Chapter 8 Aphasias Flashcards

1
Q

Argument for classifying aphasias

A

Different areas of the brain control different functions so,
different types of aphasias have different lesion sites and,
different lesion sites produce distinctively different types of symptoms
Different types of aphasias require different forms of Tx

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2
Q

Arguments against classifying aphasias

A

Brain is thought to function as integrated unit in controlling language

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3
Q

Factors used in classifying aphasias

A
Speech fluency
Paraphasias
Repetitions
Language comprehension
Site of lesion
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4
Q

Classification by fluency

A

Non-Fluent and Fluent aphasias

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5
Q

Non Fluent aphasias

A
Speaking is slow, halting, and with effort:
Broca's
Transcortical Motor Aphasia (TMA)
Mixed Transcortical Aphasia (MTA)
Global Aphasia
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6
Q

Fluent Aphasia

A
Speaking is smooth and effortless:
Wernicke's
Transcortical Sensory Aphasia (TSA)
Conduction Aphasia
Anomic Aphasia
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7
Q

Classification by comprehension

A

Receptive Aphasia & Expressive Aphasia

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8
Q

Receptive Aphasia

A

Primary difficulties are in area of comprehension

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9
Q

Expressive Aphasia

A

Primary difficulties are in the area of expression

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10
Q

Classification by site of lesion

A

Damage to the cortical centers for language (served by the middle cerebral artery)
Damage to the association fiber tracts important for language
Damage to the subcortical area
Anomic aphasia does not have a clear localization

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11
Q

Aphasia caused by damage to the cortical area

A

Broca’s, Wernicke’s, and Global

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12
Q

Aphasia caused by damage to the association fiber tract

A

Conduction & Transcortical sensory, motor, and mixed aphasias

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13
Q

Broca’s (motor) Aphasia

A

Damage to lower part of the premotor cortex important for planning and performing expressive language
Damage to the anterior portion of the middle cerebral artery
Often accompanied by right sided hemiplegia or hemiparesis

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14
Q

Broca’s aphasia language characteristics

A
Nonfluent 
Agrammatic
Monotonous speech 
May have misarticulations 
Lacking function words
Impaired repetition
Written language similar to spoken 
Comprehension better than their expressive spoken and written language
Good self-monitoring
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15
Q

Wernicke’s (sensory) aphasia

A

Damage to posterior superior temporal lobe of language dominant hemisphere
Damage to posterior branch of the cerebral middle artery

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16
Q

Wernicke’s aphasia language characteristics

A
Fluent
Impaired auditory comprehension is dominant characteristic
Paraphasias, neologisms, jargon
Empty speech
May have circumlocution
Press of speech (logorrhea)
Handwriting resembles speech
If aphasia is mild, may be aware of errors
Less concern with speech than Broca’s
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17
Q

Global aphasia

A

Damage to trunk of middle cerebral artery which causes massive damage to the entire perisylvian area
Broca’s or Wernicke’s sometimes spared
Severe deficits are demonstrated in all areas of language function

18
Q

Global aphasia language characteristics

A

Language limited to few words, exclamations
stereotypical utterances
overlearned phrases (How are you?). May perseverate on these words,
Severely impaired comprehension
Reading and writing also terrible
Usually attentive, alert socially appropriate – different than dementia
May be able to perform nonverbal tasks OK – picture matching, etc.
Performance is better when dealing with personal information

19
Q

Conduction aphasia

A

Rare (5-10%), Probable sites of lesions: lesions in insula,

upper temporal or lower parietal lobe Damage arcuate fasiculus and /or supramarginal gyrus.

