Chapter 5: Psychopathology Flashcards

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1
Q

define psychopathology.

A

the study of mental disorders and unusual or maladaptive disorders.

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2
Q

what is statistical infrequency?

A

this occurs when an individual has a less common characteristic, for example being more depressed or less intelligent than most of the population.
any relatively usual behaviour or characteristic thought of as ‘normal’, and any behaviour that is different to this is ‘abnormal’.

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3
Q

what is an example of statistical infrequency?

A

IQ and intellectual disability disorder.
in any human characteristic, the majority of peoples scores will cluster around the average, and the further we go above or below the average, the fewer people will attain that score.
the average is set at 100. those that range from 85-115 are seen as ‘normal’ however those above or below those values are seen as abnormal.

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4
Q

what is deviation from social norms?

A

this concerns behaviour that is different from the accepted standards of behaviour in a community or society.
a social norm is how we expect society to behave without telling them what to do to behave. Those who deviate from these social norms and offend peoples sense of what is ‘acceptable’ are defined as abnormal.

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5
Q

what is failure to function adequately?

A

this occurs when someone is unable to cope with ordinary demands of day-to-day living.
a person may cross the line between ‘normal’ and ‘abnormal’ at the point when they can no longer cope with the demands of everyday life and they fail to function adequately.
we might decide that someone is not functioning adequately when they are unable to maintain basic standards of nutrition and hygiene or perhaps they cannot hold down a job or maintain relationships with people around them.

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6
Q

who proposed the signs that are used to determine when someone is not functioning adequately, and what are they?

A

rosenhan & seligman (1989).
the signs include:
-a person no longer conforms to standard interpersonal rules, for example maintaining eye contact and respecting personal space.
-a person experiences severe personal distress.
-a person’s behaviour becomes irrational or dangerous to others and themselves.

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7
Q

what is deviation from ideal mental health?

A

this occurs when someone does not meet a set of criteria for good mental health.
when there is a picture of how we should psychologically healthy then we can begin to identify who deviates from this ideal.

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8
Q

who suggested the criteria to say whether we are in good mental health, and what is it?

A

marie jahoda (1988).
the following criteria:
-we can cope with stress.
-we have good self esteem and lack guilt.
-we have a realistic view of the world.
-we can successfully enjoy our leisure.
-we are independent of other people.

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9
Q

what are phobias?
how are they characterised?

A

an irrational fear of an object or situation.
phobias are characterised by excessive fear and anxiety, triggered by an object, place or situation.

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10
Q

how can phobias be categorised?

A

they can be categorised by specific phobias or social anxiety.
specific phobias in the sense that there’s such a thing as a phobia of an object or a situation.
social anxiety in the sense that there’s such a thing as a phobia of a social situation or being in a public place.

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11
Q

what are the 3 characteristics of phobias?

A

behavioural
emotional
cognitive

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12
Q

what are the behavioural characteristics of phobias? (3)

A

this is the way those with phobias act.
panic- a phobic person may panic in response to the presence of the phobic stimulus. panic may involve a range of behaviours such as crying, screaming or running away. children can cling or have a tantrum as a reaction.
avoidance- the sufferer tends to go to a lot of effort to avoid coming into contact with the phobic stimulus. this makes it hard to go about daily life.
endurance- a sufferer remains in the presence of the phobic stimulus but continues to experience high levels of anxiety. this can be unavoidable in some situations.

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13
Q

what are the emotional characteristics of phobias? (1)

A

anxiety- phobias involve an emotional response of anxiety and fear. it is an unpleasant state of high arousal. this prevents the sufferer from relaxing and makes it very difficult to experience any positive emotion. anxiety can be long-term. fear is the immediate and extremely unpleasant response.

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14
Q

why are emotional responses seen as unreasonable?

A

the emotional responses we experience in relation to phobic stimulus go beyond what is reasonable.
e.g a person has a fear of spiders and this will involve a very strong emotional response to a tiny and harmless spider. though this is widely disproportionate to the danger posed by any spider this person is likely to see in a shed.

