Chapter 13, 14, 20 Flashcards
overload
the need to stress the body in order to create adaptations
increased capacity of a system in response to training above the level to which it is accustomed
what are the 3 key components of overload
intensity
duration
frequency
can you have too much overloading
yes which would lead to overtraining or overreaching
reversibility
when training is stopped, the training gains are lost quickly
specificity: training effect is specific to
muscle fibers recruited during exercise
type of contraction (eccentric or concentric)
energy system involved (aerobic vs anaerobic)
suppose you test the VO2 max of highly trained rowers, cyclists, skiers on uphill running on a treadmill and then during their sport specific activity. What changes in VO2 max would be observed?
VO2 max attained by all athletes during their SPORT specific activity was as high or higher than the values obtained on the treadmill
influence of sex on training
men and women respond similarly to training programs even though exercise training Rx should be personalized
how does fitness level relate to training improvement
training improvement is always greater in individuals with lower initial fitness
50% increase in VO2 max in sedentary adults
10-20% improvement in normal, active subjects
3-5% improvement in trained athletes
influence of genetics on training
genetics plays an important role in how people respond to training, however is not the only factor as variability still exists
high responders (genotype E)
individuals with the ideal genetic makeup required for champion endurance athletes
posses a relatively high untrained VO2 max
low responders (genotype A)
possess a relatively low untrained VO2 max
often exhibit limited exercise training response
such as McArdles patients
what is more genetically determined: aerobic or anaerobic capacity
anaerobic capacity is more genetically determined than aerobic capacity
can training improve anaerobic performance
only to a small degree
what anaerobic capacity largely dependent on
fast type IIx fibers which is determined early in development
even if you increase hypertrophy, if you don’t have a lot of these fibers it is hard to increase performance
3 main pillars of performance
VO2 max
economy
LT/VT - how close to VO2 max can I sustain HR for longer
adaptations in muscle as a result of endurance training
repeated excitation and contraction of muscle fibers during endurance training stimulate changes in their structure and function
changes seen in:
muscle fiber type
capillary density
Mb content
mitochondrial function
mitochondrial oxidative enzymes
as a result of endurance training what happens to muscle fiber type
fast to slow shift in muscle fiber type
reduction in the cross sectional area of fast fibers and increase in the number of slow fibers
magnitude of muscle fiber type change is determined by
duration, training, type of training, and genetics
as a result of endurance training what happens to capillary density
increased number of capillaries surrounding muscle fibers leading to enhanced diffusion of O2 (since moving blood as slowly as possible) and the improved removal of wastes
as a result of endurance training, what happens to Mb content
endurance training increases muscle Mb content by 75-80% which supports a muscles increased capacity for oxidative metabolism after training
time course of training mitochondrial changes
muscle mitochondria adapt quickly to training
the # of muscle mitochondria doubles within 5 weeks of training
endurance training effects of mitochondrial volume
endurance training increases the volume of both sarcolemmal (SS) and intermyofibrillar (IMF) mitochondria in muscle fibers
as a result of increased volume of SS and IMF mitochondria, what happens to oxidative capacity
improved oxidative capacity and ability to utilize fat as fuel
share workload across other mitochondria (1 mitochondria- use CHO, 2+ mitochondria, use FATs)
endurance training effects on mitochondrial turnover
increases mitochondrial turnover (which is the breakdown of damaged mitochondria and replacement with healthy mitochondria)