Class 1 nursing management and interventions in CV Flashcards
HTN management
-DASH diet
-Low sodium diet 1200-1500 mg with max of 2000 mg per day
-Healthy body weight and moderate exercise activity
-Limit alcohol consumption and tobacco use
-Stress management
HTN drug management
-Multiple drugs often used, especially with Type 2 diabetes
-Replace multiple antihypertensive agents with single pill combination therapy
-Single pill combinations or monotherapy should be considered for initial antihypertensive therapy.
-Low doses of multiple drugs may be more effective and better tolerated than higher doses of fewer drugs
-Don’t mix ACE&ARBs
-In combination therapy, an ACE inhibitor plus a long-acting dihydropyridine CCB is preferred
Hypertensive crisis
-Severe and abrupt
-Treatment goals is to decrease MAP to 10-20% in first 1 to 2 hours, with gradual reduction in the next 24 hours
Hypertensive emergency IV medications
-Vasodilators (sodium nitroprusside*, nitroglycerin, hydralazine)
-Adrenergic inhibitors (phentolamine, labetalol, esmolol)
-Ace Inhibitors (Enalapril)
Nursing considerations in hypertensive emergency
-Monitor BP with an art line
-Continuous ECG monitoring to observe for dysrhythmias
-Hourly urine output
-Bed rest to avoid cerebral ischemia and fainting
Hypertensive emergency therapy goals
-Minimize end-organ damage (cardiac, renal, respiratory, and retinal damage), maintain adequate perfusion with special assessment of neuro, cardiac, and renal monitoring
Hypertensive urgency
-Can be managed with PO medications (captopril and clonidine)
Acute hypertension
-Evidenced by hypoperfusion to the brain, heart & kidneys
Acute hypotension caused by blood or fluid loss tx
-IV fluids, blood, or blood products
Acute hypotension caused by primary CV dysfunction tx
-AMI, cardiac tamponade & pulmonary embolism will result in a fall in CO and may need drug intervention
Acute hypotension caused by secondary CV dysfunction tx
-Result of negative chronotropic & inotropic effects caused by beta blockers, digoxin, or opioids and may need to administer vasoconstrictive agents
Non-cardiac factors causing decreased O2 supply
-Anemia
-Low blood volume
-Hypoxemia
-Pneumonia
-Asthma
-COPD
Non-cardiac factors that increase myocardial O2 demand
-Anxiety
-Cocaine
-Physical exertion
-HTN, hyperthermia, hyperthyroidism
Cardiac factors that decrease O2 supply
-Coronary artery spasm & thrombis
-Dysrhythmias
-HF
-Valve disorders
Cardiac factors that increase O2 demand
-Aortic stenosis
-Cardiomyopathy
-Dysrhythmias
-Tachycardia