Chapter 9 and 10 Flashcards
what does amniotic fluid do to the fetal lungs
inhalation helps growth of fetal lung tissue, absorption accelerates during labor, birth and a few hours after which means decreased pulmonary resistance to blood flow initating air breathing
What do chemical factors do to initiate first breath
hypercarbia, acidosis, hypoxia - these factors stimulate respiratory system of brain to initiate breathing
what do sensory factors do to initiate the first breath
overwhelming new stimuli like tactile visual olfactory and auditory
what do thermal factors do to initiate first breath
drastic change in temp stimulates respiratory response to prevent cold stress and respiratory depression need to dry in place infant skin to skin
what do mechanical factors due to initiate first breath
fetal chest compression thoracic squeeze during vaginal delivery pushes fluid out of lungs, chest recoil after newborn trunk delivered creates intrathoracic pressure the air replaced the fluid that was squeezed out remaining lung fluid pushed into lymphatic and circulatory system and absorbed within a few hours this doesn’t happen in C-section so respiratory distress
what are some complications from O2 therapy
Broncopulmonary dyspnea and retinopathy of prematurity
what is a L/S ratio and how do you get it
lectin-to-sphingomyelin - Mature longs =2:1, less then 35 wks= less lactin, An amniocentesis can be done to get ratio
what happens to the fetal lungs after birth
lung expansion after birth stimulates surfactant release which decrease in surface tension within alveoli thus preventing alveolar collapse
what are some factors that interfere with fetal breathing
prematurity, birth asphyxiation can adversely affect lung compliance and surfactant production, respiratory distress, translate tachypnea of newborn
ss of respiratory distress syndrome
grunting, accessory muscle, retractions, nasal flaring, tachypnea
what is the treatment for respiratory distress syndrome
air maintenance and oxygenation by humidified O2 continuous positive airway pressure (CPAP) it is less invasive than intubation, but want to wean ASAP so they don’t become dependent
what is transient tachypnea of newborn
aka respiratory distress syndrome type 2 - delayed clearance of fetal lung fluid (usually resolves 2-3 days)
what are the risk factors for transient tachypnea of newborn
c-section, large baby, late preterm (34-36wks)
how is transient tachypnea of newborn
via blood gasses showing respiratory acidosis
what are the ss of transient tachypnea of newborn
tachypnea, grunting, retractions, cyanosis, nasal flaring
what is the treatment for transient tachypnea of newborn
hold oral feeds over 6oz to decrease risk of aspiration, CPAP at 40% for 24-48 hours
what does hypoxia trigger
impulses from chemoreceptors to stimulate respiratory center in medulla
what does sudden temp change trigger
sensors in the skin to send impulses that stimulate breathing
when does the foramen ovale and how
closes when the pressure in the LA is higher then RA pressure, semi closes 1-2 hours after birth and permanent closure by 6mths
when and how does the ductus arteriosus close
constricts preventing deoxygenation blood from pulmonary artery from entering aorta, usually closes within 72 hours with incomplete closure you can hear murmur in 2nd intercostal space/left sternal border, permeant closure at 3-4 wks and then called ligamentum arteriosum
when and how does the ductus venosus close
clamping umbilical cord occludes blood flow into umbilical circulation so closes ductus venosus which forces blood flow to liver, name is now ligamentation venosum by first week
what position do premies need to maintian body temp
flexed position
what increases heat production in babies
peripheral vasoconstriction , nonshivering thermogenesis
how do you prevent hyperthermia in newborns
sweatglands no developed until 1 mth so the baby does peripheral vasodilation and evaporation of insensible H2O loss
what are the effects of cold stress
Increase O2 requirements may result of worsening respiratory distress, decreased surfactant = collapsed alveoli and further respiratory distress, hypoglycemia common metabolic acidosis the increase of metabolism of glucose and decrease pressure of O2 related to increased demand for O2 leading to acid production, jaundice from metabolism of brown fat produces fatty acids that interfere with transport of bilirubin to liver
what is the neutral thermal environment of a newborn
the range of temp and which newborns body temp can be maintained with minimal metabolic demands and O2 consumption, most newborns can stabilize temp within two to three hours
what is non shivering thermogenesis
uses brown adipose tissue to provide heat and cold stress newborns, formation of brown adipose tissue begins at 26-30 weeks
where is brown adipose tissue located on newborns
mid scapular area, neck, axillae, deeper deposits around trachea, esophagus, abdominal aorta, kidneys and adrenal gland it is highly vascular fat
how much blood volume does a full term newborn have and what is it determined by
80-90ml/kg determined by time of clamping delayed clamping is more blood
where does erythropoiesis occur in an infant
in the liver for six months then hematopoiesis occurs in bone marrow lifespan is 90 days
what do glucose and glycogen levels do in a newborn
during last 4-8wks glucose is stored as glycogen in liver and muscles the infant used the glycogen stores for energy but depleted at 12 hours of life, glucose levels decrease during first three hours of life and gradually increase over the next 3-4hrs to reach steady state of 40-60
what is the treatment for hypoglycemia
feeding - it can be difficult because of the signs and symptoms, so if not send to NICU or give Iv glucose
what are the pros of delayed cord clamping
prevents anemia, potentially improved transition related to increased pulmonary perfusion, additional iron stores
what are the cons of delayed cord clamping
increase risk for jaundice related to increased volume of erythrocytes and possible polycythemia (lots of RBC’s they will look red)
what is the value in signs and symptoms of hypoglycemia in newborns
less than 35, jitteriness, apnea, seizures, lethargy, hypothermia, tachypnea, poor suck, requires immediate attention to prevent brain damage
what is pathologic jaundice and when should you treat
prevents at birth or within the 1st 24 hours of life related to underlying medical cause need treatment when bilirubin is over 13 for term or over 15 for preterm
what are the signs and symptoms of jaundice
yellow color of the skin, sclera, oral mucous membranes
what is psychologic jaundice and what is it caused by
occurs after first 24-48hrs total serum bilirubin over five to seven peak bilirubin level between 3rd and 5th day caused by liver immaturity, delayed feeding, cold stress, excessive bruising
How do you prevent psychological jaundice
maintaining temp, encourage feedings, monitor stools
what is breast milk jaundice
light on set after first week of life, peaks around day 10, the bilirubin levels continue to rise and peaks two to three weeks, infant eating well, stooling well and no signs and symptoms of hemolysis
what is the treatment for breast milk jaundice
phototherapy infant has to wear eye Shields cover genitals assess hydration by bowel movement and pee, take clothes off, monitor temp, turn off to feed, watch for skin breakdown
what is breastfeeding jaundice
early onset jaundice occurs from decreased intake which causes decreased meconium
what are the interventions for breastfeeding jaundice
initiate breastfeeding ASAP 11:50 feeds per day, assess latch and suckle