chp 29 Recognition and Care of the Shock Patient Flashcards

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1
Q

Anaphylactic Shock

A

Type of shock caused by an overreaction of the immune system when exposed to an allergen.

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2
Q

Cardiogenic Shock

A

Type of shock caused when the heart can no longer pump blood adequately, resulting in a decrease in cardiac output and thus a decrease in perfusion.

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3
Q

Compensated Shock

A

when the patient is developing shock but the body is still able to maintain perfusion.

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4
Q

Decompensated Shock

A

when the body can no longer compensate for low blood volume or lack of perfusion. Late signs such as decreasing blood pressure, become evident

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5
Q

Hemorrhagic Shock

A

Type of shock caused by a loss of blood.

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6
Q

Hypoperfusion

A

inadequate perfusion of blood to an organ or organs. Also called shock.

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7
Q

Hypovolemic Shock

A

type of shock caused by a sudden decrease in body fluids (blood or other body fluids)

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8
Q

Neurogenic Shock

A

type of shock caused when the vessels dilate abnormally in response to injury to the spinal cord.

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9
Q

Obstructive Shock

A

a form of shock that blocks the forward movement of blood within the circulatory system.

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10
Q

Perfusion

A

the supply of oxygen to and removal of wastes from the cells and tissues of the body as a result of the flow of blood through the capillaries.

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11
Q

Psychogenic Shock

A

type of shock caused by a sudden and temporary dilation of the blood vessels from psychological causes.

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12
Q

Respiratory/Metabolic Shock

A

a form of shock caused by a disruption in the ability of cells to utilize oxygen effectively.

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13
Q

Septic Shock

A

Type of shock caused by severe infections that abnormally dilate the blood vessels.

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14
Q

Shock

A

inadequate perfusion of blood to an organ or organs. Also called hypoperfusion.

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15
Q

29-2 Describe the physiology of maintaining adequate perfusion.

A

Cells require an adequate supply of oxygen and glucose for aerobic metabolism, which ensures production of ATP. In addition to oxygen and glucose, good perfusion requires an adequate volume of blood, a properly functioning heart, and an intact system of blood vessels. Without one of these components, hypoperfusion happens.
S/S of adequate perfusion are:
* Normal Skin Signs
* Normal mental Status
* Normal Vital Signs
S/S of inadequate perfusion are:
* Abnormal skin signs (pale, cool, and moist)
* Altered mental status (agitation, restlessness, sluggishness, confusion, or decreased responsiveness)
* Abnormal vital signs (increased pulse and respiratory rate, decreased blood pressure)

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16
Q

29-3 Explain the Pathophysiology of shock (hypoperfusion)

A

a progressive condition that occurs when a patient does not receive an adequate supply of well oxygenated blood to all organs of the body. It can occur for several reasons, including damage to the heart, loss of blood, and abnormal dilation of the vessels. When enough cells of an organ suffer from inadequate perfusion it malfunctions and shuts down.

17
Q

29-4 Describe how inadequate vascular volume, inadequate heart function, ad decreased peripheral vascular resistance can lead to shock.

A

Inadequate vascular volume, inadequate heart function, and decreased peripheral vascular resistance can lead to shock.

  • Inadequate Volume - by bleeding uncontrollably, or dehydrated, either case results in a decrease in cardiac preload and in turn cause a decreased stroke volume. The heart compensates by increasing heart rate. If that does not work the blood pressure will drop.
  • Pump failure - Inability of the heart to pump with enough force to supply blood to the body. This comes from three things.
    • Damage to the heart muscle (myocardial infarction.
    • trauma
    • buildup of blood or fluid in the pericaridial sac that surrounds the heart causing compression of the heart (pericardial tamponade)
  • Inadequate blood vessel tone - A minimum amount of systemic vascular resistance (SVR) is required to maintain adequate blood pressure. When faced with a failing SVR due to lack of smooth muscle tone, the heart rate increases until it can not keep up output and systolic pressure drops.
18
Q

29-5 Explain the mechanisms and pathophysiology of each of the following categories and types of shock: Cardiogenic, Distributive (anaphylactic, septic, and neurogenic), hypovolemic (hemorrhagic and nonhemorrhagic), metabolic or respiratory, and obstructive.

