Chapter 14: Abnormal Psychology Flashcards

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1
Q

How did Hippocrates explain the cause of mental disorders?

A

Hippocrates explained the cause of mental disorder resulted from the relative amount of “humors” or bodily fluids a person possessed. For instance, having too much black bile led to melancholia, or extreme sadness and depression. The idea that bodily fluids cause mental illness was abandoned long ago, however.

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2
Q

Explain the difference between a categorical and dimensional model of mental disorders.

A

A categorical model of mental disorders is separated on disorders based on what he could observe: groups of symptoms that occur together. For example, he separated disorders of mood from disorders of cognition. This idea was adopted with the DSM, which is the idea that if disorders can be grouped based on similar etiologies and symptoms, then figuring out how to treat those disorders should be easier. DSM 5 disorders are described in terms of observable symptoms.
A dimensional approach would be to consider psychological disorders along a continuum in which people vary in degree rather than in kind. With categorization, the approach can be compared to a simple switch that turns on or off (black/white). The dimensional approach works more like a dimmer switch, that psychological disorders are extreme versions of normal feelings. We are all a little sad at times, but sometimes we feel more sad than usual. But no specific amount of sadness passes a threshold for depressive disorders. In the third section of DSM, researchers are encouraged to examine whether a dimensional approach might be helpful for understanding psychological disorders, particularly personality disorders (maladaptive extremes of Big 5 personality traits).

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3
Q

What are weaknesses of a categorical approach?

A

A problem with DSM approach is that it implies that a person either has a psychological disorder or does not, which is known as categorical approach. This in turn fails to capture differences in the severity of a disorder.

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4
Q

What is comorbidity?

A

Comorbidity is the idea that many psychological disorders occur together even though the DSM 5 treats them as separate disorders - for example, depression and anxiety, or depression and substance abuse. Accordingly, people who are found to be depressed should also be examined for comorbid conditions. This is possible because of common underlying factors. Although DSM separates disorders involving anxiety from those involving depression, both types involve the trait neuroticism, the tendency to experience frequent and intense negative emotions.

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5
Q

What is the diathesis-stress model?

A

The diathesis-stress model is a model that proposes that a disorder may develop when an underlying vulnerability is coupled with a precipitating event. In this model, an individual can have an underlying vulnerability (known as a diathesis) to a psychological disorder. This diathesis can be biological, such as a genetic predisposition to a specific disorder, or it can be environmental, such as childhood trauma. The vulnerability may not be enough to trigger a disorder, but the addition of stressful circumstances can tip the scales. If the stress level exceeds an individual’s ability to cope, the symptoms of psychological disorder will occur. A family history of psychopathology would suggest vulnerability

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6
Q

Describe how a strict behaviorist would explain the etiology (cause) of a mental disorder and how a cognitive-behavioral perspective would differ.

A

Learning: those are behaviorists who believe behavior is shaped purely by environment; followed by cognitive revolution, you’re not just a rat in a cage and you interpret what is going on
Abnormal psych: strict behaviorists will say, whatever your problem is it’s in your environment just changing structure and environment and how you learn will fix it; cognitive is not just what happens to you is both how you learn it and how you interpret it.

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7
Q

Describe the distinction between internalizing and externalizing disorders.

A

Internalizing disorders are characterized by negative emotions, and they can be grouped into categories that reflect the emotions of distress and fear. These disorders can include major depressive disorder, generalized anxiety disorder, and panic disorder.
Externalizing disorders are characterized by disinhibition. These disorders include alcoholism, conduct disorders, and antisocial personality disorders. The disorders associated with internalizing are more prevalent in females, and those with externalizing are more prevalent in males.

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8
Q

What are some trends regarding disorders

A

A disorder with a large influence from biological factors is likely to be more similar across cultures. A disorder heavily influenced by learning, context, or both is likely to differ across cultures. A major mental health problem around the world might be depression, while manifestations of depression vary by culture.

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9
Q

Describe agoraphobia.