20
Q

Conduction aphasia language characteristics

A

Wernicke’s thought by most to be spared Comprehension somewhat spared
Most dominant feature - great difficulty with repetition
Multisyllabic words more difficult than monosyllabic words
Repetition impairment is disproportionate to other impairments.
Rate, intonation and stress are usually normal
May produce paraphasias.
Difficulties reading out loud because oral reading depends on communication between Wernicke’s and Broca’s.
Word retrieval problems are usually quite evident – resulting in inappropriate pauses and/or empty speech.
Writing is usually legible but may contain spelling errors & letter omissions.
Alert and attentive and aware of errors.
self-correction aren’t typically successful

21
Q

Transcortical aphasias

A

Spares Broca’s, Wernicke’s, and arcuate fasciculus

Defining characteristic is that repetition is preservered

22
Q

Transcortical Motor Aphasia (TMA)

A

Damage to anterior superior frontal lobe and association pathways
Areas affected are supplied by anterior cerebral artery and anterior branch of middle cerebral artery

23
Q

TMA language characteristics

A

Markedly reduced speech output
Good repetition (this differentiates it from Broca’s)
Good auditory comprehension
Disturbed writing
Difficulty initiating speech- inertia
May have right sided hemiparesis or hemiplegia
Attentive and alert

24
Q

Transcortical Sensory Aphasia (TSA)

A

Damage to posterior temporoparietal region sparing Wernicke’s

25
Q

TSA language characteristics

A

Preserved repetition – unlike Wernicke’s
No problem with inertia in initiating speech (unlike TMA)
May often have echolalia
Lesion isolates Wernickes area from rest of brain, so major deficits in comprehension of spoken & written language occur.
Visual cortex is isolated from Wernicke’s causing difficulty in reading.
Fluent but empty speech (like Wernicke’s)
Don’t have press of speech (not like Wernicke’s)
Difficulty following commands

26
Q

Mixed Transcortical Aphasia (MTA)

A

Rare, Damage that spares the major language areas but separates them from rest of brain
Damage often occurs in watershed area
Often occurs following conditions that reduce blood flow through cerebral arteries such as: carbon monoxide poisoning, cerebral hypoxia, etc.

27
Q

(MTA) language characteristics

A

Similar to global aphasia except with better repetition
Parrot like repetition of what others say - like an echolalia
Nonfluent - very limited spontaneous output
NO functional auditory comprehension
Severely impaired writing and reading skills

28
Q

Anomic aphasia

A

Naming difficult, no clear localization, damage may occur to angular gyrus or inferior temporal gyrus

29
Q

Anomic aphasia language characteristics

A

Sever word retrieval
May have mild auditory comprehension impairment
Circumlocution and substitutions
Reading and writing is good

30
Q

Subcortical aphasias

A

Anterior syndrome
Posterior syndrome
Anterior-posterior syndrome
Thalamic aphasia

31
Q

Anterior syndrome

A

Caused by damage to internal capsule & lenticular process extending into anterior white matter

32
Q

Anterior syndrome language characteristics

A
Hemiplegic
Slow dysarthric speech
Good comprehension
Good repetition
Poor oral reading and writing
33
Q

Posterior syndrome

A

Damage to putamen and internal capsule extending into posterior white matter

34
Q

Posterior syndrome language characteristics

A
Hemiplegic
Fluent speech without dysarthria
Poor comprehension
Good single word repetition
Impaired reading and writing
Poor confrontational naming
35
Q

Anterior-posterior syndrome

A

Damage to internal capsule and putamen with both anterior and posterior extensions of white matter

36
Q

Anterior-posterior language characteristics

A

Mixture of symptoms consistent with both Broca’s and Wernicke’s aphasia.

37
Q

Thalamic aphasia

A

Damage to the thalamus

38
Q

Thalamic aphasia language characteristics

A
Hemiplegic
Difficulty initiating speech
Speech is echolalic and neologistic
Auditory comprehension and reading is good
Writing is impaired
Word-finding problems are common
39
Q

Aphasia a with fair to good comprehension

A

Broca’s, conduction, anomic, and transcortical motor

40
Q

Aphasia with poor comprehension

A

Wernicke’s, transcortical sensory, global

41
Q

Aphasias with poor repetition

A

Broca’s (labored), global