A
  • Distributive Shock - an over dilation of the blood vessels when exposed to an allergen (anaphylactic), severe infection (septic), in response to injury of the spinal cord (Neurogenic).
  • Cardiogenic Shock - the heart can no longer pump blood adequately, resulting in a decrease in cardiac output and thus a decrease in perfusion.
  • Hypovolemic Shock - Loss of blood or other fluids such as through severe diarrhea which is dehydration.
  • Obstructive Shock - when forward movement of blood flow is obstructed. Conditions such as pulmonary embolism, tension pneumothorax, and pericardial tamponade.
  • Respiratory/metabolic shock - caused by a disruption in the ability of cells to utilize oxygen effectively. Certain poisons such as cyanide and carbon monoxide can cause this type of shock.
19
Q

29-6 Differentiate between early (compensated) and late decompensated/irreversible) signs of shock.

A

Compensated shock is where shock is developing, but the body is still able to compensate for it by releasing adrenaline, raising the heart rate and force of contractions, constricting the capillaries and constricting the blood vessels, increase in respirations.
Decompensated shock is when the body can’t keep producing the adrenaline, The heart continues to try to beat faster but the blood vessels, even constricted, are not full and therefore blood pressure starts failing and their mental status starts failing.
Irreversible is when the heart slows down as it can no longer keep up and the patient’s pulse finally goes away into cardiac arrest.

20
Q

29-7 Explain how compensatory mechanisms to shock are maintained through direct nerve stimulation and release of hormones.

A
  • Direct nerve stimulation happens when the sympathetic nervous system is activated and sends signals to the heart and blood vessels in an attempt to maintain normal BP. These effects are Increased heart rate; Increase in the force of contractions, Constriction of the blood vessels (increased SVR)
  • The adrenal glands also release hormones epinephrine and norepinephrine. This produces a more sustained sympathetic effect. epinephrine stimulates both alpha and beta receptors, while norepinephrine affects mostly alpha receptors. The stimulation of the beta receptors affects the heart and causes an increase in strength and constriction. The stimulation on the alpha receptors affects the constriction of the blood vessels.
21
Q

29-8 Describe the progression of shock through the compensated, decompensated (progressive), and irreversible stages.

A

Early S/S of shock are increased pulse, increased breathing and pale skin. This is in Compensated Shock.
A major sign of entering decompensated shock is is a significant drop in blood pressure, in addition the patient will present with a significant decrease in mental status.
During irreversible shock the patient’s pulse becomes very weak until it finally goes away and they experience cardiac arrest.
Vital signs:
*Compensated
*Increased respirations
*Increased pulse
*Normal to low Systolic
*Normal to high diastolic
*Skin is pale, cool, moist
*Pupils are sluggish but responsive
*Mental Status is slightly altered
*Decompensated
*Decreased Respirations
*Pulse becomes weaker
* Blood Pressure decreasing to absent
*Skin is pale, cool, moist
*Pupils are sluggish to fixed and dilated
*Mental Status is very altered to unresponsive

22
Q

29-9 Explain the influence of age on the assessment and management of patients with shock.

A

In pediatric patients they can compensate till nearly half of their blood is gone. Their condition deteriorates very quickly and death is sudden. Initiate care and transport as soon as possible.
* In Geriatrics may show signs of shock more easily than young people, Their blood vessels are stiffer and less responsive to sympathetic stimulus. They may also be on beta blockers whick won’t allow their heart to become tachycardic as expected. As a result they may progress to decompensated shock more quickly.

23
Q

29-10 Explain how to identify the patient who is in a shock state and demonstrate the assessment of patients to identify shock

A

During primary assessment obtain baseline vitals if possible and give oxygen asap. Look for signs of shock at that moment and with future vitals every 5 minutes with unstable medical or Trauma patients with significant MOIs

24
Q

20-11 Discuss the pre-hospital management of patients with shock.

A
  • Conduct an appropriate Scene Size-up and BSI
  • Manually stabilize spine as appropriate for MOI
  • Perform a primary assessment and initiate supplemental oxygen.
  • Control any external bleeding
  • Keep the patient supine, If the cause of shock is due to illness, you may elevate the legs 8-12 inches.
  • Conserve Body heat by covering the patient with a blanket when appropriate.
  • Place the patient on a long backboard as appropriate for the MOI
  • Initiate Transport and request an ALS intercept if available
  • Perform reassessments as appropriate and be alert for changing vital signs (trending).