A

Agoraphobia is when people with this disorder are afraid of being in situations in which escape is difficult or impossible. They may fear being in a crowded shopping mall or using public transportation - the fear is so strong it causes panic attacks. As a result, people who suffer from agoraphobia avoid going into open places or places where there might be crowds. They also fear having a panic attack in public.

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10
Q

What two factors do anxiety disorders rely on?

A

Anxiety disorders run on two factors: biased thinking when they tend to think of themselves as threatening and focus excessive attention on perceived threats and learning, where a person can develop a fear of flying by observing another person’s fearful reaction to the closing of cabin doors. Such a fear could generalize to other enclosed spaces, resulting in claustrophobia.
Those with inhibited temperamental style are usually shy and tend to avoid unfamiliar people, so they are more likely to develop social anxiety.

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11
Q

What are obsessions?

A

Obsessive compulsive disorder involves frequent intrusive thoughts and compulsive actions. Obsessions are recurrent, intrusive, and unwanted thoughts or ideas or mental images. They often include fear of contamination, of accidents, or of one’s own aggression. The individual typically attempts to ignore or suppress such thoughts but sometimes engages in particular behaviors to neutralize his or her obsessions.

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12
Q

What are compulsions? How might basic learning processes take a role?

A

Compulsions are particular acts that the OCD patient feels driven to perform over and over. The most common compulsive behaviors are cleaning, checking, and counting. The person might continually check to make sure a door is locked because of an obsession that his or her home might be invaded, or a person mgiht engage in superstitious counting to protect against accidents, such as counting the number of telephone poles. These actions are taken to prevent something dreadful from happening. Those who have OCt fear what they might do or might have done. Checking is one way to calm the anxiety.
People are aware of their obsessions, yet they are unable to stop them. One explanation is that the disorder results from conditioning. Anxiety is somehow paired to a specific event, probably through classical conditioning. As a result, the person engages in behavior that reduces anxiety and therefore is reinforced through operant conditioning. This reduction of anxiety is reinforcing and thus increases the person’s chance of engaging in that behavior again.
Ie hand washing paired with a reduction of anxiety- ocd would reduce this anxiety only temporarily, so this behavior will recur.

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13
Q

Explain the possible role of the caudate nucleus in OCD.

A

OCD has been researched to be in part genetically influenced. Particularly OCD genes appear to control the neurotransmitter glutamate. The caudate, a brain structure involved in suppressing impulses, and is smaller and has structural abnormalities in people with OCD. Brian scans show abnormal activity in the caudates of people with OCD. Because the caudate is involved in impulse suppression, dysfunction in this region may result in the leak of impulses into consciousness. This overworks the prefrontal cortex which is involved in conscious control of behavior.

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14
Q

How might strep infection play a role in OCD?

A

A streptococcal infection apparently can cause a severe form of OCD in some young children. The affected children can suddenly display odd symptoms of OCD, such as engaging in repetitive behaviors, developing irrational fears and obsesions, and having facial tics. An autoimmune response damages the caudate, producing the symptoms of OCD.

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15
Q

What is learned helplessness?

A

Learned helplessness is a second cognitive model of depression. Learned helplessness means that people come to see themselves as unable to have any effect on events in their lives. When animals are placed in aversive situations they cannot escape (such as an unescapable shock), the animals eventually become passive and unresponsive. They lack the motivation to try new methods of escape if given the opportunity. Similarly, people suffering from learned helplessness come to expect that bad things will happen to them and believe they are powerless to avoid negative events. The attributions or explanations they make for negative events refer to personal factors that are stable and global rather than to situational factors that are temporarily and specific. This attributional pattern leads people to feel hopeless about making positive changes in their lifetime. Dysfunctional cognitive components are a cause rather than a consequence of depression.

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16
Q

Describe the primary symptoms of autism spectrum. Read the section on autism and vaccines.

