Cholelithiasis Flashcards
Cholecystitis
- inflammation of gallbladder- assoc w obstr. From stones or biliary sludge
Cholelithiasis
gallbladder stones from bile salts, cholesterol, Ca+
Can remain in gallbladder or migrate to common bile duct, cystic duct
Population for Cholelithiasis
Higher in white, multiparous women over 40.
High incidence in Navaho + pima.
Related to sedentary lifestyle/genetic/obesity
Clinical Manifestations of Cholelithiasis
Can be silent, severity depends on stones/obstruction
Restlessness,
diaphoresis,
NV
Physical Manifestations of Cholelithiasis
Indigestion/pain in RUQ, deferred to R shoulder/scapula.
Adb rigidity
Biliary Colic
spasm 2nd to movt of stones or obstr. Severe pain for 1 hr. tachy.sweat. Residual RUQ tenderness Pain 3-6 hrs after heavy meal or when laying down
Total Biliary Obstruction
- Dark amber urine, clay stools
(steatorrhea) - pruritus, jaundice, bleeding
Labs associated with Cholelithiasis
CBC (inflammation) Bilirubin (obstruct)
Alkaline phosphatase-ALT/AST. Amylase (pancreas involved)
Pain control for cholelithiasis
Analgesics: Ketorolac
Non surgical treatment for cholelithiasis
Fluid replacement, anticholinergic (atropine) antibiotics, diet, ercp, surgery, lithotripsy
Surgical treatment for cholelithiasis
Cholecystectomy- incision
o Choledocholithotomy
Choledocholithotomy
– incision in common bile duct to remove stone
Post op treatment for Choledocholithotomy
respp., cough/deep breath,
early abulation,
antiemetics,
NPO (NG tube for decompression)
Cirrhosis
chronic progressive disease- breakdown of hepatocytes, leads to scar tissue
Types of cirrhosis
EtoH/portal/nutritional- most common, related to nutrition
Biliary causes- primary biliary cirrhosis/primary sclerosing cholangitis
Cardiac- chronic rhf