chapter 10 Flashcards

1
Q

energ itnake

A

energy we get by eating food and to survve and thrive

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1
Q

energy

A

ablitiy to do wrok

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2
Q

energy expenditue

A

fuel for basal meabloci , physical activity food processing

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3
Q

energy balance

A

expenditure = intakae

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4
Q

energy intake sources

A

energy intake sources are 3 energy yieling nutirens carb lipid protein , cellular respiration brek bonds and atp found , alchol provides eenrgy but not nutirent

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5
Q

energy expenditure

A

basal maetbalic rate , basic energy life sustainign functin over time , breathing , heatbeat ,60-70 % energy we burn

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6
Q

fast metabloism

A

they burn calories to sustain bdy even beore physicla actiity

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7
Q

low metablism

A

expnd fewer calories on life sustaining pocess , ahrder time achieving energy balance need to consume fewer calorie or burn more to achieve balance

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8
Q

bmr factors

A

lean body mass msot important . geneics , age , height . gender ,

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9
Q

diet induced thermogenesis

A

danmic effect , thermic effect it accounts for 10 %energ ependiture , more energy is needed to process protein , les for lipids , fat lower thermic effect

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10
Q

physical activity

A

element energy expenditure is nde rcontrol is physicala ctivty , voluntary movemnts expenditure of calories ,

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11
Q

energy balance

A

hnumber calories from carb lipid protein matches the energy we exmed from bmr diet induced and physcial activity

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12
Q

increase body weight

A

increase in adipocytes cells

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12
Q

energy storage

A

whiel most of energy is stored as carbs and glycogen , most i stored as fat aadipocytes cell , adipocytes empty looking they contain , triglycrides and fatty aacids

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13
Q

energy retrivial

A

energy deficit when consume less energy tha our bodies need to perform functions to meet requirments ,

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14
Q

what happens in energy deficit

A

glycogen wil turn into glucose and body proteins will beused fo r aminaocids metbalozied from atp . most energyr etrival omes from afipocytes , if constanly calori eficit adipocytes become smaller and smaller and lose ft and wright loss , prtiens also lean body amss

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15
Q

weight gain

A

lean mss increase muscle mass , fat mass imcrease glycogen storage water fluctate increase more cell more voloume for warer , larger people maye ore likely to see shits water wights form day to dya

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16
Q

weight loss

A

lsoe lean mass , muslce mass decrease , energy decrease with age , water losses , fad dies promtoe water loss , gained back quicky , loss eergy stored as fat , for every 10 kg body fat 1.6 kg water and 8.4 carbon dixoide from lungs through energy or heat

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17
Q

obesity

A

hormone leptin , ecreate by adipocytes comunicaies with brain m promote fullness and energy expeniture promote energy balance , adipokines messenger m communitace tissues maintain hoemstasis

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18
Q

below certian levela fat mass adipokiones

A

poduc ehelath promoting effect dcrease inflmaitiom

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19
Q

obesity chronic diesase

A

excess fats tored in body , long term , sytematic approach , body structure affct and fnctiom , sick fat diseas and fat mass disease

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20
Q

sick aft disease

A

lwo grade infalmtion

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21
Q

fat amss disease

A

weight places on boddy , qualiy life neagtive effect

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22
Q

geentics and body size

A

good chance resmebl gentics , poleygentic

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23
Q

complex cause of oebsity

A

social psycology , physiology , inidviudal psycology , indiviudal phsyial activity , food onsomption , foodproduction , physoal anevuronmn

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24
Q

food consumptiom

A

appetite drive to consume food unflunced by hunger phsyilogical drive to consume foods , saiety fullness hypotalamus to appetite centre , recive message frombody energy balance , limbi cnetre ,obesity have mroe change to appetite genes

25
Q

food production

A

environemtn affect , processfood , nutirent poor food memory emtoion , abundanc enad food chepa food obesogenic environment

26
Q

indivudiual psycology

A

menatl helthy energy state , depression , emotial brain communicatin with appetie cnetre