A

The primary symptoms of autism spectrum include are people being seemingly unaware of others. As babies, they do not smile at caregivers, do not respond to vocalizations, and may actively reject physical contact with others. Children may show attention to the eyes before 2 months of age, but after 6 months stop making eye contact. Deficits in communication are the second major cluster of behaviors characteristic of autism spectrum disorders. Children with autism show severe impairments in verbal and nonverbal communication. Even if they vocalize, it is often not with any intent on communicating. They exhibit odd speech patterns (repeating words people have spoken). A third category of deficits includes restricted activities and interests. They appear oblivious to people around them, but they are acutely aware of their surroundings. Although most children automatically pay attention to the social aspects of a situation, those with autism may focus on small insignificant details.
1998: Wakefield claimed to find a connection in 12 children between receiving vaccinations to prevent measles, and developing autism. This was widely reported on the media even though most were skeptical and urged people to wait until replciation was possible. People instead panicked, Jenny McCarthy in 2007 blamed the vaccine for her son’s autism and moved on to become a spokesperson for the anti vaccine movement. Unfortunately, the Wakefield study was fraudulent and he alteredm edical records and lied about his study. Further analysis ended up revealing that there was no actual link between the two factors. Fear of ASD, linked to child vaccinations even though studies prove the opposite led many parents around the globe to forgo vaccinations. As a result, this fear and subsequent denial to get vaccines has led to more and more parents forgoing vaccines, and outbreaks of diseases normally treatable by conventional vaccines. Wakefield conducted this study because the parents told him they remembered the autism starting right after their children got immunized. We realize instead that the symptoms of ASD become apparent at the same period vaccines are normally given. Others might include having lower molars grow. Cases of ASD have increased even though thimerosal is no longer used in vaccines and the number of vaccinated children has dropped. ASD and vaccination are negatively correlated.

17
Q

Describe the primary symptoms of ADHD.

A

The primary symptoms of ADHD include hyperactivity, restlessness, inattentiveness, and impulsiveness. Although these children are often friendly and talkative, they can have trouble making and keeping friends because they miss subtle social cues and make unintentional social mistakes. Many of these symptoms are exaggerations of typical toddler behavior, and thus the line between normal and abnormal behavior is hard to draw. The DSM requires >= 6 symptoms of inattension and >=6 symptoms of hyperactivity or impulsiveness that for at least 6 months interfere with functioning or development. Several of these symptoms must be prior to age 12 and occur in multiple settings.

18
Q

What are the primary findings with respect to possible biological causes of/influences on autism?

A

Gene mutations may play a role in addition to autism being heritable. The rare mutations involve cells having an abnormal number of copies of DNA segments. The mutations may affect the way neural networks are formed during childhodd development. Autism and schizophrenia seem to share the same gene mutations. There are also similarities in the symptoms including impairment and avoiding eye contact. Early childhood events that may result in brain dystunction. The brains of children with autism grow unusually large during the first two years of life, then growth sliows until age 5. The brains of children with autism do not develop normally during adolescence. Genetic factors and such as mutations and nongenetic factors that mgith explain this overgrowth undergrowth pattern. Exposure to antibodies in the wowmb may affect brain development. There’s faulty wiring in the brain, some associated with social thinking, others attention to social aspects of the environment.

19
Q

What are the different ways to define what it means to be psychologically abnormal? What are the weaknesses associated with these different approaches?

A

Statistical abnormality
Violation of cultural standards
Maladaptive behavior, that is behavior that have negative consequences such as runing into traffic mgiht be antisocial personlaity disorder? Emotional distress (subjective perception of suffering) if it’s causing you distress and I believe you, we don’t need to draw a line before I classify you as abnormal/in pain

20
Q

What is the DSM? How does it work?

A

Standing for the Diagnostic and Statistical Manual Of Mental Disorders, it has diagnostic does in it which mostly is what doctors use when facilitating insurance reimbursement (You can’t get reimbursed unless you have the code).

21
Q

What are some of the weaknesses associated with the DSM system of diagnosis?