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15
Q

what are the cognitive characteristics of phobias? (3)

A

this refers to the process of thinking a phobic has while viewing their phobic stimulus. selective attention- if a sufferer can see the phobic stimulus it is hard to look away from it. keeping our attention on something really dangerous is a good thing as it gives us the best chance of reacting quickly to a threat, but this is not so useful when the fear is irrational.
irrational beliefs- a phobic may hold irrational beliefs in relation to phobic stimuli. e.g ‘i must always sound intelligent.’ this kind of belief increases the pressure on the sufferer to perform well in social situations.
cognitive distortions- the phobics’ perceptions of the phobic stimulus may be distorted.

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16
Q

what are the behavioural characteristics of depression? (3)

A

activity levels- sufferers of depression have reduced levels of energy making them lazy. in extreme cases, this can be so severe that the sufferer cannot get out of bed.
disruption to sleep- sufferers may experience reduced sleep or increased need for sleep.
eating behaviour- appetite may increase or decrease, leading to weight gain or loss.

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17
Q

what are the emotional characteristics of depression? (2)

A

lowered mood- more pronounced than the daily experiences of feeling lazy or sad. sufferers often describe themselves as ‘worthless’ or ‘empty’.
anger- on occasion, such emotions lead to aggression or self-harming behaviour.

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18
Q

what are the cognitive characteristics of depression? (2)

A

poor concentration- sufferers may find themselves unable to stick with a task as they usually would, or they might find simple decision making difficult.
absolutist thinking- ‘black and white thinking’, when a situation is unfortunate it is seen as an absolute disaster.

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19
Q

what are the behavioural characteristics of OCD? (2)

A

compulsions- actions that are carried out repeatedly. the same behaviour is repeated in a ritualistic way to reduce anxiety.
avoidance- the OCD is managed by avoiding situations that trigger anxiety.

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20
Q

what are the emotional characteristics of OCD? (2)

A

anxiety and distress- obsessive thoughts are unpleasant and frightening, and the anxiety that goes with these can be overwhelming.
guilt and disgust- irrational guilt, e.g over a minor moral issue, or disgust which is directed to oneself.

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21
Q

what are the cognitive characteristics of OCD? (2)

A

obsessive thoughts- about 90% of OCD sufferers have obsessive thoughts, e.g about being contaminated with dirt.
insight into excessive anxiety- awareness that thoughts and behaviour are irrational. in spite of this, sufferers experience catastrophic thoughts and are hypervigilant of their obsession.

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22
Q

what is used to explain the behavioural approach in phobias?

A

the two-process model.

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23
Q

what is the two-process model?

A

where phobias are learned by classical conditioning and maintained by operant conditioning.

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24
Q

what is classical conditioning in regards to phobias? include an example

A

classical conditioning involves learning by association.
e.g an UCS, being bitten, triggers a fear response, where fear is a UCR.
the NS is the dog at first, creating no response.
but once the UCS and NS are paired together, it creates the UCR, fear.
the NS, the dog, becomes a CS producing fear, which is now a CR.

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25
Q

explain the Little Albert study.

A

watson and raynor showed how fear of rats could be conditioned in ‘little albert’.
-whenever Albert played with a white rat, a loud noise was made close to his ear. the noise (UCS) caused a fear response (UCR).
-the rat (NS) did not create fear unti the bang and the rat had been paired several times.
-Albert showed a fear response (CR) every time he came into contact with the rat, now a CS.

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26
Q

what occurred as a consequence of the Little Albert study?

A

Albert now generalised his fear to other stimuli, e.g white fury objects, including Santa Claus.

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27
Q

what is operant conditioning in regards to phobias?

A

operant conditioning takes place when our behaviour is reinforced or punished.
negative reinforcement is used in particular. this is where an individual produces behaviour that avoids something unpleasant.
when a phobic avoids a phobic stimulus, they escape the anxiety that would have been experienced.
this reduction in fear negatively reinforces the avoidance behaviour and the phobia is maintained.

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28
Q

what two behavioural approaches are used to treat phobias?

A

systematic desensitisation
flooding

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29
Q

what is systematic desensitisation (SD)?

