Chapter 5 - Anxiety Disorders Flashcards

1
Q

What are the DSM-5 Anxiety Disorders?

A

-Separation Anxiety Disorder
-Selective Mutism
-Specific Phobia
-Social Anxiety Disorder
-Panic Disorder
-Agoraphobia
-Generalized Anxiety Disorder

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2
Q

What is Anxiety?

A

-response to perceived or anticipated threat (vs actual immediate threat - fear)

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3
Q

What are the physiological symptoms of anxiety?

A

-racing heart
-difficulty breathing
-GI distress
-sweating
-tense muscles
-trembling/shaking

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4
Q

What are the cognitive symptoms of anxiety?

A

-anticipate harm
-worry
-exaggerate danger
-difficulty concentrating
-hypervigilance

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5
Q

What are the behavioural symptoms of anxiety?

A

-escape
-avoidance

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6
Q

How does the Flight or Fight response work?

A

-when our brain detects danger, it sends a signal to mobilize our body to prepare
-sympathetic nervous system activated the body for fight or flight
-it is necessary for survival, adaptive
-can be helpful - prepare or motivate us

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7
Q

What is the Yerkes-Dodson Law?

A

-this law states that we have an optimal arousal level which results in optimal performance
-low arousal & weak performance = no motivation
-high arousal & weak performance = overwhelmed/distracted

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8
Q

When does anxiety become a disorder?

A

-when symptoms arise in the absence of real threat (unfounded fear) and it is…
–excessive, inappropriate, and/or pervasive
–causes significant distress and/or impairment

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9
Q

What is the prevalence of anxiety disorders?

A

-anxiety disorders are the most common mental health condition in the U.S.

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10
Q

How does the biological dimension explain anxiety?

A

-fear circuitry in the brain
-genetic influences

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11
Q

What is the Fear circuitry in the brain explanation of anxiety?

A

-brain structures implicated in anxiety disorders:
–amygdala, hippocampus, prefrontal cortex
–HPA activity triggers “fight-or-flight”
–overactivation may lead to AD

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12
Q

How does genetics influence anxiety?

A

-moderate heritability: numerous genes affect vulnerability
-neurotransmitter abnormality: variation in serotonin transporter gene associated with reduction in serotonin; increased anxiety and fear related behaviours
-behaviourally inhibited temperament (more shy/cautious)

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13
Q

How does the psychological dimension explain anxiety?

A

-behavioural factors
-cognitive factors

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14
Q

What are the behavioural factors of anxiety?

A

-classical conditioning: conditioned fear response in absence of danger; stimulus generalization (more feared stimuli)
-modeling feared responses

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15
Q

What maintains anxiety, according to the behavioural factors?

A

-avoidance or safety behaviours can reduce anxiety in the moment (negative reinforcement)
-safety behaviours: overt/covert actions performed to reduce distress associated with feared stimulus
-consequence: prevents learning of new information about the consequences of the feared event

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16
Q

What is negative appraisals (cognitive factors of anxiety)?

A

-interpreting [ambiguous] events as threatening):
-overestimation of likelihood of feared outcome;
-overestimation of severity (catastrophizing) if feared outcome were to occur
-being able to reappraise or look at a situation from different perspectives can minimize negative responses

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17
Q

What is anxiety sensitivity (cognitive factors of anxiety)?

A

-a tendency to interpret physiological changes in the body as signs of danger
-predicts development of panic attacks and anxiety disorders
-perceived threat –> anxiety –> increased autonomic activity –> symptoms interpreted as catastrophic (circle)

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18
Q

What are other cognitive factors that explain anxiety?

A

-limited sense of self-control and mastery (can’t tackle issues/threats)
-biased information processing/selective attention: increased attention toward threat; more quickly to notice feared cues even at low levels; results in selective attention –> threat-focused

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19
Q

How does the social and sociocultural dimension explain anxiety?

A

-stressful life events trigger existing vulnerabilities
-sociocultural factors: poverty, sex/gender
-exposure to discrimination and prejudice can increase anxiety

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20
Q

Why are women more frequently diagnosed with AD than men?

A

-women more anxious/worry more; more likely to report symptoms and seek treatment
-different exposure to trauma
-therapist bias in diagnosis

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21
Q

How is anxiety presented in different cultures?

A

-Western: social anxiety is fear of embarrassing oneself
-Asian: social anxiety is worries about being offensive to others
-Taijin Kyofusho (TKS): social anxiety in Japanese and Korean cultures

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22
Q

What are the DSM-5 Criterias for Specific Phobia?