A

Thinks of categories instead of what’s in common -> conditions don’t say why you have it or what you can do to treat it, it’s just conditions. Your health practitioner at Searle might be giving you a diagnosis in terms of whether you meet the criteria, just so insurance can reimburse you (think they would just look for cold symptoms if you go in with say a cough).
There is an illusion of objectivity; a committee votes on when they want to put a new category/condition in it. Disorders prevent cultural changes (making it not objective)
Everything is abnormal -> you’re more inclined to think you have a disorder going down the symptoms in the manual when you really don’t have one
But we still need a way to communicate and to classify our disorders

The Trouble With Labels

  • > some people have a name for it & google it to find support
  • > Names can be stigmatizing (worthy of disgrace or disapproval) as it changes how we see that person giving us biased interpretations
22
Q

Generalized Anxiety Disorder

A

Includes phobias, which is when anxiety attaches to something and you feel anxiety when you see the thing. You generally are inexplicably tense and in a general state of automatic arousal, you might have free floating anxiety.

23
Q

Panic Disorder

A

Experience of recurrent unexpected panic attacks (where you actually feel like you’re dying and can’t just take a deep breath to make it better), which being unexpected you won’t know when it will happen and will make you even more anxious
You’re often worried and concerned that you’ll have more attacks

24
Q

Major Depressive Disorder

A

For at least 2 weeks, depressed mood for most of the day nearly everyday (arbitrary) can be 12 days for example. You feel worthless, guilt, fatigue, have sleep problems, eating problems, find it hard to concentrate, and have thoughts of death.

25
Q

Persistent Depressive Disorder

A

You have a depressed mood for at least 2 years with no break
It’s becoming more common over generations, simplistic theories of a neurotransmitter deficiency are not accurate (it’s not a chemical imbalance)! It’s mostly accidental if anything the condition treated the patient who just in teh end had increased seratonin Think headache you don’t have an aspirin deficiency when you have a headache. It’s heterogeneous, lots of faces differnt people can have very different symptoms. Medication helps, but not as much or often as people think, leaving people with little to no improvement. Thoughts matter, as people are prone to being more pessimistic CBT helps and can help lower relapse rates, it tends to recur but it also tends to get better. Cardio can help against depression.

26
Q

Bipolar Disorder

A

When you have episodes of depression, but one manic episode
Depression alternating with manic episodes, abnormally elevated mood, inflated self esteem, decreased need for sleep, unusually talkative, racing thoughts
Our treatment options are limited - primarily we can use lithium
It has a high genetic loading (mom, dad, sibling are at risk if you have it)
Creative types are more at risk (risk goes up particularly in writers, particularly in poets)
May involve over-sensitivity in the brain.

27
Q

Schizophrenia

A

The split: Lost grasp on reality
It’s heterogeneous -> some never hallucinate, and there’s not really one thing or one set of symptoms to describe schizophrenia. Key neurotransmitters involved are dopamine and glutamate. Structural brain abnormalities: frontal lobes, ventricles (enlarged, loses brain mass), and the thalamus
There’s an excessive proning of synapses in adolescence
Prenatal environment matters (ie if your mom smoked, was in a polluted area, etc)
Genes and enviornment matter (not really passed on)
Pollution is a risk, media oversimplifies it;

28
Q

What influences behavior and what influences psychological disorders?

A

Behavior: Person + Environment

Psychological Disorder: Bio + Social

29
Q

With respect to schizophrenia, describe the difference between positive and negative symptoms

A

Positive symptoms are not positive, rather think positive like adding another symptom (positive punishment anyone?)
Adding symptoms such as delusion when you think of a false belief, or hallucination when you experience sensory experiences (hearing, seeing something not there)
Adding symptoms such as disorganized speech and behavior (known as word salad) where you mgiht be in a quiet library but you’re screaming a mix of words in a sentence

Negative symptoms take away some functioning, such as the Flat affect where you do not show emotion on your face and are less reactive in general
Antedonia -> loss of ability to feel pleasure
Asociality -> Withdrawal and you won’t talk to others
John Nash -> tells himself it’s not real