A

a type of therapy which aims to gradually reduce anxiety through counterconditioning.

30
Q

what is SD based on?

A

classical conditioning, where instead of a phobic stimulus produces fear, it’s paired with relaxation and that becomes a new conditioned response.

31
Q

what is reciprocal inhibition?

A

where it is not possible to be afraid and relaxed at the same time and so one emotion prevents the other.

32
Q

what is the formation of an anxiety hierarchy?

A

where the patient and therapist design an anxiety hierarchy which is a list of fearful stimuli arranged in order from least to most frightening.

33
Q

how is relaxation involved in the anxiety hierarchy?

A

the phobic individual is first taught relaxation techniques and this is demonstrated into the anxiety hierarchy where the patient works up through each level in a relaxed state.
this takes place over several sessions starting at the bottom of the hierarchy.

34
Q

when do we know if treatment of SD is successful?

A

when the patient can stay relaxed in situations high on the hierarchy.

35
Q

what is flooding?

A

immediate exposure to the phobic stimulus.
the phobic patient is bombarded with the phobic patient with the phobic patient without a gradual build-up.

36
Q

how is flooding able to treat phobias?

A

without the option of avoidance behaviour, the patient quickly learns that the phobic object is harmless through the exhaustion of their fear response. this is known as extinction.

37
Q

what are the two cognitive approaches to explaining depression?

A

Beck’s cognitive theory of depression.
Ellis’s ABC model.

38
Q

what is beck’s cognitive theory of depression?

A

beck suggested that some people are more prone to depression because of faulty information processing, where they think in a flawed way.
they attend to the negative aspects of a situation and ignore positives. they tend to blow small problems out of proportion and think in ‘black and white’ terms.

39
Q

what are negative self-schemas?

A

this is something depressed people have. they interpret all information about themselves in a negative way.

40
Q

what is the negative triad?

A

a person develops a dysfunctional view of themselves because of three types of negative thinking that occur automatically.

41
Q

what are the 3 elements of the negative triad? include examples.

A

negative views of the world, e.g ‘the world is a cold hard place’.
negative views of the future, e.g ‘i’ll never get a job’.
negative views of the self, e.g ‘i am a failure’.

42
Q

what is Ellis’s ABC model?

A

Activating events
Beliefs
Consequences

43
Q

what are ‘activating events’ in Ellis’s ABC model? include an example.

A

where depression arises from irrational thoughts.
according to Ellis depression occurs when we experience negative events, e.g failing an important test.

44
Q

what are ‘beliefs’ in Ellis’s ABC model? include an example.

A

where negative events trigger irrational beliefs. the event may not be negative but you have irrational interpretations of the event, e.g your friend didn’t say ‘hi’ to you and so you believe that they never dislike you and never want to talk to you again.

45
Q

what are ‘consequences’ in Ellis’s ABC model? include an example.

A

when an activating event triggers irrational beliefs, there are emotional and behavioural consequences, e.g you will now ignore your friend and delete their number since they don’t wish to talk to you.

46
Q

what are the two cognitive approaches to treating depression?

A

Beck’s Cognitive Behaviour Therapy (CBT)
Ellis’s Rational Emotive Behaviour Therapy (REBT)

47
Q

what is Beck’s CBT?

A

where the patient and therapist work together to clarify the patient’s problems.
they identify where there might be negative or irrational thoughts that will benefit from challenges.

48
Q

how is challenging negative thoughts in negative triad beneficial in CBT?

A

identifying the negative thoughts about the self, the world and the future and the patient challenging these thoughts takes an active role in their treatment.

49
Q

what does the ‘patient as scientist’ mean in CBT?

A

where patients are encouraged to test the reality of their irrational beliefs.
they may be given homework e.g to record when people were nice to them.
in future sessions if patients say no-one is nice to them, the therapist can produce this evidence to prove that the patient’s belief is incorrect.

50
Q

what is Ellis’s REBT?

A

REBT extends to the ABC model to an ABCDE model.
D is for dispute; challenging irrational beliefs.
E is for effect.

