Chapter 11 Flashcards
Cardiovascular chronic adaptations as a result of aerobic training
HEART at rest
Decrease in heart rate, increase in stroke volume, unchanged cardiac output
Decrease in heart rate
A slower heart rate is more efficient because it requires less oxygen than a faster beating heart with the same cardiac output
Increase stroke volume
Due to increased left ventricle volume and mass. Which Is due to reduced cardiac and arterial stiffness.
Increased diastolic filling time. Increased cardiac contractility.
Cardiac output
Remains unchanged at rest due to the balance of HR and SV
Cardiac output at max exercise
Increased Q increases the amount of blood and allows for a more rapid removal of by products.
Cardiovascular chronic adaptations as a result of aerobic training
BLOOD VESSLES
Increase in capillaries, slight decrease of blood flow to the heart, decrease of myocardium O2 consumption, increased capillarisation of skeletal muscle, increased number of capillaries, increased blood flow to skin and increase HDL
Cardiovascular chronic adaptations as a result of aerobic training
HEART
Hypertrophy of the heart muscle, increase in size of the left ventricular activity, decrease in heart rate, increase in stroke volume, cardiac output remains unchanged(rest)
Blood vessels: increase in capillaries
Increase in capillaries results in more blood flow to the heart, which means more oxygen is delivered to the heart muscle to meet the demands of the myocardium
Blood flow to the heart at rest
BLOOD VESSELS
Slight decrease
Myocardium oxygen consumption
BLOOD VESSELS
Decreases because stroke volume increases and heart rate decreases
Increased number of capillaries
BLOOD VESSELS
More capillaries around the muscle leads to an increase in the supply of oxygen and nutrients and an increase in removal of waste products
Blood vessels:
At rest and sub max
Decrease blood flow to the working muscles due to the increased ability of the muscles to deliver, extract and use oxygen
Increased blood flow to the skin
BLOOD VESSELS
Results in greater removal of heat
Decrease LDL Increase HDL
BLOOD VESSELS
HDL’s remove plaque from the artery wall and delivers it to the liver
BLOOD
Increase plasma volume and red blood cell volume, increased haemoglobin in the blood, decrease blood lactate concentration
BLOOD
increase in plasma
Assists in SV because increase in volume of the blood can fill the heart during diastole
Blood
Plasma volumes
Assist in regulation of body temperature
Blood
Increased haemoglobin
Haemoglobin transports oxygen from the lungs to the working muscles
Blood
Blood pressure
Reduces at rest and sub max and has no change during max
Blood
DecreaseBlood lactate concentration
Endurance athletes show a decreased blood lactate and the ability to extend exercise levels before OBLA
Respiratory chronic adaptations as a result of aerobic training
Structural adaptations
Increase lung volume, increased pulmonary function= Increase lung volume, increase diffusion (rest and sub max)
Respiratory chronic adaptations as a result of aerobic training
Functional adaptations
Sub max and rest
Endurance athletes have lower ventilation rates compared to untrained athletes.
Oxygen consumption is the same or slightly lower
Respiratory chronic adaptations as a result of aerobic training
Functional adaptations
Increased ventilation, increase in tidal volume and breathing frequency = increase max ventilation, increase oxygen requirements, increased max oxygen consumption.
Muscular adaptations as a result of aerobic training
Muscle structure
Increased aerobic capacity of slow twitch fibres, increase size of slow twitch fibres (STF are closely associated with increased capillary density surrounding the fibres
Muscular adaptations as a result of aerobic training
A-vO2 diff
Increase in the amount of oxygen extracted from the blood by the muscles(increase a-vo2 diff)
Increased size of slow twitch fibres is due to capillarisation of the fibre which increases diffusion of O2 and CO2
Muscular adaptations as a result of aerobic training
Myoglobin and mitochondria
Increase myoglobin content in STF, increased mitochondria size number and surface area, enhancing the capacity of the muscle to produce ATP
Muscular adaptations as a result of aerobic training
Oxidation of fats
Increased oxidation of free fatty acids.
During sub max exercise: endurance athletes are able to oxidise fatty acids more readily which is beneficial because they can use glycogen sparing.
