Chapter 23 Flashcards
1
Q
What is anemia
A
reduction in oxygen carrying capacity of blood
2
Q
What are the s/s of anemia
A
- pallor
- dyspnea
- tachycardia
- decreased energy
- decreased regeneration of epithelial tissue
3
Q
Hemolytic anemia
A
- Premature destruction of red blood cells
- Retentions of iron and other production of hemoglobin destruction
- Increase in erythropoiesis
- Some RBCs look normal
- Hyperactive bone marrow – immature RBCs in circulation
4
Q
sickle cell anemia
A
- Autosomal recessive disorder
- The red blood cells become deoxygenated, the hemoglobin changes the shape into sickle
5
Q
s/s of sickle cell anemia
A
- Appear 8-10 weeks of age
- Tachycardia
- Splenomegaly in young adults
- Vascular occlusion and infarction – painful crises and damamge to tissue and organs
6
Q
treatment of sickle cell anemia
A
hydroxyurea
7
Q
thalassemia
A
- Not producing enough of the beta chains or the alpha chains
- Anemia results from defect in hemoglobin
- Cells look Hypochromatic and Microcytic
- found in african americans, asians
8
Q
s/s of thalassemia
A
- Increased hemolysis
- Growth and development is impaired
- Heart failure
- Splenomegaly
- Hepatomegaly
9
Q
treatment of thalessemia
A
- Blood transfusions
- Blood cell transplant
10
Q
iron deficient anemia
A
- Dietary intake may be low
- Increased demand – where utilization of iron may be high as in preganncy and adolescence
- Slow or chronic blood loss – bleeding ulcer, cancer, excessive menstrual flow – decreased iron recycling
- Impair absorption – malabsorption syndrome
- Blood cells look hypochromic and microcytic
11
Q
s/s of iron deficient anemia
A
- Mild – asymptomatic
- Pallor of skin and mucous membranes related to cutaneous vasoconstriction
- Fatigue
- Cold itnolderance due to decreased cell metabolism
- Palpitations, tachycardia, dyspnea
- Delayed healing
12
Q
treatment of iron deficient anemia
A
- Treat underlying problem
- Take iron rich food/iron supplements (iron sulfate/gluconate)
13
Q
pernicious anemia
A
- Caused by gastric atrophy – decreased in parietal cells and the formation of intrinsic factor