chapter 2 Flashcards
what are the two main thoughts on mental disorders
biological and environmental
Biological aspects
downplay the influence of experience
Environmental influences
emphasize external factors (poverty and parenting styles)
what are the three main approaches to viewing mental disorder
Biological & psychodynamic, Humanistic & existential, Behavioural & cognitive
Biological & psychodynamic
view dysfunctional behaviour as the product of forces beyond the individual’s control
Bio encourages a physical basis for disorders – leads to
formulation of a diagnostic system that classifies people as disorder & implies that physical interventions should be the treatment
Humanistic & existential
lay responsibility for action & choices on the individual
Personal experience provides the basis for the development of self-directed behaviour
Behavioural & cognitive
a mix of external and internal factors produce dysfunctions
The way that people are conditioned to learn and the way they think or perceive the world causes the
development
what does the behavioural cognitive approach emphasize
classification of behaviours not people
Seek environmental events that shape dysfunctional responses
behavioural and cogntive approach treatment
manipulating environment or modifying perception / schema / beliefs of people
regarding experience and self
Adopting one of these theories is influenced by
the prevailing social belief system & the individual’s disposition to see human behaviour as being determined by factors beyond or within their control
what are the leves of theories
single factor explanation and interactionist explanation
sing-factor explanation
: attempts to trace the origins of a disorder to one factor
Interactionist explanation
behaviour is the product of interactions between a variety of factors
Can be classified according to their level of explanation
Ex: Maslow’s theory of self-actualization tries to explain all human behaviour while Freud’s try to explain abnormal behaviour
Theories embody 3 features
- Integrate most of what is currently known about the phenomena in the simplest way (parsimony)
- Make testable predictions about aspects of the phenomena that weren’t previously thought of
- Make possible to specify what evidence would deny the theory
Why do theories gain strength?
Evidence supports their predictions
Alternative explanations are rejected
Null Hypothesis
proposes that the prediction made from the theory is false
When you reject it, it provides support for a theory BUT theories are NOT factors and can NEVER be PROVEN to be true
Etiology
the causes or origins of a disorder
General aims of theories
Explain the etiology of behaviour
Identify factors that maintain the behaviour
Predict the course of the disorder
Design effective treatments
Factors involved in the etiology may not be involved in
its maintenance
In disorders where there is a clear biological cause
environmental manipulations may alleviate or prevent
the development of the most serious symptoms
Phenylketonuria (PKU) found in the diet
When detected in newborns, it’s possible to prevent development of severe symptoms like
retardation by administering a diet low in PKU-containing foods
using cognitive therapy methods in depressed & anxious people can change their neurobiology
Decreases activation of the amygdala / hippocampal regions associated w/ negative affect
Increased activation of areas involved in cognitive control of negative emotion
Theories of the etiology of mental disorders
Biological
Psychodynamic
Behavioural & Cognitive
Humanistic & Existentialist
Socio-cultural
Integrative
Biological Models
Adopt the language of medicine = patients, symptoms, treatments
CNS damage is the focus + PNS dysfunction (somatic + autonomic) + endocrine system dysfunction
CNS
Brain has ~100 billion neurons & thousands of billions of glia cells
what three brain regions are in the CNS
forebrain, midbrain, hindbrain
forebrain
speech, perception, memory, learning, planning
midbrain
reticular activating system = control arousal and attention
hindbrain
directs the function og the autonomic NS
Current theories about the brain bases of abnormal behaviour
They focus on the role of neurotransmitters and not neuronal damage
Most research is done on GABA, norepinephrine, serotonin, dopamine
Abnormal behaviour can result from disturbances in ligands in many ways
Too little / much of the ligand produced or released into the synapse
Too few / many receptors on the dendrites
Too few / many ligand-deactivating subs in the synapse
The reuptake process is too rapid / slow
problems with any of the ligand =
alterations in the brain circuits
current research shows that disturbance sin the ligan systems have
general effects
interactions of ligands and subtypes are related to behaviour
Inferring a causal relationship btw disturbances in ligands & abnormal behaviour
Dopamine antagonists are used to treat schizophrenia
neurochemistry and behaviour are bidirectional
Brain Plasticity
capacity of the brain to reorganize its circuitry – influenced by
experienced that occur pre- and postnatally
Ligand function affects behaviour but
behaviour affects neurochemistry
PNS
Somatic and autonomic NS
Autonomic (ANS)
sympathetic + parasympathetic
These systems work cooperatively but in terms of stress
they act antagonistically
Its response is exaggeratedly strong or remarkably week
Sympathetic
readies body for action
Parasympathetic
shuts down digestive process
Overactive ANS
increase readiness to acquire phobias or other anxiety disorders
Deficits in regulation of ANS functions can influence
disordered behaviour
patient with GAD tend to show
decreased parasympathetic regulation of heart rate &
respiration – also show chronic muscle tension (somatic system)
there’s an inflexibility of
the autonomic and somatic systems in GAD
ES and CNS interact in a
feedback loop
It maintains homeostatic levels of hormones circulating in the bloodstream
order of ES2
ES
hypothalamus
pituitary (“master gland”)
adrenal cortex
cortisol (anti-inflammatory)
HPA: hypothalamic pituitary-adrenal cortex axis
Has been studied in regard to anxiety and depression
It involves the release of cortisol into the bloodstream by the adrenal cortex
It increases the # of intracellular glucocorticoid receptors, leading to anti-inflammatory effects & other survival benefits
Sensitivity to stress is implicated in
the etiology of depression and anxiety
Cretinism
dwarf-life appearance and mental disability – result of defective thyroid gland
Hypoglycemia
pancreas fails to produce insulin
Genetics and behaviour
Idea that human behaviour is inherited
Inherited features interact with the environment to produce behaviour