Factors that increase the ability of the muscles to oxidise fats are
Increase in intramuscular triglycerides, increase in free fatty acids and increase in oxidative enzymes
Muscular adaptations as a result of aerobic training
Oxidation of glycogen
Increase the ability of the skeletal muscle to oxidise glycogen
Adaptations that cause an increase in the energy generating capacity of the muscles are
Increase in mitochondria size number and surface area. Increase in enzyme activity and increase in muscle glycogen stores
Cardiovascular adaptations as a result of anaerobic training
Increase Thickness of left ventricle wall, slight increase of systolic function of the left ventricle (eject blood more forcefully).
Cardiovascular adaptations as a result of anaerobic training
Rest and sub max
Anaerobically trained athletes usually have a lower systolic and diastolic blood pressure at rest and sub max workloads compared to untrained individuals
Muscular adaptations as a result of anaerobic training
Increase capacity of the ATP PC system and the anaerobic glycolysis system, increase ATPase activity, increased glycolytic capacity, increase in the muscular store of ATP, PC and glycogen
Muscular adaptations as a result of anaerobic training
Changes that occur to the skeletal muscle
Increase in energy substrate levels, increase enzyme activity, increase glycolytic capacity (these occur in both twitch fibres)
Muscular adaptations as a result of anaerobic training
Increase fuel stores allow for
Improved performance in events that require high power output
Muscular adaptations as a result of anaerobic training
Increased glycolytic capacity
Increase in glycolytic enzymes and glycogen stores = the rate which glycogen can be broken down into lactic acid is increased
Muscular adaptations as a result of resistance training
Neural adaptations
Increase in strength, increase in motor unit recruitment (greater force), increase in the ability to recruit high threshold motor units, increased firing rate for motor units, increase coordination of muscle movements
Muscular adaptations as a result of resistance training
Neural adaptations
Increase in the ability to recruit high threshold motor units
Muscle fibres are recruited according to size
Muscular adaptations as a result of resistance training
Neural adaptations
Increase in the recruitment of fast twitch fibres
And the time for which the contraction can be maintained
Muscular adaptations as a result of resistance training
Neural adaptations
Increased firing rate of motor units
Results in the Increase in strength and duration of muscular contractions
Muscular adaptations as a result of resistance training
Hypertrophy
Increase muscle size, increase cross sectional areas of the muscle, increase In Contractile proteins, connective tissue will thicken
Muscular adaptations as a result of resistance training
Hypertrophy
Increased muscle size results in one or more of these changes
Increased number and size of myofibrils, increased contractile proteins and increase in size and strength of connective tissue
Muscular adaptations as a result of resistance training
Hypertrophy
Increase cross sectional areas of the muscle
Is a result of increase number and size of myofibrils.
Muscular adaptations as a result of resistance training
Hypertrophy
An increase in contractile proteins
Increase the contractile capacity of the muscle
Muscular adaptations as a result of resistance training
Hypertrophy
Connective tissue will strengthen and thicken
Increase in tendon thickness which assists in force production
VO2 max
The maximum amount of oxygen that can be taken up, transported and utilised per minute. Measured in ml of oxygen/kg of body weight/ minute. Eg 15ml/kg/min
Absolute vo2 max
Doesn’t consider body weight
Relative vo2 max
Considers body weight
Haemoglobin
Oxygen carrying compound found in red blood cells
Mitochondria
Part of a cell, the site of aerobic respiration in muscles. It’s role is to produce ATP
Myoglobin
Oxygen carrying pigment found in muscle cells
What is glycogen sparing
Allows fats to be used more readily and earlier during performances
How does glycogen sparing occur
It results in less use of the anaerobic glycolysis system and allows glycogen to be used later in performances
Adaptations that increase LIP
The mitochondria size, number and surface area increases which increases the capability to oxidise food fuels resulting in an athlete working at higher intensities for longer periods of time before reaching their LIP
How one adaptation can cause an increase or decrease in another
At rest: the decrease of heart rate and increase of stroke volume results in cardiac output to remain unchanged