Classifications of Periodontal Disease Flashcards

1
Q

What are necrotising periodontal diseases?

A

A unique type of periodontal disease that involves tissue necrosis. They are non-contagious and occur in less than 1% of people in developed countries.

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2
Q

What are the symptoms of necrotising periodontal disease?

A
  • Painful bleeding gums
  • Ulceration and necrosis of papilla
  • ‘Punched out’ appearance of gums
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3
Q

Categorise necrotising periodontal diseases into three broad types.

A
  • Necrotising gingivitis (only gingival tissues affected)
  • Necrotising periodontitis (progresses to PDL and alveolar bone)
  • Necrotising stomatitis (progresses to deeper tissue beyond mucogingival line, including lip/cheek/tongue)
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4
Q

What conditions predispose a patient to necrotising stomatitis?

A

Malnutrition and HIV

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5
Q

What is cancrum oris?

A

Necrotising destructive infection of the mouth and face, not technically periodontal disease - but some evidence suggests it can develop from necrotising periodontal disease.

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6
Q

What are the symptoms of necrotising gingivitis?

A

Ulcerated and necrotic papillae, ulcers are covered by yellow grey psudomembrane, that when removed causes bleeding.

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7
Q

What is the psudomembrane of necrotising gingivitis made of?

A

A slime with no coherence, made of fibrin, necrotic tissue, leukocytes, erythrocytes, and a mass of bacteria.

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8
Q

What are the symptoms of necrotising periodontitis?

A

Ulcerations similar to that of NG, but associated with deep pockets. Psudomembrane not obvious if present at all. Adenopathies are found in the most severe cases of the disease.

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9
Q

What are the symptoms of necrotising stomatitis?

A

Disease develops through bone, causing necrotic tissue to form. Sequestrated bone may appear, with large areas of osteitis/OAF.

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10
Q

Why is it difficult to test for NPD?

A

Histopathology is not pathognomic (not characteristic) for NPD - so biopsy isn’t able to confirm.

Microbiology is not characteristic either, as there are a large array of bacterial types which don’t necessarily indicate NPD.

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11
Q

List five diseases that can be confused with NPD.

A
  • Oral mucositis
  • HIV associated periodontitis
  • Herpes simplex virus (HSV)
  • Scurvy
  • Gingivostomatitis
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12
Q

How can you differentiate between NPD and primary herpetic gingivostomatitis (PHG)?

A

NPD won’t have identifiable bacteria, will rarely appear outside the gingiva, and result in permanent destruction of periodontal tissue.

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13
Q

What are the risk factors for NPD?

A
  • Stress
  • Sleep deprivation
  • Poor OH
  • smoking
  • Immunosuppression
  • Malnutrition
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14
Q

What treatment is there for necrotising periodontal disease?

A
  • Superficial debridement (PMPR) softly and under LA if required
  • Tooth brushing discouraged as can interrupt healing
  • Chlorhexidine mouthwash, 0.12-0.2% twice daily.
  • If systemic symptoms unresolved consider antimicrobial prescription.
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15
Q

What antibiotic and what dose should be recommended for a NPD patient who has not responded to initial treatment?

A

Metronidazole 400mg TID for 3 days

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16
Q

Why should locally delivered antimicrobials not be recommended to treat NPD?

A

Not enough concentration of drug to deal with the high levels of bacteria, so they will be ineffective.

17
Q

How often should a patient be followed up with after presenting with NPD?

A

Daily if possible, and as signs and symptoms start to improve strict OH measures and PMPR should be used.

18
Q

What aesthetic consequence of NPD are most common?

A

Gingival craters

19
Q

How can you treat gingival craters?

A
  • Gingivectormy
  • Gingivoplasty
  • PD flap surgery
  • Regenerative surgery
20
Q

What should you advise the patient of, if they have no predisposing risk factors but present with NPD?

A

They may want to be screened for HIV, as it could be suggestive of a HIV infection.

21
Q

Why is the 4mm threshold for BPE critical for determining PD disease stability?

A
  • A higher probing depth may not represent active disease.
  • Probing depth may not recover quickly after therapy.
22
Q

What is the definition periodontal health?

A
  • Patients with an intact periodontum
  • Patients with a reduced periodontum not due to PD
  • Patients with a reduced periodontum due to previous PD, but currently stable.
23
Q

Why may a BPE of 3/4 not necessarily indicate full periodontitis?

A

The gingiva may be severely inflamed, and no bone loss may be present. Can be inflamed by:

  • Poor restoration margins
  • Pregnancy (epulis- non plaque induced)
  • Puberty induced
  • Drug induced
24
Q

What non-plaque induced gingival diseases are there?

A
  • Hereditary gingival fibromatosis
  • Herpetic gingival stomatitis
  • Lichen planus (immune reaction)
  • Benign mucous membrane pemphigoid (immune reaction)
  • Vit C deficiency
25
Q

What systemic diseases may manifest periodontal disease?

A
  • Papillon Lefevre Syndrome
  • Leukocyte Adhesion Deficiency
  • Hypophosphatasia
  • Down’s syndrome
  • Ehlers-Danlo
26
Q

Under the 2017 periodontal disease classification system, what are the 10 different categories for periodontal disease?

A
  1. Health
  2. Plaque induced gingivitis
  3. Non-plaque induced gingivitis
  4. Periodontitis
  5. Necrotising periodontal disease
  6. Perio from systemic disease
  7. Systemic diseases affecting PDL
  8. Periodontal abscess
  9. Periodontal endodontic lesions
  10. Mucogingival deformities