Chapter 31: Trauma Induced Conditions Flashcards

1
Q

Post-trauma continuum of care

A

ICU-acute-inpatient-outpatient

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2
Q

Post-trauma OT interventions

A

Preparatory methods
Purposeful activity/occupation based
Education

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3
Q

Pediatric SCI incidence

A

1.99 times per 100,000 children
1455 new injuries per year

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4
Q

Pediatric SCI gender discrepancy

A

Boys are twice as likely to experience SCI than girls
- Risk takers, fast drivers, more likely to be impulsive

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5
Q

Causes of SCI

A

Traumatic (motor vehicle accident, violence (guns), falls, sports injury)
Medical (spinal tumor, spinal procedure, disease process)

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6
Q

Children are more likely to have what type of SCI and why?

A

Upper cervical (C1-C3) due to having a larger head in proportion to their body and weak ligaments

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7
Q

What level of SCI allows a child to use an adapted environmental control?

A

C6

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8
Q

What is the major muscle innervated by C7 that is a major help with ADLs?

A

Triceps
- Provides extension movement

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9
Q

What is the main focus of intervention for a child with a SCI?

A

Play
Community mobility
Emotional aspects
Social interaction
Self care
Bowel-bladder control

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10
Q

Pediatric TBI incidence

A

Approximately 1.7 million people per year
Ages most likely to incur TBI
Young children: 0-4 years
Teenagers: 15-19 years
Senior citizens: over 65 years

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11
Q

Traumatic causes of TBI

A

Falls
Car accidents
Sports related injuries
Non-accidental trauma
Violence-related

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12
Q

Acquired stroke causes of TBI

A

Anoxia
Arteriovenous
Malformation rupture
Tumor resection
Seizure activity
Infection
Metabolic disorders

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13
Q

Seizure foci resection

A

Taking out part of the brain to stop seizures from occurring

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14
Q

Infection (meningitis and encephalitis)

A

Brain inflammation

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15
Q

Metabolic disorders TBI

A

Condition leads to high BP or stroke that causes TBI

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16
Q

Functional prognosis of TBI

A

Severity
Location
Extent

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17
Q

Localized injury

A

Hit on one section of head

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18
Q

Diffuse injury

A

Brain shifts and rotates (worse)

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19
Q

Premorbid factors of TBI

A

Low SES
Behavior issues/poor academic performance

20
Q

Glasgow Coma Scale

A

Hope for improvement, measures how they improve over time

21
Q

Mild TBI brain injury

A

Loss of consciousness: <30 mins
Glasgow Coma Scale score : 13-15
Post-traumatic amnesia: <24 hours

22
Q

Moderate TBI brain injury

A

Loss of consciousness: 30 minutes-24 hours
Glasgow Coma Scale score: 9-12
Observable finding on EEG, CT, or MRI

23
Q

Severe TBI brain injury

A

Loss of consciousness: > 24 hours
Glasgow Coma Scale score: 3-8
Significant findings on EEG, CT, or MRI

24
Q

Rancho Levels of Cognitive Functioning

A

Progression of recovery

25
Q

What kind of impairments occur for a pediatric TBI?

A

Motor, neurological, cognitive
Return of function is much less predictable- younger can rewire better

26
Q

Pediatric burn injury incidence

A

Approximately 450,000 per year.
Over 50% of that number consists of children.
Approximately 30,000 children per year require hospitalization for burns.

27
Q

Causes of burns

A

Scald- 60% (steam or hot water)
Flame- 25%
Contact- 10%
Electrical/chemical- 5%

28
Q

First degree burns

A

Does not enter dermis

29
Q

Second degree burns

A

Enters dermis
Blister, painful
Can heal on own but might take ~2 weeks

30
Q

Third degree burns

A

Entire dermis is damaged
White/charred
Need most attention

31
Q

What is the first layer of skin?

A

Epidermis

32
Q

What is the second layer of skin?

A

Dermis

33
Q

What is the third layer of skin?

A

Subcutaneous

34
Q

What is the #1 way to treat a burn that is not healing on its own?

A

Skin grafting

35
Q

Autografting

A

Using person’s own skin for graft

36
Q

Sheet grafting

A

Take piece of healthy skin with dermatome and replace it over burn, donor site becomes painful

37
Q

Meshed grafting

A

Covering a large wound, run good skin through machine making it into a grid to cover more areas, not used often

38
Q

What is a scar?

A

Develops any time the dermal layer of the skin is damaged.

39
Q

Hypertrophic scarring

A

The collagen fibers in hypertrophic scarring are orientated in a “whorl-like” pattern, as compared to normal skin in which collagen aligns in a parallel pattern (Bumpy scar)

40
Q

Keloid scar

A

Scar grows beyond original wound

41
Q

Why is scar management important?

A

Scar tissue is estimated to have 12 times the contractile strength of normal skin, which is clearly strong enough to pull features and joints out of place.
- Affects function

42
Q

Functional problems that occur from scarring

A

If a scar crosses a joint, it can limit range of motion and cause functional deficits.
Can’t talk or eat as well

43
Q

Where are major impairments with pediatric burns?

A

Soft tissue, result in motor deficits

44
Q

Treatment focus for burns

A

Maximizing skin integrity and function, then resuming participation in occupations
- Scar management
- Functional positioning to stop scars from taking over joints
- Discuss changes with patient (disfiguring disability is traumatizing)

45
Q

In pediatric trauma care, who experiences orthostatic hypotension?

A

ALL (burns, SCI, TBI)

46
Q

Who experiences autonomic storming?

A

TBI

47
Q

Who experiences autonomic dysreflexia?

A

SCI