Chronic Pian management Flashcards

1
Q

how to safeguard against future failed efforts

A
  • identify yellow flags
  • maladaptive believes/expectations/behaviours
  • presence of major life stressor
  • look for signs of self efficacy
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2
Q

what are maladaptive beliefs and pain behaviours

A

-reflexive behaviours that are not the result of intentional make

(continued avoidance of mvmt, disengagement, catastrophizing, fear avoidance, anticipation of pain)

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3
Q

what in the most important determinant of disability

A

Self efficacy

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4
Q

examples of classification systems in management (3)

A
  1. stages of healing
  2. Pain mechanisms
  3. injury mechanisms
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5
Q

features of inflammatory stage of healing

A

0-7d

  • transient vasoconstriction leads to microvascular dilation
  • pain w mvmt, evidence of gaurding
  • night pain
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6
Q

what to do w pt in inflammatory stage

A
  • educate, reassure, protect

- unloaded movement with pain free available ranges of motion

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7
Q

what to do w pt in proliferative phase

A
  • unloaded movement, working towards full ROM within 4-6w

- Introduce controlled loading within pain free postures and jt pos.

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8
Q

What to do w the patient in the repair and remodelling stage of healing

A
  • Use dif loading stats to continually remodel connective tissue (end range loading, EC)
  • time under tension to build endurance and challenge greater ROM in combo w multi jt movements (usually lack of loading when not progressing)
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9
Q

what is nociceptive inflammatory pain

A
  • typically localized, intermittent or consistent-described as swollen, full, throbbing
  • earliest stages of tissue healing OR too much too soon in later stages, increased pain in morning/evening
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10
Q

What is nociceptive ishemic and when does it happen

A

-occurs later in healing response (>12W)

  • insufficient vascularization and oxidation of tissues
  • worse as the day progresses or activity dependent
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11
Q

neurogenic/neuropathic pain mechanisms

A

localized (receptor field)

-less predictable 24hr period, often worse at night

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12
Q

what are affective pain mechanisms

A

Involves central pathways related to negative emotions, perceptions and behaviours

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13
Q

What are central sensitization pain mechanisms

A

Altered cognition and interpretation of nociceptive signals occurring in the CNS

-persistence nociceptive input can induce CNS changes

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14
Q

What are motor/autonomic pain mechanisms and Ex,

A
  • facilitated inflammatory mechaniss, autonomic/vasomotor dysfunctions, neuroplastic/cortical changes
  • widespread, non anatomical distribution w inconsistent 24hr behaviour

ex- CRPS

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15
Q

What is derangement syndromes and how to TX

A

-disturbance of normal resting pos of jt surfaces,resulting in pain

  • avoid mvmt direction that exacerbates pain and adress faulty mvmts patterns
  • Utalize mvmt strats that abolish/reduce pain
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16
Q

What are dysfunction syndromes and how to tx

A

-deformation of struccturally impaired msk tissues (end range/restricted mvmt)

  • tissue deconditioning/fatigue almost always a factor
  • mobailzing exercises in the direction of dysfunction or direction of reproduction of pain (restore ROM)
17
Q

Tissue fatigue syndrom and how to tx

A
  • High volume of repativi physical stress to tissues over prolonged duration (slow + progressive onset)
  • Reduce cumulative tissue load with active and passive strats, identify contributing environmental factors
18
Q

What is postural stress syndrome and how to tx

A
  • pain arrives w static positioning and abates w moveemnt

- pt education on mechanism behind cumulative postural strain, pacing, general fitness/conditioning

19
Q

5w of massage for fibromyalgia did what

A

recuded anxiety/depression

20
Q

HVLA effects

A
  • increase pain thresholds and decrease motor neuron excitability measured by the H reflex
  • SMT can decrease mm spindle activity, reduce motor neuron excitability and reduce EMG

-not better than other tx tho

21
Q

how does nerve sliding initiate tension (3)

A
  1. 1st 1/3 of mvmt takes up slack
  2. 2nd 1/3 of mvmt is where sliding occurs
  3. 3rd 1/3 of mvmt is where tension develops
22
Q

General pa guidlines

A
  • No correct activity excists
  • pt guided, shorter duration, low intensity
  • adequate recovery periods
  • can have discomfort day but not a flared day following
23
Q

psychological benefits of exercise

A

BDNF enhance the survival and differentiation of neurons and stim neurogenesis
-improves the quality of sleep, counteracts the mental decline w age, effectove at tx depression + anxiety

24
Q

running vs weight training for depression

A

both similar; no sig difference

25
Q

best exercise for OA

A

stregening/full body/aerobic probably best

26
Q

effectiveness of ex for fibromyalgia

A

sig benefits

  • most prefered arobic compared to strength
  • 8w of aerobic was superior
  • pool therapy was good too
27
Q

tx for acute/chronic LBP

A

acute- reassurence, adviem self care strats

chronic- education, self management, stay active, sup exercise, multimodal rehab

28
Q

Exercise choses for chronic lowbackpain

A

exercise should mimic most vulnerable mvmts

29
Q

incomprlete vs complete tears strain %

A

incomplete start at >6%

Complete occur at 8-10%

-research suggests that tendinosis may be the result of chronic underloading and not overloading

30
Q

traffic light system for exercise prescription

A

Green- pain is no worse than baseline or pain improves and resolves after 6hr

Yellow- pain persists but no worse than during activity, takes >6hrs to resolve

Red- Pain worsens during exercise and it stops you from performing the activity any longer