51
Q

how challenging irrational beliefs work (abcDe)?

A

a patient may talk about how unlucky they have been and an REBT therapist would percieve this as an irrational belief and challenge it. they would use two arguments to go against the belief; an empirical argument and a logical argument.

52
Q

what is an empirical argument?

A

disputing whether there is evidence to support the irrational belief.

53
Q

what is a logical argument?

A

disputing whether the negative thought follows from the facts.

54
Q

what is behavioural activation in REBT?

A

as individuals become more depressed, they tend to increasingly avoid situations and become isolated, which maintains or worsens symptoms.
behavioural activation aims to decrease their avoidance and isolation and increase their engagement in activities that have been shown to improve mood, e.g exercising or going out to dinner.

55
Q

what are the two biological approaches to explaining OCD?

A

genetic explanations
neural explanations

56
Q

explain candidate genes as a genetic explanation of OCD.

A

researchers have identified specific genes which create a vulnerability for OCD, these are candidate genes.
some of these genes are involved in regulating the development of the serotonin system.

57
Q

what is the COMT gene?

A

this may contribute to OCD. it regulates the production of the neurotransmitter dopamine that has been implicated in OCD. it produces higher levels of dopamine which regulates the mood in OCD.

58
Q

what is the SERT gene?

A

this affects the transport of the serotonin, creating lower levels of this neurotransmitter. these lower levels are also implicated in OCD.

59
Q

what is the diathesis-stress model?

A

where certain genes leave some people more likely to suffer a mental disorder but it is not certain.
some environmental experience is necessary to trigger the condition.

60
Q

how is the idea that ‘OCD is polygenic’ a genetic explanation for OCD?

A

OCD is not caused by one single gene but several genes are involved.
taylor (2013) found evidence that up to 230 different genes may be involved in OCD.

61
Q

how is the idea that there are ‘different types of OCD’ a genetic explanation for OCD?

A

one group of genes may cause OCD in one person but a different group of genes may cause the disorder in another person.
different types of OCD from these different genes may be the result of particular genetic variations, such as religious obsessions.

62
Q

explain ‘low levels of serotonin’ as a neural explanation for OCD.

A

neurotransmitters, serotonin, are responsible for relaying information from one neuron to another. and so if a person has low serotonin levels then normal transmission of mood-relevant info does not take place and mood is affected.

63
Q

how does the ‘decision-making systems in the frontal lobes being impaired’ act as a neural explanation for OCD?

A

some cases of OCD, e.g having a hoarding disorder, seem to be associated with impaired decision making.
this may be associated with abnormal functioning of the lateral frontal lobes of the brain, where the frontal lobes are responsible for logical thinking and making decisions.

64
Q

what is the biological approach to treating OCD?

A

drug therapy

65
Q

how is drug therapy effective in treating OCD?

A

it aims to increase/decrease levels of neurotransmitters in the brain or to increase/decrease their activity.
in this case, drugs works in various ways to increase the levels of serotonin in the brain.

66
Q

what are SSRIs and how do they work?

A

they are selective serotonin reuptake inhibitors.
they prevent the reabsorption and breakdown of serotonin in the brain. this increases its levels in the synapse and therefore serotonin continues to stimulate the postsynaptic neuron.
this compensates for whatever is wrong with the serotonin system in OCD.

67
Q

how is combining SSRI’s with CBT effective as treatment in OCD?

A

the drugs are able to reduce a patients emotional symptoms, such as feeling anxious or depressed.
this means patients can engage more effectively in CBT.

68
Q

what are the alternative drugs to SSRI’s?

A

tricyclics
SNRI’s

69
Q

what are tricyclics?

A

they are an older type of antidepressant. they have the same effect on the serotonin system, but produce more severe side-effects.
they are used as a second line of defence for patients who don’t respond to SSRI’s.

70
Q

what are SNRI’s?

A

they are serotonin-noradrenaline reuptake inhibitors. they increase levels of serotonin as well as a different neurotransmitter; noradrenaline. they are a second line of defence for patients who don’t respond to SSRI’s, like tricyclics.