27
Q

social pysoclogy

A

work , time , lack of time , rush ,

28
Q

physical activity

A

hwo we move our body work , leisure time exercise

29
Q

envirmt physcil activiyt

A

tarsnity weather governetn

30
Q

physiology

A

microbiome nad neergy ablance diffrent bmr genetics m hormonal activity

31
Q

microbiome an energy balance

A

ovesity higher absoibrtio bacteria firmicutes , harvet energy indigestibe carbs metbalozie shor fafty acids

32
Q

gut brain axis

A

affect apeptie , comuncation system

33
Q

key appetite hromones

A

leptinghrelin , gl1

34
Q

leptin

A

r, leptin n is secreted by our fat cells to alert the brain of their energy status, promtoe staiety , smaller adipocytes les sleptin ,

35
Q

ghrelin

A

stomacj , prmtoing appetite , sleep goodnight , good sleep promote caori deficity, hunger sinal

36
Q

glp 1

A

intesitinal cell srepsonse to food intake promote aatiety , ligaliude increase glp 1 lower appetite

37
Q

body composition

A

fat mass / lean body mass

38
Q

weight and eprfcet fat

A

percent body fat ot mroe standard

39
Q

viscerla and subcatonus fat

A

sub located below skin ,vsiecor around abdoim internal organs

40
Q

measing body comp

A

dexa , dual ray absepromty , boen density , body compp, water air displacment , body percentagefat density voloume , skinfold caliper , bimo ,overestimation percentage fat , bmi ,populatiosm wasit curcimfrence , 88 women 102 men

41
Q

weight managment

A

best weight , whatever weight they
achieve while living the healthiest lifestyle they can truly enjoy. There comes a point when a person cannot simply eat
less or exercise more and sƟ ll like their lif

42
Q

weight loss

A

keto idet promot caori decit , low carb diet promtoe calori deficit

43
Q

national weight ocntrol registsry

A

modified food intak emonitro calories , low fa , fibres more ,veggies protein , phsycial activity icnrease calori deficity 500-1000kcal per day att loss over time

44
Q

portion control ,

A

portion six doubles , 35 % aveergae . 10 percent decrease portion 10 decrease in claorie consumptiomm ,

45
Q

hunegr control

A

more protien and fibre foods , slow eating ,

46
Q

willpower

A

oevide signals ith thinking abilities , skipllpower m meal prep cloth prep ,

47
Q

self efficiay

A

ablitiy beleie fot do certian task ,

48
Q

emotiona l eating

A

regualte emotions , crye speak jouranl

49
Q

fad diets

A

ex[enssve gain weight easilu , nutrient poor , cycle nvr ending detox ,

50
Q

medical weight maagemnt

A

orlisat , liragltide , nalr=trexone

51
Q

orlistat

A

blocks activity lipid digestin enezyme small inestine m lipids cant be digest into fatty acidds promtoees withh lsos fat basprbion blocked , fat sooubl evitamins nto absorvbed unabsorbed fat pomtoe oily sols fecal amd gastronteinal issues

52
Q

liraglutide

A

acitivy signaling molecule glp1 m small inteins relase glp1 , huptlamus promtoes satiet reduce food itnake

53
Q

naltrexone

A

reduce food craving , brain drived food seeking , expensive

54
Q

bariatric surgery

A

smalle stomack size , receive less fod , decrease energy itske , 50 to 70 % highes wieght 50 % reduction , dibetes type 2 cv

55
Q

weight gain

A

bm rgenetic cant gain weight , oil nuts , avocado , mroe caories thna thye burn ,

56
Q

binge eatitng

A

loss control , eating severyly cant sto overconsuiming feeling uitl o shame, 2.8 %women 1% men , promtoe obesity ,

57
Q

builiia nervosa , comepnsation electrolytes lost vomiting ,

A
58
Q

aenroxia

A

shape bpdy soze wpem 1.4 % lower body weight enamia , amenhorea , self worth

59
Q

ngih eaitng syndrome

A

25 %calroies binged after dinner wake up mmiddle night to ea , lack appetite in mronign and insomnia , obesity risk

60
Q

orthrexia nervosa

A

check nutrient fact boxes no helathy food , c;eam detox ,nmicronutirent deffecitny social pressure

61
Q

treatment for eating disorders ,

A

cbt cognetive behavoural therpahy