

Midbrain -> Dura -> petris part of the temporal bone in middle ear, apex of it is like a guitar string.
If brain squeezes this due to e.g. blood clot, the parasympathetic fibres won’t work [outside of fibre motor, inside is sensory]. This causes pupil dilation.

45
Q

How to test for dilated pupils?

A

Pen torch each eye. Direction pupillary reflex and look. The indirect pupillary reflex when shine another eye and look at fixed pupil. Test for direct and indirect pupillary reflex basically [think can do by swinging reflex maybe?].

46
Q

Why is important to test the indirect pupillary reflex?

A

pt could have a blind eye.

47
Q

Give example of how sympathetic vs parasympathetic NS works?

A

Friday evening, go to a pub at 8pm lights down pupils up. See someone pupils dilates she’s interested. Go to loo even though it’s been 10 minutes ago. Blushing, tachycardia. Feeling flustered, executive functioning not very good. Basically me on a coffee I think.

48
Q

What is Horner’s syndrome and how does it relate to PS/S nervous system?

A

Horner’s syndrome:

  • miosis, pitosis and anhedrosis
  • miosis [constriction of pupil] is due to damage of the sympathetic NS of the face
49
Q

Briefly describe the course of the sympathetic NS

A

nerve from brainstem, hitch a lift on a artery, through upper thoracic nerve roots, sympathetic chain, internal carotid artery

50
Q

Which artery supplies the skin [I think around the face]?

A

External carotid artery

51
Q

If person comes in unconscious at 3am and need more info, should call relatives etc.?

A

Yes, get a collateral history immediately [regardless of time]

52
Q

Patient comes in unconscious, pulse 140bpm, 80% sats, 40C temp. Housekeeping for unwell patient such as this? 8am she is fine, up and having toast, what likely happened?

A

Housekeeping
- put her on O2
- r/o infection for temp blood cultures then start Abx, Rx paracetamol/aspirin
Likely happened
- secondary brain problem. Though had chronic subdural okay to manage and turns out main problem was pneumonia so just needed Abx

53
Q

What are the 15 elements of the pain tool?

A

Look up slides

54
Q

What are the 7 elements of the domain tool?

A

Look up slides

55
Q

If in doubt when taking history, what should you do?

A

SCAN!

56
Q

Which type of questions best for taking a neuro history?

A

Closed questions [not leading questions thought]

57
Q

What can question about sleep tell you about the condition?

A

Severity

58
Q

Important questions asked pain tool not asked SCRATES

A

Prceipitating factors, prgoression, disturbing sleep, ever had before, response to conservative measures, significantly interfering lifestyle

59
Q

What is a myelopthy?

A

Look up

60
Q

Which nerve does carpal tunnel effect? Which weakness does this cause and how may this present?

A

Median, gripping so keep dropping coffee mug [patient-speak phrases]

61
Q

Strategy for patient with multiple Sx

A

Either take most significant tot hem and all other Sx associated/preceding
or could take pain as most prominent Sx

62
Q

What is Rett syndrome?

A

Global neurological syndroem, random genetic inherited.
Fine until 2y, then regress after this age.
Sleep-wake distubrance, can soil self, look normal but no verbal abilities, use of hands angel prayer, struggles swalloing, needs feeding, has feeding problems, near repetitive hand movements, feeding problmes, wheelchair bound, no insight danger

63
Q

Type of neurological diseases have sleep disturbances?

A

Global neurological deficits [like PD]

64
Q

Grades for domain tool?

A

1 to 7 [look up], with 7 being bed bound or hoist transfer

65
Q

RFs for pressure sores

A

Nutrition, bed bound, incontinence etc.

66
Q

Red tray hospital systems

A

Means people need help with feeding [coordination/vision/swalloing etc. problem]

67
Q

If pt can’t communicate mouth, how may communciate?

A

With eye and IR camera

68
Q

What does personal hygiene/continence relate to?

A

Sphincter control [wearing pads], reducing social circle e.g. cuada equina exmaple

69
Q

Interperonsal relationships: lost job 6m ago, estreanged family and friends and don;t do hobbies etc.?

A

possible demenita or deprression

70
Q

What should exclude if person withdrawing from life?

A

Bullying

71
Q

For sleep, best way to start Tx

A

Sleep hygiene, melatonin can do. Last thing want to do is sedate someone.

72
Q

What is the hypothesis-based approach to neurological examination?

A

Core examiantion [gen app, vital signs, gait, fundscopy for pappiloedema/optic atrophy, long tract signs, vib sense] + one or two signs associated with the two or three clinical syndromes shortlisted from history

73
Q

If in doubt in examination ….

A

SCAN

74
Q

Should you routinely do dermatones?

A

brain dead approach check all dermatomes. Can really just ask in the history: “do you have any numbness”?

75
Q

What are the vital signs?!

A

BP, oxygen sat, temp, pulse, , RR

76
Q

Which parts of the examination are important as they won’t be told during the history?

A

UMN signs, back of eyes, vib sense

77
Q

Knee and ankle reflex important?

A

Not really

78
Q

Anatomical pathway of the long tracts

A

Parametical cell in the cortex, goes through capsule, through brainstem and then synapsing at the anterior horn cell

79
Q

Other names for UMN signs

A

Long tract, pyramidal signs, ascending tract sign

80
Q

Most common cause of myelopathy?

A

OA

81
Q

Late sign of myelopathy, and early sign

A

Early sign: UMN signs

Late sign: lack of sensation

82
Q

The 6 signs for UMN

A

Babinski, clonus, cross adductors, Hoffman’s, open and close hands, delto-pector reflex

83
Q

Progression of myelopathy for most people

A

Insidious

84
Q

two syndromes where signs before Sx in neuro. How to screen for these?

A

Myelopathy, peripheral neuropathy.

UMN signs and also vibration sense.

85
Q

Which tuning fork vibration sense?

A

128Hz

86
Q

Two large pieces of kit in the body nerve distributed by?

A

Jelly fish: avoid extreme temp, simple kit, unmyelinated, C fibres, few genes, pain
Evolutionary new: advances, myelinated, A fibres, large diameters, fast conducting

87
Q

Whihc fibres effected by peripheral neuropahy?

A

Computer kit problems, light touch/vibration/proprioception effected

88
Q

5 sentence structure to presenting a patient?

A
  1. The pt, PC
  2. HPC
  3. PMH
  4. Examination
  5. Summary and DDx