Chapter 55 Flashcards
Causes of acute gastritis?
NSAID, alcohol, radiation, smoking, stress, infection of H.pylori, staph, strep, e.coli, salmonella
Difference in origins between Type A chronic and Type B chronic
Type A is genetic, autosomal pattern , presence of antibodies to parietal cells and intrinsic factor
Causes of local irritation in chronic Type B?
Caffeine, ETOH, Radiation, smoking
Bacteria most often associated with chronic gastritis?
helicobacter pylori
Features of acute gastritis?
rapid onset nausea, vomiting hematemesis (vomit blood) gastric hemorrhage dyspepsia anorexia
Features of chronic gastritis?
vague reports of epigastric pain that is related to foods anorexia nausea vomiting intolerance of fatty or spicey foods pernicious anemia
What is pernicious anemia?
deficiency of vitamin B12
Interventions for chronic gastritis?
eliminate the disease eliminate H. pylori H2 agonist antacids proton pump inhibitors
Name the H2 agonists
famotidine
nizatidine
What class of medds is used as a buffering agent?
antacids, magnesium hydroxide, aluminum hydroxide, simethicone
Name the proton pump inhibitors
omeprazole
pantoprazole
Whats does H2 agonist do?
block gastric secretions
What does proton pump inhibitors do?
suppress gastric acid secretions
Hematemesis?
vomiting bright red blood or coffee-ground blood
What does hematemesis indicate?
bleeding above the duodenojejunal junction, referred to as an upper GI bleed
what is melena?
dark tarry stool
symptoms of a hemorrhage due to ulcers
Vomits bright red or coffee-ground blood, black tarry stool
What is a perforated ulcer?
entire thickness of stomach or duodenum is worn away
Symptoms of perforation
sudden sharp pain starts at the mid-epigastric region and spreads over entire abdomen, patient may be in fetal position
Danger of perforation?
leakage into the peritoneal cavity causing bacterial septicemia and hypovolemic shock