A

-Marked fear/anxiety about a specific object or situation
-phobic object or situation almost always provokes immediate fear/anxiety
-phobic object or situation is actively avoided or endured with intense fear/anxiety
-the fear/anxiety is out of proportion to actual danger
-lasting 6 months or more
-cause significant distress or impairment
-not better explained by symptoms of another mental disorder

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23
Q

What are common specific phobias?

A

-animals (most common): insects; dogs; snakes
-natural environments: heights; storms; water
-situations: enclosed spaces; elevators; planes
-blood-injection-injury: needles; seeing blood
-other: clown; choking; vomiting

24
Q

What is the prevalence of specific phobia?

A

-lifetime prevalence: 12.5%
-onset childhood & early adolescence
-2x more common in women
-the specific fear can vary across different cultures
-only 8% receive treatment (avoidance & not causing distress)

25
Q

How does the biological dimension explain phobias?

A

-preparedness theory: it is easier for humans to develop fears to which we are physiologically predisposed
-fear of snakes > flowers

26
Q

What is the 2 step model of the behavioural perspective of phobias (psychological dimension)?

A
  1. Development: classical conditioning - a non-dangerous stimulus becomes associated with anxiety
  2. Maintenance: operant conditioning - avoidance works in the short-term, but it prevents the long-term extinction of learned anxiety; fear persists
27
Q

What are the evidence-based treatments for phobias?

A

-exposure therapy (most widely used)
Step 1: remove avoidance –> exposure to the feared situation
Step 2: unlearn the feared response: learn to pair relaxed response /habituation with feared stimulus (systematic desensitization)
-repeated many times until fear weakens/habituates

28
Q

What is the format of exposure therapy?

A

-in vivo exposure: exposure to the real feared stimulus (high drop out rate & not possible with all phobias)
-virtual reality exposure

29
Q

What is the pace of exposure therapy?

A

-gradual: develop fear hierarchy and start with moderately feared stimulus
-flooding: exposure at full intensity (fast-paced exposure)

30
Q

What is the success rate of exposure therapy?

A

-60-90% success rate (don’t show phobic response after therapy)

31
Q

What are the DSM-5 Criterias for Social Anxiety Disorder (SAD)?

A

-Marked fear/anxiety about one or more social situations in which the individual is exposed to possible scrutiny by others
-fears that they will act in a way or show anxiety symptoms that they will be negatively evaluated
-social situations almost always provoke fear/anxiety
-social situations are avoided or endured with intense fear/anxiety
-fear/anxiety is out of proportion to actual threat
-last 6 months or more
-cause significant distress or impairment
-not better explained by symptoms of another mental disorder

32
Q

What is the prevalence of SAD?

A

-lifetime prevalence: 12.1%
-onset adolescence
-2x more common in women

33
Q

How does the psychological dimension explain SAD?

A

-cognitive models: unrealistic high perfectionistic standards; interpret ambiguous social situations as negative; views of themselves as unattractive and socially unskilled
-hypervigilant for early cues of disapproval from others
-hypervigilant of own behaviour
-overestimate how poorly they performed in social situations

34
Q

How does the social dimension explain SAD?

A

-parental behaviours influence development of social anxiety in children: overprotections; lack of support for independence; punitive parenting style (harsh)
-victimization by peers during childhood: ongoing bullying maintain and exacerbate social anxiety

35
Q

How does the sociocultural dimension explain SAD?

A

-more common in collectivistic cultures: individual behaviours seen to reflect on entire family or group
-practice of using shame to influence behaviour

36
Q

What are the evidence-based treatments for SAD?

A

-CBT: 3 components
1. exposure to social fears
2. cognitive restructuring (challenging thoughts)
3. social skills and assertiveness training to help clients start conversations, communicate their needs, or meet the needs of others
-best long-term outcome

37
Q

What are the medications for SAD?

A

-tend not to be first line of treatment for all anxiety disorders: SSRIs, benzodiazepines
-why? prevents the experience of anxiety and learning new information about anxiety

38
Q

Which treatment method is the most effective for SAD?

A

-there is no significant difference between the treatments (whether just meds or therapy or both)
-combining treatments doesn’t increase effectiveness of treatment

39
Q

What is a Panic Attack?

A

-abrupt surge of intense fear/discomfort, reaches peak within 10 minutes, with >4 symptoms:
-palpitations, pounding heart, accelerated heart rate; sweating; trembling/shaking; sensations of shortness of breath; feelings of choking; chest pain or discomfort; nausea or abdominal distress; feeling dizzy, unsteady, light-headed; chills or heat sensations; paresthesia (numbness/tingling); derealization or depersonalization; fear of losing control or going crazy; fear of dying

40
Q

What are the DSM-5 criterias for Panic Disorder?

A

-Recurrent unexpected/un-cued panic attacks
-At least 1 of following for 1 month after a panic attack:
–persistent concern/worry about additional panic attacks or their consequences
–a significant maladaptive change in behaviour related to attacks

41
Q

What is the prevalence of Panic Disorder?

A

-lifetime prevalence 4.7%
-isolated panic attacks: 22.7%
-2x more prevalent in women than men
-panic attacks may be marker of severe psychopathology
-safety behaviours: reassurance seeking from doctor - many individuals visit the ER; medication; sitting close to an exit

42
Q

How is Agoraphobia linked to Panic Disorder?

A

-panic disorder often accompanied (but not always) by agoraphobia
-lifetime prevalence of agoraphobia: 1.3%

43
Q

What are the DSM-5 criterias for Agoraphobia?

A

-Marked fear/anxiety about 2 or more situations: using public transportation; being in open spaces; being in enclosed spaces; standing in line or being in a crowd; being outside of the home alone
-Fears escape might be difficult or help might not be available if panic-like symptoms or other embarrassing symptoms occur
-Situation almost always provoke fear/anxiety
-Situation is actively avoided or require presence of companion or is endured with intense fear/anxiety
-Fear is out of proportion to actual danger
-Last 6 months or more
-Cause significant distress or impairment

44
Q

How does the biological dimension explain Panic Disorders?

A

-fewer GABA and serotonin receptors among individuals with panic disorder
-GABA is an inhibitory neurotransmitter (slows the system down; having less increases anxiety)

45
Q

How does the psychological dimension explain Panic?

A

-anxiety sensitivity & interoceptive conditioning
–inaccurate cognitions and symptoms create a feedback loop that increases anxiety
–develop fear of internal bodily sensations (interoceptive)

46
Q

What are the evidence-based treatments for Panic?

A

-exposure therapy (specifically interoceptive exposure): induce physiological symptoms of panic in order to extinguish the conditioning
-CBT: include exposure exercises; identifying and correcting catastrophic thinking about bodily sensations

47
Q

What is the most effective treatment for Panic Disorder?

A

-a combination of CBT and interoceptive exposure

48
Q

What are the DSM-5 criterias for Generalized Anxiety Disorder (GAD)?

A

-Excessive anxiety and worry about a number of events/activities
-Difficult to control the worry
-Anxiety/worry associated with >3 symptoms, with some symptoms present for more days than not for 6 months: restlessness or feeling keyed up or on edge; easily fatigued; difficulty concentrating or mind going blank; irritability; muscle tension; sleep disturbance
-Cause significant distress or impairment

49
Q

What is the prevalence of GAD?

A

-lifetime prevalence 5.7% adults
-more common in women than men
-onset challenging to determine
-typically do not seek mental health treatment: individuals with GAD often report worries help avoid catastrophes

50
Q

How does the psychological dimension explain GAD?

A

-behavioural - worry as negative reinforcer: avoid more intense negative emotions or images; positive beliefs about worries
-cognitive - intolerance of uncertainty

51
Q

What are the evidence-based treatments for GAD?

A

-CBT: 60% showed significant symptom reduction that persisted for 12 months
-Mindfulness-based CBT: become more open and accepting of anxious thoughts and feelings; teaching to remain nonjudgmental in the presence of anxiety

52
Q

What is the relapse rate after CBT?

A

-18% relapse rate for GAD
-specific phobia + panic disorder have a relapse rate ~ 5%

53
Q

What are the medications for GAD?

A

-Benzodiazepines - issues with dependence (meant for short-term use): increases GABA
-Antidepressants (SSRIs) - lower risk of dependence

54
Q

What are the similarities across Anxiety Disorders?

A

-perceived threat or anticipation of future threat
-symptoms - physiological, cognitive, and behavioural
-impairment in functioning
-avoidance maintains anxiety

55
Q

What are the differences across Anxiety Disorders?

A

-source of distress
-content of thoughts and beliefs
-severity and duration
-specific cognitive vulnerability for certain anxiety disorders

56
Q

Why is Comorbidity a common theme across anxiety?

A

-50% of individuals with an AD meet criteria for another AD: symptom overlap; shared vulnerabilities
-75% of those with an AD meet criteria for another mental disorder: depression; substance use disorders
-also high rates of comorbidity with other medical conditions

57
Q

Why is low treatment seeking a common theme across anxiety?

A

-individuals with AD are less likely to seek treatment relative to individuals with other disorders: lack of recognition of anxiety symptoms; negative reinforcement
-comorbidity increase help seeking